G. Schultz

8.5k total citations · 1 hit paper
118 papers, 7.0k citations indexed

About

G. Schultz is a scholar working on Molecular Biology, Cellular and Molecular Neuroscience and Physiology. According to data from OpenAlex, G. Schultz has authored 118 papers receiving a total of 7.0k indexed citations (citations by other indexed papers that have themselves been cited), including 78 papers in Molecular Biology, 20 papers in Cellular and Molecular Neuroscience and 14 papers in Physiology. Recurrent topics in G. Schultz's work include Ion channel regulation and function (30 papers), Receptor Mechanisms and Signaling (22 papers) and Neuroscience and Neuropharmacology Research (15 papers). G. Schultz is often cited by papers focused on Ion channel regulation and function (30 papers), Receptor Mechanisms and Signaling (22 papers) and Neuroscience and Neuropharmacology Research (15 papers). G. Schultz collaborates with scholars based in Germany, United States and Canada. G. Schultz's co-authors include J. Hescheler, Walter Rosenthal, W. Trautwein, Doris Koesling, Christiane Kleuss, B. Wittig, Eycke Böhme, Karsten Spicher, Stefan Offermanns and Klaus Aktories and has published in prestigious journals such as Nature, Proceedings of the National Academy of Sciences and Journal of Biological Chemistry.

In The Last Decade

G. Schultz

117 papers receiving 6.8k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

The GTP-binding protein, Go9 regulates neuronal calcium channels

1987 763 citations J. Hescheler, Walter Rosenthal et al. profile →

Peers

Countries citing papers authored by G. Schultz

Since Specialization

Citations

This map shows the geographic impact of G. Schultz's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by G. Schultz with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites G. Schultz more than expected).

Fields of papers citing papers by G. Schultz

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by G. Schultz. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by G. Schultz. The network helps show where G. Schultz may publish in the future.

Co-authorship network of co-authors of G. Schultz

This figure shows the co-authorship network connecting the top 25 collaborators of G. Schultz. A scholar is included among the top collaborators of G. Schultz based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with G. Schultz. G. Schultz is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

#	Work	Indexed citations
1	Regulation of heterologously expressed transient receptor potential-like channels by calcium ions 1998 ·Neuroscience ·Alexander G. Obukhov, G. Schultz, Andreas Lückhoff	26
2	Reconstitution of mutant V2 vasopressin receptors by adenovirus-mediated gene transfer. Molecular basis and clinical implication. 1997 ·Journal of Clinical Investigation ·Torsten Schöneberg, Volker Sandig, Jürgen Wess, Thomas Gudermann, G. Schultz	55
3	IMIDAZOLINE BINDING SITES AND SIGNAL TRANSDUCTION PATHWAYS 1996 ·Clinical and Experimental Pharmacology and Physiology ·Ian Musgrave, Dietmar Krautwurst, G. Schultz	16
4	γ-Aminobutyric acid secretion from pancreatic neuroendocrine cells 1996 ·Gastroenterology ·(unknown), (unknown), Carsten Strübing, Hans Scherübl, G. Schultz, E.O. Riecken, Bertram Wiedenmann	44
5	Clonidine and Cirazoline Inhibit Activation of Nicotinic Channels in PC‐12 Cells^fn1 1995 ·Annals of the New York Academy of Sciences ·Ian Musgrave, Dietmar Krautwurst, J. Hescheler, G. Schultz	23
6	Guanylyl cyclase receptors. 1994 ·Molecular Biology of the Cell ·David L. Garbers, Doris Koesling, G. Schultz	93
7	Stimulation of mitogen-activated protein kinase activity by different secretory stimuli in rat basophilic leukemia cells. 1994 ·The Journal of Immunology ·Stefan Offermanns, S V Jones, (unknown), G. Schultz	54
8	G12 and G13 α-Subunits Are Immunochemically Detectable in Most Membranes of Various Mammalian Cells and Tissues 1994 ·Biochemical and Biophysical Research Communications ·Karsten Spicher, Frank Kalkbrenner, Andrea Zobel, Rainer Harhammer, Bernd Nürnberg, Ariane Söling, G. Schultz	58
9	Novel potent inhibitor of receptor-activated nonselective cation currents in HL-60 cells. 1993 ·Molecular Pharmacology ·Dietmar Krautwurst, J. Hescheler, D. Arndts, (unknown), Rudolf Hammer, G. Schultz	56
10	Inversion of Ca²⁺ current modulation during recovery of neuroblastoma cells from pertussis toxin pretreatment 1993 ·FEBS Letters ·Pascal Friederich, Bernd Nürnberg, G. Schultz, J. Hescheler	10
11	Molecular cloning and expression of a new α‐subunit of soluble guanylyl cyclase Interchangeability of the α‐subunits of the enzyme 1991 ·FEBS Letters ·Christian Harteneck, (unknown), Doris Koesling, (unknown), Eycke Böhme, G. Schultz	134
12	Identification of the G-protein α-subunit encoded by αo2 cDNA as a 39 kDa pertussis toxin substrate 1991 ·Biochemical and Biophysical Research Communications ·Karsten Spicher, Franz‐Josef Klinz, Uwe Rudolph, Juan Codina, Lutz Birnbaumer, G. Schultz, Walter Rosenthal	28
13	Agonist‐sensitive binding of a photoreactive GTP analog to a G‐protein α‐subunit in membranes of HL‐60 cells 1990 ·FEBS Letters ·(unknown), Rainer Schäfer, Barbara Hoffmann, (unknown), Karsten Spicher, Klaus‐Dieter Hinsch, G. Schultz, Walter Rosenthal	43
14	Phosphorylation of G-protein α-subunits in intact adipose cells: evidence against a mediating role in insulin-dependent metabolic effects 1989 ·European Journal of Pharmacology Molecular Pharmacology ·Hans‐Georg Joost, C. Schmitz-Salue, Klaus‐Dieter Hinsch, G. Schultz, Walter Rosenthal	3
15	Adipocyte plasma membranes contain two G_i subtypes but are devoid of G_o 1988 ·FEBS Letters ·Klaus‐Dieter Hinsch, Walter Rosenthal, Karsten Spicher, Thomas Binder, Heinrich Gausepohlꝉ, Rainer Frank, G. Schultz, Hans‐Georg Joost	47
16	Acetylcholine and cholecystokinin receptors functionally couple by different G‐proteins to phospholipase C in pancreatic acinar cells 1988 ·FEBS Letters ·(unknown), Hrvoje Banfíƈ, (unknown), G. Schultz, Irene Schulz	52
17	Guaninnucleotid-bindende Proteine als membranäre Signaltransduktionskomponenten und Regulatoren enzymatischer Effektoren 1988 ·Journal of Molecular Medicine ·Walter Rosenthal, G. Schultz	4
18	Funktionen Guaninnucleotid-bindender Proteine bei der rezeptorvermittelten Modulation spannungsabhängiger Ionenkanäle 1988 ·Journal of Molecular Medicine ·Walter Rosenthal, G. Schultz	1
19	Effects of sodium nitroprusside and other smooth muscle relaxants on cyclic GMP formation in smooth muscle and platelets. 1978 ·Munich Personal RePEc Archive (Ludwig Maximilian University of Munich) ·Eycke Böhme, Graf H, G. Schultz	133
20	Reduction of adenylate cyclase activity in lysates of human platelets by the alpha-adrenergic component of epinephrine. 1976 ·Munich Personal RePEc Archive (Ludwig Maximilian University of Munich) ·(unknown), (unknown), G. Schultz	136

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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