László Kopper

2.1k citations
81 papers · 1.6k indexed · h-index 26

László Kopper

80 papers receiving 1.5k citations

Peers

László Kopper
Comparison fields: 5 of 105
  • Cancer Research 343
  • Oncology 512
  • Cell Biology 215
  • Immunology and Allergy 79
  • Molecular Biology 821
Replace Hiroyuki Hamakawa with:
Hiroyuki Hamakawa Japan
Rachel Tsan United States
Su Yin Lim Australia
Ying C. Henderson United States
Claes G. Tropé Norway
Weiren Luo China
Hiroko Asanuma Japan
Yoshiyasu Nakamura Japan
Satoru Shinriki Japan
Julie Izzo United States
László Kopper relative to Hiroyuki Hamakawa Japan Hiroyuki Hamakawa's profile →
Citations per field
00.5×1.5×
Hiroyuki Hamakawa · 1×
Citations per year

Countries citing papers authored by László Kopper

Since Specialization
Citations

This map shows the geographic impact of László Kopper's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by László Kopper with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites László Kopper more than expected).

Fields of papers citing papers by László Kopper

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by László Kopper. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by László Kopper. The network helps show where László Kopper may publish in the future.

Co-authorship network

The 25 scholars most cited alongside László Kopper, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with László Kopper Line = papers co-authored together László Kopper links everyone, so they are left out of the graph.

All Works

20 of 20 papers shown
#Work
1 20184
2 201619
3 201325
4 201330
5 201224
6 20116
7 20090
8 20075
9 200740
10 200532
11 200523
12 200451
13 20039
14 20031
15 200225
16 199849
17 19975
18 19964
19 199622
20 19901

About László Kopper

László Kopper is a scholar working on Oncology, Cancer Research, Pathology and Forensic Medicine, Genetics and Immunology and Allergy, having authored 81 papers that have together received 1.6k indexed citations. Recurring topics across this work include Lung Cancer Treatments and Mutations (13 papers), Cell death mechanisms and regulation (8 papers), Chronic Lymphocytic Leukemia Research (7 papers), PI3K/AKT/mTOR signaling in cancer (7 papers), Proteoglycans and glycosaminoglycans research (7 papers), Lymphoma Diagnosis and Treatment (7 papers), Glycosylation and Glycoproteins Research (6 papers) and Ubiquitin and proteasome pathways (6 papers). The work is most often cited by research in Cancer Research (343 citations), Oncology (512 citations), Cell Biology (215 citations), Immunology and Allergy (79 citations) and Molecular Biology (821 citations). László Kopper has collaborated with scholars based in Hungary, United States and Italy. Frequent co-authors include Anna Sebestyén, József Tı́már, Sándor Paku, G. Gordon Steel, István Peták, Rudolf Mihalik, Gábor Barna, Titanilla Dankó, Péter Nagy and Zsolt Orosz. Their work appears in journals such as Pathology & Oncology Research, British Journal of Haematology, Modern Pathology, Applied immunohistochemistry & molecular morphology and Cytokine.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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