G. Wei Xu

447 total citations
13 papers, 252 citations indexed

About

G. Wei Xu is a scholar working on Molecular Biology, Cancer Research and Hematology. According to data from OpenAlex, G. Wei Xu has authored 13 papers receiving a total of 252 indexed citations (citations by other indexed papers that have themselves been cited), including 10 papers in Molecular Biology, 3 papers in Cancer Research and 2 papers in Hematology. Recurrent topics in G. Wei Xu's work include Ubiquitin and proteasome pathways (6 papers), Protein Degradation and Inhibitors (5 papers) and RNA modifications and cancer (2 papers). G. Wei Xu is often cited by papers focused on Ubiquitin and proteasome pathways (6 papers), Protein Degradation and Inhibitors (5 papers) and RNA modifications and cancer (2 papers). G. Wei Xu collaborates with scholars based in Canada, China and United States. G. Wei Xu's co-authors include Sirano Dhe‐Paganon, Aaron D. Schimmer, Rose Hurren, Mohsin Ali, Neil MacLean, John C. Reed, David R. Rose, Troy Ketela, Xinliang Mao and Reza Beheshti Zavareh and has published in prestigious journals such as Circulation, Blood and PLoS ONE.

In The Last Decade

G. Wei Xu

10 papers receiving 249 citations

Peers

G. Wei Xu
Li-Ya Chiu Germany
Robert J. Van Gulick United States
Jasmin Batliner Switzerland
Li-Ya Chiu Germany
G. Wei Xu
Citations per year, relative to G. Wei Xu G. Wei Xu (= 1×) peers Li-Ya Chiu

Countries citing papers authored by G. Wei Xu

Since Specialization
Citations

This map shows the geographic impact of G. Wei Xu's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by G. Wei Xu with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites G. Wei Xu more than expected).

Fields of papers citing papers by G. Wei Xu

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by G. Wei Xu. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by G. Wei Xu. The network helps show where G. Wei Xu may publish in the future.

Co-authorship network of co-authors of G. Wei Xu

This figure shows the co-authorship network connecting the top 25 collaborators of G. Wei Xu. A scholar is included among the top collaborators of G. Wei Xu based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with G. Wei Xu. G. Wei Xu is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

13 of 13 papers shown
1.
Zhao, Yanru, Dai B, Zhongwei Yin, et al.. (2024). LncRNA DCRT Protects Against Dilated Cardiomyopathy by Preventing NDUFS2 Alternative Splicing by Binding to PTBP1. Circulation. 150(13). 1030–1049. 16 indexed citations
2.
Yang, Jun, et al.. (2024). Oncostatin M promotes osteogenic differentiation of tendon-derived stem cells through the JAK2/STAT3 signalling pathway. Journal of Orthopaedic Surgery and Research. 19(1). 407–407. 5 indexed citations
3.
Song, Yongxiang, et al.. (2023). Ras-ERK1/2-YAP axis mediates posterior cell proliferation in cancer cell clusters. The European Physical Journal Special Topics. 232(16). 2751–2759.
4.
Liu, Tao, Zhengfeng Zhu, Yongxiang Song, et al.. (2023). Roles of circRNAs in regulating the tumor microenvironment. Medical Oncology. 40(11). 329–329. 14 indexed citations
5.
Li, Mingyong, Jun Fu, Xiaoming Fan, et al.. (2023). sTRBC1 and cyTRBC1 Expression Distinguishes Indolent T-Lymphoblastic Proliferations From T-Lymphoblastic Leukemia/Lymphoma. The American Journal of Surgical Pathology. 47(11). 1325–1331. 1 indexed citations
6.
Chen, Cheng, et al.. (2022). Review of outcomes of delayed chest closure following lung transplantation: a meta-analysis. Journal of Cardiothoracic Surgery. 17(1). 122–122. 7 indexed citations
8.
Barghout, Samir H., Neil MacLean, G. Wei Xu, et al.. (2018). A Genome-Wide CRISPR/Cas9 Knockout Screen Identifies BEND3 As a Determinant of Sensitivity to UBA1 Inhibition in Acute Myeloid Leukemia. Blood. 132(Supplement 1). 1350–1350. 1 indexed citations
9.
Barghout, Samir H., Parasvi S. Patel, Xiaoming Wang, et al.. (2017). TAK-243 Is a Selective UBA1 Inhibitor That Displays Preclinical Activity in Acute Myeloid Leukemia (AML). Blood. 130(Suppl_1). 814–814.
10.
Xu, G. Wei, Julia I. Toth, Stacey-Lynn Paiva, et al.. (2014). Mutations in UBA3 Confer Resistance to the NEDD8-Activating Enzyme Inhibitor MLN4924 in Human Leukemic Cells. PLoS ONE. 9(4). e93530–e93530. 33 indexed citations
11.
Xu, G. Wei, Julia I. Toth, Stacey-Lynn Paiva, et al.. (2013). Mutations In UBA3 Confer Resistance To The NEDD8-Activating Enzyme Inhibitor MLN4924 In Human Leukemic Cells. Blood. 122(21). 2527–2527. 6 indexed citations
12.
Ali, Mohsin, Vijay Shahani, G. Wei Xu, et al.. (2011). Identification of NAE Inhibitors Exhibiting Potent Activity in Leukemia Cells: Exploring the Structural Determinants of NAE Specificity. ACS Medicinal Chemistry Letters. 2(8). 577–582. 37 indexed citations
13.
Xu, G. Wei, Mohsin Ali, Tabitha E. Wood, et al.. (2010). The ubiquitin-activating enzyme E1 as a therapeutic target for the treatment of leukemia and multiple myeloma. Blood. 115(11). 2251–2259. 132 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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