Fuminori Saitoh
- Molecular Biology
- Cellular and Molecular Neuroscience top 10%
- Cell Biology top 10%
- Immunology
- Physiology
- Co-authors
- Toshiyuki ArakiShuji WakatsukiTatsuo SuzukiKatsuo KumagaiKazuyuki ItohRyoji SuzukiShuji HinumaHiroki Fujieda
- Topics
- Neuroscience and Neuropharmacology Research (5 papers)Retinal Development and Disorders (4 papers)Nerve injury and regeneration (4 papers)
- Partner nations
- JapanUnited States
In The Last Decade
Fuminori Saitoh
14 papers receiving 576 citations
Peers
Comparison fields: 5 of 77
- Molecular Biology 331
- Cellular and Molecular Neuroscience 202
- Cell Biology 120
- Immunology 115
- Physiology 60
Countries citing papers authored by Fuminori Saitoh
This map shows the geographic impact of Fuminori Saitoh's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Fuminori Saitoh with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Fuminori Saitoh more than expected).
Fields of papers citing papers by Fuminori Saitoh
This network shows the impact of papers produced by Fuminori Saitoh. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Fuminori Saitoh. The network helps show where Fuminori Saitoh may publish in the future.
Co-authorship network of co-authors of Fuminori Saitoh
This figure shows the co-authorship network connecting the top 25 collaborators of Fuminori Saitoh. A scholar is included among the top collaborators of Fuminori Saitoh based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Fuminori Saitoh. Fuminori Saitoh is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 0 | |
| 2 | 30 | |
| 3 | 4 | |
| 4 | 8 | |
| 5 | 22 | |
| 6 | 18 | |
| 7 | 121 | |
| 8 | 59 | |
| 9 | 41 | |
| 10 | 20 | |
| 11 | 39 | |
| 12 | 91 | |
| 13 | Induction of Ley antigen by 5-aza-2'-deoxycytidine in association with differentiation and apoptosis in human pancreatic cancer cells. | 20 |
| 14 | Induction by 5-aza-2'-deoxycytidine, an inhibitor of DNA methylation, of Le(y) antigen, apoptosis and differentiation in human lung cancer cells. | 14 |
| 15 | 111 |
About Fuminori Saitoh
Fuminori Saitoh is a scholar working on Cellular and Molecular Neuroscience, Cell Biology and Pharmaceutical Science, having authored 15 papers that have together received 598 indexed citations. Recurring topics across this work include Neuroscience and Neuropharmacology Research (5 papers), Retinal Development and Disorders (4 papers) and Nerve injury and regeneration (4 papers). The work is most often cited by research in Developmental Neuroscience (49 citations), Cellular and Molecular Neuroscience (202 citations) and Cell Biology (120 citations). Fuminori Saitoh has collaborated with scholars based in Japan and United States. Frequent co-authors include Toshiyuki Araki, Shuji Wakatsuki, Tatsuo Suzuki, Katsuo Kumagai, Kazuyuki Itoh, Ryoji Suzuki, Shuji Hinuma, Hiroki Fujieda, Hideo Shimizu and Hiroshi Takagi. Their work appears in journals such as Journal of Biological Chemistry, Journal of Neuroscience and The Journal of Immunology.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.