Tal Burstyn‐Cohen

3.3k total citations
37 papers, 2.6k citations indexed

About

Tal Burstyn‐Cohen is a scholar working on Immunology, Molecular Biology and Physiology. According to data from OpenAlex, Tal Burstyn‐Cohen has authored 37 papers receiving a total of 2.6k indexed citations (citations by other indexed papers that have themselves been cited), including 20 papers in Immunology, 14 papers in Molecular Biology and 8 papers in Physiology. Recurrent topics in Tal Burstyn‐Cohen's work include Phagocytosis and Immune Regulation (18 papers), Erythrocyte Function and Pathophysiology (6 papers) and Axon Guidance and Neuronal Signaling (5 papers). Tal Burstyn‐Cohen is often cited by papers focused on Phagocytosis and Immune Regulation (18 papers), Erythrocyte Function and Pathophysiology (6 papers) and Axon Guidance and Neuronal Signaling (5 papers). Tal Burstyn‐Cohen collaborates with scholars based in Israel, United States and Germany. Tal Burstyn‐Cohen's co-authors include Greg Lemke, Chaya Kalcheim, Avihu Klar, Avi Maimon, Patrick Burrola, Mary J. Heeb, Carla V. Rothlin, Dalit Sela‐Donenfeld, Ehud Cohen and Ayala Frumkin and has published in prestigious journals such as Cell, Proceedings of the National Academy of Sciences and Journal of Biological Chemistry.

In The Last Decade

Tal Burstyn‐Cohen

37 papers receiving 2.5k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Tal Burstyn‐Cohen Israel 25 1.1k 1.0k 554 370 285 37 2.6k
Fabrizio G. Mastronardi Canada 21 1.3k 1.2× 932 0.9× 527 1.0× 223 0.6× 172 0.6× 32 3.0k
Ermelinda Porpiglia United States 15 912 0.8× 444 0.4× 418 0.8× 342 0.9× 179 0.6× 21 1.8k
Stefan Frentzel Switzerland 30 1.1k 1.0× 531 0.5× 408 0.7× 587 1.6× 197 0.7× 67 2.9k
Claus Munck Petersen Denmark 33 1.7k 1.6× 428 0.4× 894 1.6× 1.0k 2.7× 1.0k 3.6× 69 3.6k
Katrin Busch Germany 14 1.4k 1.2× 1.9k 1.8× 313 0.6× 90 0.2× 372 1.3× 18 3.5k
Qingxian Lu United States 27 1.2k 1.1× 1.8k 1.8× 472 0.9× 255 0.7× 259 0.9× 53 3.3k
Michal Breker Israel 15 1.1k 1.0× 1.7k 1.6× 176 0.3× 112 0.3× 252 0.9× 20 3.0k
Gwendalyn D. King United States 34 1.2k 1.1× 588 0.6× 283 0.5× 183 0.5× 168 0.6× 57 2.9k
Lluı́s Espinosa Spain 32 2.7k 2.5× 743 0.7× 187 0.3× 145 0.4× 773 2.7× 85 4.0k
Pavel Urbánek Germany 17 1.9k 1.7× 879 0.8× 152 0.3× 136 0.4× 155 0.5× 28 3.0k

Countries citing papers authored by Tal Burstyn‐Cohen

Since Specialization
Citations

This map shows the geographic impact of Tal Burstyn‐Cohen's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Tal Burstyn‐Cohen with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Tal Burstyn‐Cohen more than expected).

Fields of papers citing papers by Tal Burstyn‐Cohen

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Tal Burstyn‐Cohen. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Tal Burstyn‐Cohen. The network helps show where Tal Burstyn‐Cohen may publish in the future.

