Eyal Reinstein
- Molecular Biology top 10%
- Genetics top 5%
- Cellular and Molecular Neuroscience top 5%
- Oncology top 10%
- Epidemiology
- Co-authors
- Aaron CiechanoverAlan L. SchwartzChristopher J. CummingsHarry T. OrrYong‐hui JiangYaling SunHuda Y. ZoghbiBarbara Antalffy
- Topics
- Connective tissue disorders research (10 papers)Ubiquitin and proteasome pathways (8 papers)Cardiomyopathy and Myosin Studies (4 papers)
- Partner nations
- IsraelUnited StatesNetherlands
In The Last Decade
Eyal Reinstein
41 papers receiving 1.5k citations
Hit Papers
Peers
Comparison fields: 5 of 97
- Molecular Biology 1.0k
- Genetics 359
- Cellular and Molecular Neuroscience 342
- Oncology 324
- Epidemiology 235
Countries citing papers authored by Eyal Reinstein
This map shows the geographic impact of Eyal Reinstein's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Eyal Reinstein with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Eyal Reinstein more than expected).
Fields of papers citing papers by Eyal Reinstein
This network shows the impact of papers produced by Eyal Reinstein. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Eyal Reinstein. The network helps show where Eyal Reinstein may publish in the future.
Co-authorship network of co-authors of Eyal Reinstein
This figure shows the co-authorship network connecting the top 25 collaborators of Eyal Reinstein. A scholar is included among the top collaborators of Eyal Reinstein based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Eyal Reinstein. Eyal Reinstein is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | Frequent aneuploidy in primary human T cells after CRISPR–Cas9 cleavagebreakdown → | 122 |
| 2 | 3 | |
| 3 | 1 | |
| 4 | 7 | |
| 5 | 8 | |
| 6 | 13 | |
| 7 | 21 | |
| 8 | 33 | |
| 9 | 15 | |
| 10 | 12 | |
| 11 | 1 | |
| 12 | 57 | |
| 13 | 10 | |
| 14 | 29 | |
| 15 | 26 | |
| 16 | 12 | |
| 17 | 1 | |
| 18 | 22 | |
| 19 | Immunologic aspects of protein degradation by the ubiquitin-proteasome system. | 22 |
| 20 | 65 |
About Eyal Reinstein
Eyal Reinstein is a scholar working on Cell Biology, Genetics and Cardiology and Cardiovascular Medicine, having authored 41 papers that have together received 1.5k indexed citations. Recurring topics across this work include Connective tissue disorders research (10 papers), Ubiquitin and proteasome pathways (8 papers) and Cardiomyopathy and Myosin Studies (4 papers). The work is most often cited by research in Cellular and Molecular Neuroscience (342 citations), Molecular Biology (1.0k citations) and Genetics (359 citations). Eyal Reinstein has collaborated with scholars based in Israel, United States and Netherlands. Frequent co-authors include Aaron Ciechanover, Alan L. Schwartz, Christopher J. Cummings, Harry T. Orr, Yong‐hui Jiang, Yaling Sun, Huda Y. Zoghbi, Barbara Antalffy, Arthur L. Beaudet and David L. Rimoin. Their work appears in journals such as Proceedings of the National Academy of Sciences, Journal of Biological Chemistry and Neuron.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.