Christopher J. Cummings

4.0k total citations · 2 hit papers
12 papers, 3.2k citations indexed

About

Christopher J. Cummings is a scholar working on Molecular Biology, Cellular and Molecular Neuroscience and Neurology. According to data from OpenAlex, Christopher J. Cummings has authored 12 papers receiving a total of 3.2k indexed citations (citations by other indexed papers that have themselves been cited), including 12 papers in Molecular Biology, 10 papers in Cellular and Molecular Neuroscience and 2 papers in Neurology. Recurrent topics in Christopher J. Cummings's work include Mitochondrial Function and Pathology (10 papers), Genetic Neurodegenerative Diseases (10 papers) and Ubiquitin and proteasome pathways (5 papers). Christopher J. Cummings is often cited by papers focused on Mitochondrial Function and Pathology (10 papers), Genetic Neurodegenerative Diseases (10 papers) and Ubiquitin and proteasome pathways (5 papers). Christopher J. Cummings collaborates with scholars based in United States, Israel and Japan. Christopher J. Cummings's co-authors include Huda Y. Zoghbi, Harry T. Orr, Barbara Antalffy, Michael A. Mancini, Donald Defranco, Beena T. Koshy, Ivan A. Klement, Pamela J. Skinner, Kristian Helin and Antonio Servadio and has published in prestigious journals such as Nature, Neuron and Nature Genetics.

In The Last Decade

Christopher J. Cummings

12 papers receiving 3.1k citations

Hit Papers

Chaperone suppression of aggregation and altered subcellu... 1998 2026 2007 2016 1998 1999 200 400 600
What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

  1. Chaperone suppression of aggregation and altered subcellular proteasome localization imply protein misfolding in SCA1
    1998 635 citations Christopher J. Cummings, Michael A. Mancini et al. Nature Genetics profile →
  2. Mutation of the E6-AP Ubiquitin Ligase Reduces Nuclear Inclusion Frequency While Accelerating Polyglutamine-Induced Pathology in SCA1 Mice
    1999 409 citations Christopher J. Cummings, Eyal Reinstein et al. Neuron profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Christopher J. Cummings United States 11 2.8k 2.2k 542 437 269 12 3.2k
Annie Sittler France 20 2.1k 0.8× 1.3k 0.6× 395 0.7× 371 0.8× 327 1.2× 29 2.4k
Jillian K. Cooper United States 10 2.2k 0.8× 2.0k 0.9× 676 1.2× 161 0.4× 257 1.0× 16 2.5k
Francesca Persichetti Italy 28 2.1k 0.8× 1.7k 0.8× 735 1.4× 207 0.5× 221 0.8× 51 2.6k
Andrew D. Strand United States 18 1.9k 0.7× 1.2k 0.5× 375 0.7× 127 0.3× 183 0.7× 24 2.4k
Michael A. Kalchman Canada 11 2.1k 0.8× 2.0k 0.9× 790 1.5× 208 0.5× 147 0.5× 11 2.5k
Makoto Minamiyama Japan 17 1.6k 0.6× 1.1k 0.5× 295 0.5× 313 0.7× 116 0.4× 24 2.0k
Yaohui Chai United States 11 1.4k 0.5× 1.1k 0.5× 237 0.4× 314 0.7× 120 0.4× 13 1.7k
Etienne Régulier Switzerland 22 1.4k 0.5× 929 0.4× 343 0.6× 227 0.5× 359 1.3× 33 2.0k
Tammy Gillis United States 22 1.5k 0.5× 1.3k 0.6× 506 0.9× 147 0.3× 210 0.8× 36 1.9k
Christian Landles United Kingdom 19 1.6k 0.6× 1.5k 0.7× 460 0.8× 139 0.3× 147 0.5× 25 2.6k

Countries citing papers authored by Christopher J. Cummings

Since Specialization
Citations

This map shows the geographic impact of Christopher J. Cummings's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Christopher J. Cummings with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Christopher J. Cummings more than expected).

Fields of papers citing papers by Christopher J. Cummings

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Christopher J. Cummings. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Christopher J. Cummings. The network helps show where Christopher J. Cummings may publish in the future.

Co-authorship network of co-authors of Christopher J. Cummings

This figure shows the co-authorship network connecting the top 25 collaborators of Christopher J. Cummings. A scholar is included among the top collaborators of Christopher J. Cummings based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Christopher J. Cummings. Christopher J. Cummings is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

12 of 12 papers shown
1.
Cummings, Christopher J.. (2001). Over-expression of inducible HSP70 chaperone suppresses neuropathology and improves motor function in SCA1 mice. Human Molecular Genetics. 10(14). 1511–1518. 376 indexed citations
2.
Cummings, Christopher J.. (2000). Fourteen and counting: unraveling trinucleotide repeat diseases. Human Molecular Genetics. 9(6). 909–916. 340 indexed citations
3.
Cummings, Christopher J. & Huda Y. Zoghbi. (2000). Trinucleotide Repeats: Mechanisms and Pathophysiology. Annual Review of Genomics and Human Genetics. 1(1). 281–328. 235 indexed citations
4.
Lin, Xi, Christopher J. Cummings, & Huda Y. Zoghbi. (1999). Expanding Our Understanding of Polyglutamine Diseases through Mouse Models. Neuron. 24(3). 499–502. 62 indexed citations
5.
Stenoien, David L., Christopher J. Cummings, Henry P. Adams, et al.. (1999). Polyglutamine-Expanded Androgen Receptors Form Aggregates That Sequester Heat Shock Proteins, Proteasome Components and SRC-1, and Are Suppressed by the HDJ-2 Chaperone. Human Molecular Genetics. 8(5). 731–741. 373 indexed citations
6.
Cummings, Christopher J., Harry T. Orr, & Huda Y. Zoghbi. (1999). Progress in pathogenesis studies of spinocerebellar ataxia type 1. Philosophical Transactions of the Royal Society B Biological Sciences. 354(1386). 1079–1081. 40 indexed citations
7.
Cummings, Christopher J., Eyal Reinstein, Yaling Sun, et al.. (1999). Mutation of the E6-AP Ubiquitin Ligase Reduces Nuclear Inclusion Frequency While Accelerating Polyglutamine-Induced Pathology in SCA1 Mice. Neuron. 24(4). 879–892. 409 indexed citations breakdown →
8.
Cummings, Christopher J., Michael A. Mancini, Barbara Antalffy, et al.. (1998). Chaperone suppression of aggregation and altered subcellular proteasome localization imply protein misfolding in SCA1. Nature Genetics. 19(2). 148–154. 635 indexed citations breakdown →
9.
Cummings, Christopher J., E. Jill Dahle, & Huda Y. Zoghbi. (1998). Analysis of the genomic structure of the human glycine receptor alpha2 subunit gene and exclusion of this gene as a candidate for Rett syndrome.. PubMed. 78(2). 176–8. 8 indexed citations
10.
Skinner, Pamela J., Beena T. Koshy, Christopher J. Cummings, et al.. (1997). Ataxin-1 with an expanded glutamine tract alters nuclear matrix-associated structures. Nature. 389(6654). 971–974. 461 indexed citations
11.
Matilla‐Dueñas, Antoni, Beena T. Koshy, Christopher J. Cummings, et al.. (1997). The cerebellar leucine-rich acidic nuclear protein interacts with ataxin-1. Nature. 389(6654). 974–978. 209 indexed citations
12.
Cummings, Christopher J. & Tim Fowler. (1996). Secretion of Trichoderma reesei β-glucosidase by Saccharomyces cerevisiae. Current Genetics. 29(3). 227–233. 20 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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