Co-authorship network of co-authors of Tal Burstyn‐Cohen

This figure shows the co-authorship network connecting the top 25 collaborators of Tal Burstyn‐Cohen. A scholar is included among the top collaborators of Tal Burstyn‐Cohen based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Tal Burstyn‐Cohen. Tal Burstyn‐Cohen is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Burstyn‐Cohen, Tal, et al.. (2023). TAM receptors in phagocytosis: Beyond the mere internalization of particles. Immunological Reviews. 319(1). 7–26. 18 indexed citations
2.
Maimon, Avi, Kerem Ben‐Meir, Shivam Priya, et al.. (2021). Myeloid cell–derived PROS1 inhibits tumor metastasis by regulating inflammatory and immune responses via IL-10. Journal of Clinical Investigation. 131(10). 54 indexed citations
3.
Minson, Katherine A., John J. Tentler, Stacey M. Bagby, et al.. (2018). Inhibition of MERTK Promotes Suppression of Tumor Growth in BRAF Mutant and BRAF Wild-Type Melanoma. Molecular Cancer Therapeutics. 18(2). 278–288. 26 indexed citations
4.
Nassar, Maria, Yaara Tabib, Tal Capucha, et al.. (2018). Multiple Regulatory Levels of Growth Arrest-Specific 6 in Mucosal Immunity Against an Oral Pathogen. Frontiers in Immunology. 9. 1374–1374. 7 indexed citations
5.
Farago, Marganit, Merav Hecht, Reba Condiotti, et al.. (2017). Programming asynchronous replication in stem cells. Nature Structural & Molecular Biology. 24(12). 1132–1138. 6 indexed citations
6.
Burstyn‐Cohen, Tal. (2017). TAM receptor signaling in development. The International Journal of Developmental Biology. 61(3-4-5). 215–224. 24 indexed citations
7.
Abboud-Jarrous, Ghada, Shivam Priya, Avi Maimon, et al.. (2017). Protein S drives oral squamous cell carcinoma tumorigenicity through regulation of AXL. Oncotarget. 8(8). 13986–14002. 32 indexed citations
8.
Abboud-Jarrous, Ghada, et al.. (2017). Protein S Negatively Regulates Neural Stem Cell Self-Renewal through Bmi-1 Signaling. Frontiers in Molecular Neuroscience. 10. 124–124. 16 indexed citations
9.
Abboud-Jarrous, Ghada, et al.. (2016). Protein S Regulates Neural Stem Cell Quiescence and Neurogenesis. Stem Cells. 35(3). 679–693. 29 indexed citations
10.
Ben‐Gedalya, Tziona, Lorna Moll, Michal Bejerano‐Sagie, et al.. (2015). Alzheimer's disease‐causing proline substitutions lead to presenilin 1 aggregation and malfunction. The EMBO Journal. 34(22). 2820–2839. 26 indexed citations
11.
Capucha, Tal, Gabriel Mizraji, Ronnie Blecher‐Gonen, et al.. (2015). Distinct Murine Mucosal Langerhans Cell Subsets Develop from Pre-dendritic Cells and Monocytes. Immunity. 43(2). 369–381. 70 indexed citations
12.
Silva, Eugenio Antonio Carrera, Pamela Chan, Leonel Joannas, et al.. (2013). T Cell-Derived Protein S Engages TAM Receptor Signaling in Dendritic Cells to Control the Magnitude of the Immune Response. Immunity. 39(1). 160–170. 143 indexed citations
13.
Burstyn‐Cohen, Tal, et al.. (2012). Genetic Dissection of TAM Receptor-Ligand Interaction in Retinal Pigment Epithelial Cell Phagocytosis. Neuron. 76(6). 1123–1132. 135 indexed citations
14.
Jaouni, Tareq, Edward Averbukh, Tal Burstyn‐Cohen, et al.. (2012). Association of Pattern Dystrophy With an HTRA1 Single-Nucleotide Polymorphism. Archives of Ophthalmology. 130(8). 987–91. 16 indexed citations
15.
Grunin, Michelle, Tal Burstyn‐Cohen, Shira Hagbi-Levi, Amnon Peled, & Itay Chowers. (2012). Chemokine Receptor Expression in Peripheral Blood Monocytes from Patients with Neovascular Age-Related Macular Degeneration. Investigative Ophthalmology & Visual Science. 53(9). 5292–5292. 54 indexed citations
16.
Lemke, Greg & Tal Burstyn‐Cohen. (2010). TAM receptors and the clearance of apoptotic cells. Annals of the New York Academy of Sciences. 1209(1). 23–29. 185 indexed citations
17.
Burstyn‐Cohen, Tal, Mary J. Heeb, & Greg Lemke. (2009). Lack of Protein S in mice causes embryonic lethal coagulopathy and vascular dysgenesis. Journal of Clinical Investigation. 119(10). 2942–2953. 134 indexed citations
18.
Prasad, Dipti, Carla V. Rothlin, Patrick Burrola, et al.. (2006). TAM receptor function in the retinal pigment epithelium. Molecular and Cellular Neuroscience. 33(1). 96–108. 212 indexed citations
19.
Kalcheim, Chaya & Tal Burstyn‐Cohen. (2005). Early stages of neural crest ontogeny: formation and regulation of cell delamination. The International Journal of Developmental Biology. 49(2-3). 105–116. 42 indexed citations
20.
Burstyn‐Cohen, Tal & Chaya Kalcheim. (2002). Association between the Cell Cycle and Neural Crest Delamination through Specific Regulation of G1/S Transition. Developmental Cell. 3(3). 383–395. 106 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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