Enzo Cohen
Impact in
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- Neurogenetic and Muscular Disorders Research
- Genomics and Rare Diseases
Papers in
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- Mitochondrial Function and Pathology 1
- RNA regulation and disease 1
- Nuclear Structure and Function 1
- RNA Research and Splicing 1
- ATP Synthase and ATPases Research 1
- RNA and protein synthesis mechanisms 1
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- Growth Hormone and Insulin-like Growth Factors 1
- Co-authors
- Gisèle Bonne (2 shared papers)François Rivier (1 shared paper)Dalil Hamroun (1 shared paper)Rabah Ben Yaou (1 shared paper)Francesco Battaglia (1 shared paper)Louise Benarroch (1 shared paper)A. Bertrand (1 shared paper)G. Rodier (1 shared paper)
- Journals
- Journal of Clinical Medicine (1 paper)Neuromuscular Disorders (1 paper)PubMed (1 paper)
- Partner nations
- France
In The Last Decade
Enzo Cohen
4 papers receiving 44 citations
Peers
Comparison fields: 5 of 18
- Genetics 10
- Neurology 6
- Molecular Biology 28
- Cardiology and Cardiovascular Medicine 8
- Cellular and Molecular Neuroscience 7
Countries citing papers authored by Enzo Cohen
This map shows the geographic impact of Enzo Cohen's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Enzo Cohen with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Enzo Cohen more than expected).
Fields of papers citing papers by Enzo Cohen
This network shows the impact of papers produced by Enzo Cohen. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Enzo Cohen. The network helps show where Enzo Cohen may publish in the future.
Co-authors
The 25 scholars most cited alongside Enzo Cohen, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2021 | 32 | |
| 2 | 2021 | 7 | |
| 3 | [Atypical phenotype of Fabry disease. Evidence of a new mutation]. | 2000 | 4 |
| 4 | Contribution of GHR and IGFALS Mutations to Growth Hormone Resistance - Identification of New Variants and Impact on the Inheritance Pattern | 2016 | 1 |
About Enzo Cohen
Enzo Cohen is a scholar working on Molecular Biology, Endocrinology, Diabetes and Metabolism, Physiology, Infectious Diseases and Organic Chemistry, having authored 4 papers that have together received 44 indexed citations. Recurring topics across this work include Lysosomal Storage Disorders Research (1 paper), Mitochondrial Function and Pathology (1 paper), RNA regulation and disease (1 paper), Growth Hormone and Insulin-like Growth Factors (1 paper), Nuclear Structure and Function (1 paper), RNA Research and Splicing (1 paper), ATP Synthase and ATPases Research (1 paper) and RNA and protein synthesis mechanisms (1 paper). The work is most often cited by research in Genetics (10 citations), Neurology (6 citations), Molecular Biology (28 citations), Cardiology and Cardiovascular Medicine (8 citations) and Cellular and Molecular Neuroscience (7 citations). Enzo Cohen has collaborated with scholars based in France. Frequent co-authors include Gisèle Bonne, François Rivier, Dalil Hamroun, Rabah Ben Yaou, Francesco Battaglia, Louise Benarroch, A. Bertrand, G. Rodier, Clotilde Boulay and António Atalaia. Their work appears in journals such as Journal of Clinical Medicine, Neuromuscular Disorders and PubMed.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.