Ganesh Umapathy

935 total citations
26 papers, 580 citations indexed

About

Ganesh Umapathy is a scholar working on Neurology, Molecular Biology and Cancer Research. According to data from OpenAlex, Ganesh Umapathy has authored 26 papers receiving a total of 580 indexed citations (citations by other indexed papers that have themselves been cited), including 20 papers in Neurology, 17 papers in Molecular Biology and 10 papers in Cancer Research. Recurrent topics in Ganesh Umapathy's work include Neuroblastoma Research and Treatments (20 papers), Cancer therapeutics and mechanisms (10 papers) and Cancer, Hypoxia, and Metabolism (10 papers). Ganesh Umapathy is often cited by papers focused on Neuroblastoma Research and Treatments (20 papers), Cancer therapeutics and mechanisms (10 papers) and Cancer, Hypoxia, and Metabolism (10 papers). Ganesh Umapathy collaborates with scholars based in Sweden, Belgium and China. Ganesh Umapathy's co-authors include Ruth H. Palmer, Bengt Hallberg, Patricia Mendoza-García, Jikui Guan, Kristina Ruuth, Barbara Witek, Fredrik Hugosson, Tommy Martinsson, Abeer El Wakil and Georg Wolfstetter and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Nature Communications and The EMBO Journal.

In The Last Decade

Ganesh Umapathy

24 papers receiving 573 citations

Peers

Ganesh Umapathy
Lili T. Belcastro United States
Renata Sano United States
Karina L. Conkrite United States
JulieAnn Rader United States
Zaowen Chen United States
Ganesh Umapathy
Citations per year, relative to Ganesh Umapathy Ganesh Umapathy (= 1×) peers Christina Schönherr

Countries citing papers authored by Ganesh Umapathy

Since Specialization
Citations

This map shows the geographic impact of Ganesh Umapathy's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Ganesh Umapathy with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Ganesh Umapathy more than expected).

Fields of papers citing papers by Ganesh Umapathy

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Ganesh Umapathy. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Ganesh Umapathy. The network helps show where Ganesh Umapathy may publish in the future.

