Erin D. Lew

3.5k total citations · 1 hit paper
20 papers, 2.5k citations indexed

About

Erin D. Lew is a scholar working on Immunology, Molecular Biology and Oncology. According to data from OpenAlex, Erin D. Lew has authored 20 papers receiving a total of 2.5k indexed citations (citations by other indexed papers that have themselves been cited), including 12 papers in Immunology, 8 papers in Molecular Biology and 4 papers in Oncology. Recurrent topics in Erin D. Lew's work include Phagocytosis and Immune Regulation (12 papers), Fibroblast Growth Factor Research (5 papers) and Protein Tyrosine Phosphatases (5 papers). Erin D. Lew is often cited by papers focused on Phagocytosis and Immune Regulation (12 papers), Fibroblast Growth Factor Research (5 papers) and Protein Tyrosine Phosphatases (5 papers). Erin D. Lew collaborates with scholars based in United States, Germany and United Kingdom. Erin D. Lew's co-authors include Greg Lemke, Anna Zagórska, Paqui G. Través, Joseph Schlessinger, Patrick Burrola, Ian Dransfield, Cristina M. Furdui, Karen S. Anderson, Irit Lax and Carla V. Rothlin and has published in prestigious journals such as Nature, Cell and Proceedings of the National Academy of Sciences.

In The Last Decade

Erin D. Lew

19 papers receiving 2.5k citations

Hit Papers

TAM receptors regulate multiple features of microglial ph... 2016 2026 2019 2022 2016 100 200 300 400

Peers

Erin D. Lew
Violeta Chiţu United States
Marjan van Meurs Netherlands
Brian P. Daniels United States
Bettina Schreiner Switzerland
Aaron J. Johnson United States
Violeta Chiţu United States
Erin D. Lew
Citations per year, relative to Erin D. Lew Erin D. Lew (= 1×) peers Violeta Chiţu

Countries citing papers authored by Erin D. Lew

Since Specialization
Citations

This map shows the geographic impact of Erin D. Lew's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Erin D. Lew with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Erin D. Lew more than expected).

Fields of papers citing papers by Erin D. Lew

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Erin D. Lew. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Erin D. Lew. The network helps show where Erin D. Lew may publish in the future.

