Emily M. Schmidt

3.7k total citations · 2 hit papers
13 papers, 2.9k citations indexed

About

Emily M. Schmidt is a scholar working on Immunology, Genetics and Oncology. According to data from OpenAlex, Emily M. Schmidt has authored 13 papers receiving a total of 2.9k indexed citations (citations by other indexed papers that have themselves been cited), including 10 papers in Immunology, 6 papers in Genetics and 3 papers in Oncology. Recurrent topics in Emily M. Schmidt's work include T-cell and B-cell Immunology (7 papers), Immune Cell Function and Interaction (7 papers) and Diabetes and associated disorders (5 papers). Emily M. Schmidt is often cited by papers focused on T-cell and B-cell Immunology (7 papers), Immune Cell Function and Interaction (7 papers) and Diabetes and associated disorders (5 papers). Emily M. Schmidt collaborates with scholars based in United Kingdom, United States and Singapore. Emily M. Schmidt's co-authors include Lucy S. K. Walker, David M. Sansom, Omar Qureshi, Tie Zheng Hou, Kesley Attridge, Claire N. Manzotti, Zoe Briggs, Graham Anderson, Kyoko Nakamura and Clare E. Futter and has published in prestigious journals such as Nature, Science and Proceedings of the National Academy of Sciences.

In The Last Decade

Emily M. Schmidt

13 papers receiving 2.8k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Trans-Endocytosis of CD80 and CD86: A Molecular Basis for the Cell-Extrinsic Function of CTLA-4

2011 1.3k citations Omar Qureshi, Yong Zheng et al. Science profile →
Histone deacetylase inhibitors arrest polyglutamine-dependent neurodegeneration in Drosophila

2001 964 citations Joan S. Steffan, László Bodai et al. Nature profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Emily M. Schmidt United Kingdom	10	1.5k	1.0k	796	692	381	13	2.9k
Liyin Zhu United States	17	1.6k 1.1×	1.1k 1.0×	1.0k 1.3×	141 0.2×	278 0.7×	21	3.3k
Dara Kallop United States	13	427 0.3×	1.2k 1.1×	579 0.7×	278 0.4×	150 0.4×	13	2.1k
Tomoo Ueno Japan	20	1.5k 1.0×	541 0.5×	512 0.6×	119 0.2×	197 0.5×	23	2.2k
Wei-Hsien Ho United States	11	688 0.4×	494 0.5×	435 0.5×	410 0.6×	123 0.3×	16	1.7k
Izumi Negishi Japan	16	2.2k 1.4×	1.7k 1.7×	729 0.9×	226 0.3×	278 0.7×	34	3.8k
Ben W. Dulken United States	6	1.4k 0.9×	1.1k 1.1×	933 1.2×	107 0.2×	87 0.2×	10	2.9k
Dmitri Lodygin Germany	12	555 0.4×	1.9k 1.8×	608 0.8×	122 0.2×	89 0.2×	15	3.1k
Haishan Lin China	19	1.8k 1.1×	865 0.8×	488 0.6×	134 0.2×	120 0.3×	41	2.8k
David Davidson Canada	18	768 0.5×	842 0.8×	541 0.7×	114 0.2×	131 0.3×	29	2.0k
Gerd Munzert Germany	28	436 0.3×	1.4k 1.4×	1.1k 1.3×	146 0.2×	104 0.3×	63	3.1k

Countries citing papers authored by Emily M. Schmidt

Since Specialization

Citations

This map shows the geographic impact of Emily M. Schmidt's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Emily M. Schmidt with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Emily M. Schmidt more than expected).

Fields of papers citing papers by Emily M. Schmidt

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Emily M. Schmidt. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Emily M. Schmidt. The network helps show where Emily M. Schmidt may publish in the future.

