Franklin J. Moy
- Molecular Biology top 10%
- Oncology top 10%
- Cancer Research top 10%
- Genetics top 10%
- Cell Biology top 10%
- Co-authors
- Robert PowersPranab K. ChandaJeremy I. LevinHarold A. ScheragaPeter BöhlenAndrew P. SeddonJames ChenG.T. Montelione
- Topics
- Peptidase Inhibition and Analysis (7 papers)Protease and Inhibitor Mechanisms (6 papers)Monoclonal and Polyclonal Antibodies Research (6 papers)
- Journals
- Proceedings of the National Academy of SciencesJournal of the American Chemical SocietyJournal of Biological Chemistry
- Partner nations
- United StatesCanadaAustralia
In The Last Decade
Franklin J. Moy
30 papers receiving 1.1k citations
Peers
Comparison fields: 5 of 90
- Molecular Biology 750
- Oncology 311
- Cancer Research 216
- Genetics 196
- Cell Biology 153
Countries citing papers authored by Franklin J. Moy
This map shows the geographic impact of Franklin J. Moy's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Franklin J. Moy with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Franklin J. Moy more than expected).
Fields of papers citing papers by Franklin J. Moy
This network shows the impact of papers produced by Franklin J. Moy. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Franklin J. Moy. The network helps show where Franklin J. Moy may publish in the future.
Co-authorship network of co-authors of Franklin J. Moy
This figure shows the co-authorship network connecting the top 25 collaborators of Franklin J. Moy. A scholar is included among the top collaborators of Franklin J. Moy based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Franklin J. Moy. Franklin J. Moy is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 8 | |
| 2 | 17 | |
| 3 | 20 | |
| 4 | 73 | |
| 5 | 13 | |
| 6 | 45 | |
| 7 | 51 | |
| 8 | 44 | |
| 9 | 43 | |
| 10 | 3 | |
| 11 | 46 | |
| 12 | 23 | |
| 13 | 8 | |
| 14 | 26 | |
| 15 | 63 | |
| 16 | 74 | |
| 17 | 46 | |
| 18 | 23 | |
| 19 | 31 | |
| 20 | 40 |
About Franklin J. Moy
Franklin J. Moy is a scholar working on Oncology, Cancer Research and Molecular Biology, having authored 30 papers that have together received 1.1k indexed citations. Recurring topics across this work include Peptidase Inhibition and Analysis (7 papers), Protease and Inhibitor Mechanisms (6 papers) and Monoclonal and Polyclonal Antibodies Research (6 papers). The work is most often cited by research in Cancer Research (216 citations), Oncology (311 citations) and Molecular Biology (750 citations). Franklin J. Moy has collaborated with scholars based in United States, Canada and Australia. Frequent co-authors include Robert Powers, Pranab K. Chanda, Jeremy I. Levin, Harold A. Scheraga, Peter Böhlen, Andrew P. Seddon, James Chen, G.T. Montelione, Frederick W. Dahlquist and Philip Matsumura. Their work appears in journals such as Proceedings of the National Academy of Sciences, Journal of the American Chemical Society and Journal of Biological Chemistry.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.