Co-authorship network of co-authors of Erin D. Lew

This figure shows the co-authorship network connecting the top 25 collaborators of Erin D. Lew. A scholar is included among the top collaborators of Erin D. Lew based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Erin D. Lew. Erin D. Lew is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Yokoyama, Yumi, Erin D. Lew, Ruth Seelige, et al.. (2019). Immuno-oncological Efficacy of RXDX-106, a Novel TAM (TYRO3, AXL, MER) Family Small-Molecule Kinase Inhibitor. Cancer Research. 79(8). 1996–2008. 70 indexed citations
2.
Yokoyama, Yumi, Erin D. Lew, Ruth Seelige, et al.. (2017). Abstract 4698: Immuno-oncological efficacy of RXDX-106, a novel, selective and potent small molecule TAM (TYRO3, AXL, MER) inhibitor. Cancer Research. 77(13_Supplement). 4698–4698. 1 indexed citations
3.
Lew, Erin D., Elizabeth A. Tindall, Colin Walsh, et al.. (2017). Abstract 4191: RXDX-106, a novel, selective and potent small molecule TAM (TYRO3, AXL, MER) inhibitor, demonstrates efficacy in TAM-driven tumors. Cancer Research. 77(13_Supplement). 4191–4191. 3 indexed citations
4.
Fourgeaud, Lawrence, Paqui G. Través, Yusuf Tufail, et al.. (2016). TAM receptors regulate multiple features of microglial physiology. Nature. 532(7598). 240–244. 423 indexed citations breakdown →
5.
Lew, Erin D., et al.. (2016). Immuno-oncologic efficacy of RXDX-106, a selective, TAM family small molecule kinase inhibitor. European Journal of Cancer. 69. S31–S31. 1 indexed citations
6.
Miner, Jonathan J., Brian P. Daniels, Bimmi Shrestha, et al.. (2015). The TAM receptor Mertk protects against neuroinvasive viral infection by maintaining blood-brain barrier integrity. Nature Medicine. 21(12). 1464–1472. 114 indexed citations
7.
Dransfield, Ian, et al.. (2015). Mer receptor tyrosine kinase mediates both tethering and phagocytosis of apoptotic cells. Cell Death and Disease. 6(2). e1646–e1646. 95 indexed citations
8.
Zagórska, Anna, Paqui G. Través, Erin D. Lew, Ian Dransfield, & Greg Lemke. (2014). Diversification of TAM receptor tyrosine kinase function. Nature Immunology. 15(10). 920–928. 322 indexed citations
9.
Lew, Erin D., Patrick Burrola, Irit Lax, et al.. (2014). Differential TAM receptor–ligand–phospholipid interactions delimit differential TAM bioactivities. eLife. 3. 210 indexed citations
10.
Bhattacharyya, Suchita, Anna Zagórska, Erin D. Lew, et al.. (2013). Enveloped Viruses Disable Innate Immune Responses in Dendritic Cells by Direct Activation of TAM Receptors. Cell Host & Microbe. 14(2). 136–147. 162 indexed citations
11.
Burstyn‐Cohen, Tal, et al.. (2012). Genetic Dissection of TAM Receptor-Ligand Interaction in Retinal Pigment Epithelial Cell Phagocytosis. Neuron. 76(6). 1123–1132. 135 indexed citations
12.
Meertens, Laurent, Xavier Carnec, Florence Guivel‐Benhassine, et al.. (2012). The TIM and TAM Families of Phosphatidylserine Receptors Mediate Dengue Virus Entry. Cell Host & Microbe. 12(4). 544–557. 393 indexed citations
13.
Lew, Erin D., Cristina M. Furdui, Karen S. Anderson, & Joseph Schlessinger. (2009). The Precise Sequence of FGF Receptor Autophosphorylation Is Kinetically Driven and Is Disrupted by Oncogenic Mutations. Science Signaling. 2(58). ra6–ra6. 116 indexed citations
14.
Bae, Jae Hyun, Erin D. Lew, Satoru Yuzawa, et al.. (2009). The Selectivity of Receptor Tyrosine Kinase Signaling Is Controlled by a Secondary SH2 Domain Binding Site. 138(3). 514–524. 3 indexed citations
15.
Bae, Jae Hyun, Erin D. Lew, Satoru Yuzawa, et al.. (2009). The Selectivity of Receptor Tyrosine Kinase Signaling Is Controlled by a Secondary SH2 Domain Binding Site. 138(33). 514–524. 1 indexed citations
16.
Bae, Jae Hyun, Erin D. Lew, Satoru Yuzawa, et al.. (2009). The Selectivity of Receptor Tyrosine Kinase Signaling Is Controlled by a Secondary SH2 Domain Binding Site. Cell. 138(3). 514–524. 119 indexed citations
17.
Shams, Imad, Edyta Rohmann, Veraragavan P. Eswarakumar, et al.. (2007). Lacrimo-Auriculo-Dento-Digital Syndrome Is Caused by Reduced Activity of the Fibroblast Growth Factor 10 (FGF10)-FGF Receptor 2 Signaling Pathway. Molecular and Cellular Biology. 27(19). 6903–6912. 53 indexed citations
18.
Lew, Erin D., Jae Hyun Bae, Edyta Rohmann, Bernd Wollnik, & Joseph Schlessinger. (2007). Structural basis for reduced FGFR2 activity in LADD syndrome: Implications for FGFR autoinhibition and activation. Proceedings of the National Academy of Sciences. 104(50). 19802–19807. 35 indexed citations
19.
Furdui, Cristina M., Erin D. Lew, Joseph Schlessinger, & Karen S. Anderson. (2006). Autophosphorylation of FGFR1 Kinase Is Mediated by a Sequential and Precisely Ordered Reaction. Molecular Cell. 21(5). 711–717. 183 indexed citations
20.
Eswarakumar, Veraragavan P., Erin D. Lew, Jae Hyun Bae, et al.. (2006). Attenuation of signaling pathways stimulated by pathologically activated FGF-receptor 2 mutants prevents craniosynostosis. Proceedings of the National Academy of Sciences. 103(49). 18603–18608. 86 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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