Co-authorship network of co-authors of Ganesh Umapathy

This figure shows the co-authorship network connecting the top 25 collaborators of Ganesh Umapathy. A scholar is included among the top collaborators of Ganesh Umapathy based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Ganesh Umapathy. Ganesh Umapathy is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Fransson, Susanne, Anna Djos, Johanna Svensson, et al.. (2025). Comparative analysis of whole-genome sequencing of tumor and cfDNA in a neuroblastoma patient: a case report. Frontiers in Oncology. 15. 1569520–1569520.
2.
Djos, Anna, Johanna Svensson, Ganesh Umapathy, et al.. (2024). Loss of Chromosome Y in Neuroblastoma Is Associated With High‐Risk Disease, 11q‐Deletion, and Telomere Maintenance. Genes Chromosomes and Cancer. 63(7). e23260–e23260. 2 indexed citations
3.
Umapathy, Ganesh, Patricia Mendoza-García, Andreas Schlösser, et al.. (2024). The Alk receptor tyrosine kinase regulates Sparkly, a novel activity regulating neuropeptide precursor in the Drosophila central nervous system. eLife. 12. 1 indexed citations
4.
Pettersson, Dan, Amin Al-Awar, Ganesh Umapathy, et al.. (2024). Comparison of 177Lu-octreotate and 177Lu-octreotide for treatment in human neuroblastoma-bearing mice. Heliyon. 10(10). e31409–e31409.
5.
Djos, Anna, Roshan Vaid, Ganesh Umapathy, et al.. (2023). Telomere Maintenance Mechanisms in a Cohort of High-Risk Neuroblastoma Tumors and Its Relation to Genomic Variants in the TERT and ATRX Genes. Cancers. 15(24). 5732–5732. 3 indexed citations
6.
Umapathy, Ganesh, Patricia Mendoza-García, Andreas Schlösser, et al.. (2023). The Alk receptor tyrosine kinase regulates Sparkly, a novel activity regulating neuropeptide precursor in the Drosophila central nervous system. eLife. 12. 1 indexed citations
7.
Lai, Wei‐Yun, Ganesh Umapathy, Bengt Bergman, et al.. (2023). ALK fusion NSCLC oncogenes promote survival and inhibit NK cell responses via SERPINB4 expression. Proceedings of the National Academy of Sciences. 120(8). e2216479120–e2216479120. 15 indexed citations
8.
Umapathy, Ganesh, Wei‐Yun Lai, Jikui Guan, et al.. (2023). ALK signaling primes the DNA damage response sensitizing ALK-driven neuroblastoma to therapeutic ATR inhibition. Proceedings of the National Academy of Sciences. 121(1). e2315242121–e2315242121. 6 indexed citations
9.
Lai, Wei‐Yun, Anh T. Le, Susanne Fransson, et al.. (2022). Novel human-derived EML4-ALK fusion cell lines identify ribonucleotide reductase RRM2 as a target of activated ALK in NSCLC. Lung Cancer. 171. 103–114. 7 indexed citations
11.
Yao, Zhong, Jikui Guan, Jamie Snider, et al.. (2021). Mapping the Phospho-dependent ALK Interactome to Identify Novel Components in ALK Signaling. Journal of Molecular Biology. 433(23). 167283–167283. 5 indexed citations
12.
Umapathy, Ganesh, Wei‐Yun Lai, Barbara Witek, et al.. (2021). ALK ligand ALKAL2 potentiates MYCN‐driven neuroblastoma in the absence of ALK mutation. The EMBO Journal. 40(3). e105784–e105784. 33 indexed citations
13.
Wolfstetter, Georg, et al.. (2021). In vivo Profiling of the Alk Proximitome in the Developing Drosophila Brain. Journal of Molecular Biology. 433(23). 167282–167282. 17 indexed citations
14.
Arefin, Badrul, Yeshwant Kurhe, Ganesh Umapathy, et al.. (2021). ATR inhibition enables complete tumour regression in ALK-driven NB mouse models. Nature Communications. 12(1). 6813–6813. 23 indexed citations
15.
Mus, Liselot, Irina Lambertz, Candy Kumps, et al.. (2020). The ETS transcription factor ETV5 is a target of activated ALK in neuroblastoma contributing to increased tumour aggressiveness. Scientific Reports. 10(1). 218–218. 19 indexed citations
16.
Umapathy, Ganesh, Ruth H. Palmer, & Bengt Hallberg. (2019). P2.14-18 Upregulation of AURKA Leads to Acquired Resistance in EML4-ALK NSCLC Cell Line. Journal of Thoracic Oncology. 14(10). S836–S836. 1 indexed citations
17.
Umapathy, Ganesh, et al.. (2019). Alectinib, an Anaplastic Lymphoma Kinase Inhibitor, Abolishes ALK Activity and Growth in ALK-Positive Neuroblastoma Cells. Frontiers in Oncology. 9. 579–579. 29 indexed citations
18.
Umapathy, Ganesh, Jikui Guan, Dan Gustafsson, et al.. (2017). MEK inhibitor trametinib does not prevent the growth of anaplastic lymphoma kinase (ALK)–addicted neuroblastomas. Science Signaling. 10(507). 41 indexed citations
19.
Guan, Jikui, Elizabeth R. Tucker, Haisu Wan, et al.. (2016). The ALK inhibitor PF-06463922 is effective as a single agent in neuroblastoma driven by expression of ALK and MYCN. Disease Models & Mechanisms. 9(9). 941–952. 53 indexed citations
20.
Guan, Jikui, Ganesh Umapathy, Yasuo Yamazaki, et al.. (2015). FAM150A and FAM150B are activating ligands for anaplastic lymphoma kinase. eLife. 4. e09811–e09811. 105 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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