Co-authorship network of co-authors of Emily M. Schmidt

This figure shows the co-authorship network connecting the top 25 collaborators of Emily M. Schmidt. A scholar is included among the top collaborators of Emily M. Schmidt based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Emily M. Schmidt. Emily M. Schmidt is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

Sort: Min cites: Since: Top N: Style:

13 of 13 papers shown

Schmidt, Emily M., Katerina Mardilovich, Natalie Bath, et al.. (2021). 373 Enhancement of TCR-engineered T-cells targeting MAGE-A4 antigen by co-expression of CD8α and inhibition of AKT signaling during ex vivo T-cell expansion. Regular and Young Investigator Award Abstracts. A401–A401. 3 indexed citations

Mardilovich, Katerina, Lili Wang, Gareth Betts, et al.. (2020). 95 Inhibition of AKT signaling during expansion of TCR-engineered T-cells from patient leukocyte material generates SPEAR T-cells with enhanced functional potential in vitro. Regular and Young Investigator Award Abstracts. A60.1–A60. 1 indexed citations

Schmidt, Emily M., Fiona E. McCann, Marc Feldmann, et al.. (2014). TNFR2 increases the sensitivity of ligand-induced activation of the p38 MAPK and NF-κB pathways and signals TRAF2 protein degradation in macrophages. Cellular Signalling. 26(4). 683–690. 31 indexed citations

Kennedy, Alan, Emily M. Schmidt, Adam P. Cribbs, et al.. (2014). A novel upstream enhancer of FOXP3, sensitive to methylation‐induced silencing, exhibits dysregulated methylation in rheumatoid arthritis Treg cells. European Journal of Immunology. 44(10). 2968–2978. 47 indexed citations

Wang, Chun Jing, Frank Heuts, Vitalijs Ovcinnikovs, et al.. (2014). CTLA-4 controls follicular helper T-cell differentiation by regulating the strength of CD28 engagement. Proceedings of the National Academy of Sciences. 112(2). 524–529. 137 indexed citations

Schmidt, Emily M., Marie Davies, Prafull Mistry, et al.. (2013). Selective Blockade of Tumor Necrosis Factor Receptor I Inhibits Proinflammatory Cytokine and Chemokine Production in Human Rheumatoid Arthritis Synovial Membrane Cell Cultures. Arthritis & Rheumatism. 65(9). 2262–2273. 32 indexed citations

Wang, Chun Jing, Rupert Kenefeck, Lukasz Wardzinski, et al.. (2012). Cutting Edge: Cell-Extrinsic Immune Regulation by CTLA-4 Expressed on Conventional T Cells. The Journal of Immunology. 189(3). 1118–1122. 75 indexed citations

Qureshi, Omar, Yong Zheng, Kyoko Nakamura, et al.. (2011). Trans-Endocytosis of CD80 and CD86: A Molecular Basis for the Cell-Extrinsic Function of CTLA-4. Science. 332(6029). 600–603. 1323 indexed citations breakdown →

Wang, Chun Jing, Emily M. Schmidt, Kesley Attridge, et al.. (2011). Immune regulation by CTLA‐4—relevance to autoimmune diabetes in a transgenic mouse model. Diabetes/Metabolism Research and Reviews. 27(8). 946–950. 5 indexed citations

10.

Wang, Chun Jing, Jayne L. Chamberlain, Kesley Attridge, et al.. (2010). B1 Cells Promote Pancreas Infiltration by Autoreactive T Cells. The Journal of Immunology. 185(5). 2800–2807. 34 indexed citations

11.

Schmidt, Emily M., Chun Jing Wang, Louise E. Clough, et al.. (2009). CTLA-4 Controls Regulatory T Cell Peripheral Homeostasis and Is Required for Suppression of Pancreatic Islet Autoimmunity. The Journal of Immunology. 182(1). 274–282. 125 indexed citations

12.

Clough, Louise E., Chun Jing Wang, Emily M. Schmidt, et al.. (2008). Release from Regulatory T Cell-Mediated Suppression during the Onset of Tissue-Specific Autoimmunity Is Associated with Elevated IL-21. The Journal of Immunology. 180(8). 5393–5401. 96 indexed citations

13.

Steffan, Joan S., László Bodai, Judit Pallos, et al.. (2001). Histone deacetylase inhibitors arrest polyglutamine-dependent neurodegeneration in Drosophila. Nature. 413(6857). 739–743. 964 indexed citations breakdown →

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

Explore authors with similar magnitude of impact