Émeline Bon

409 total citations
9 papers, 320 citations indexed

About

Émeline Bon is a scholar working on Molecular Biology, Cell Biology and Genetics. According to data from OpenAlex, Émeline Bon has authored 9 papers receiving a total of 320 indexed citations (citations by other indexed papers that have themselves been cited), including 9 papers in Molecular Biology, 2 papers in Cell Biology and 1 paper in Genetics. Recurrent topics in Émeline Bon's work include Ion channel regulation and function (4 papers), Nicotinic Acetylcholine Receptors Study (3 papers) and Peroxisome Proliferator-Activated Receptors (2 papers). Émeline Bon is often cited by papers focused on Ion channel regulation and function (4 papers), Nicotinic Acetylcholine Receptors Study (3 papers) and Peroxisome Proliferator-Activated Receptors (2 papers). Émeline Bon collaborates with scholars based in France, Spain and Switzerland. Émeline Bon's co-authors include Sébastien Roger, Pierre Besson, Stéphan Chevalier, Virginie Driffort, Philippe Bougnoux, Séverine Marionneau‐Lambot, Thibauld Oullier, Ludovic Gillet, V. Joulin and Christine Collin and has published in prestigious journals such as Nature Communications, Scientific Reports and International Journal of Molecular Sciences.

In The Last Decade

Émeline Bon

9 papers receiving 319 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Émeline Bon France	7	261	67	64	35	30	9	320
Virginie Driffort France	5	330 1.3×	84 1.3×	70 1.1×	52 1.5×	33 1.1×	5	404
Stéphanie Chadet France	10	154 0.6×	24 0.4×	29 0.5×	22 0.6×	29 1.0×	24	332
Galdo Bustos Chile	8	345 1.3×	54 0.8×	61 1.0×	29 0.8×	16 0.5×	10	445
Clara Serrano‐Novillo Spain	7	285 1.1×	73 1.1×	19 0.3×	17 0.5×	22 0.7×	10	358
Robert B. Bentham United Kingdom	7	245 0.9×	29 0.4×	81 1.3×	23 0.7×	27 0.9×	10	286
Xiahui Tan Australia	11	169 0.6×	42 0.6×	25 0.4×	73 2.1×	25 0.8×	13	361
Yiping Liu China	13	283 1.1×	12 0.2×	116 1.8×	15 0.4×	101 3.4×	21	432
Yening Jin China	5	231 0.9×	50 0.7×	85 1.3×	159 4.5×	27 0.9×	9	354
Otto Lindemann Germany	10	156 0.6×	22 0.3×	16 0.3×	147 4.2×	17 0.6×	11	332
Iván Meneses‐Morales Mexico	11	180 0.7×	13 0.2×	44 0.7×	9 0.3×	36 1.2×	19	294

Countries citing papers authored by Émeline Bon

Since Specialization

Citations

This map shows the geographic impact of Émeline Bon's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Émeline Bon with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Émeline Bon more than expected).

Fields of papers citing papers by Émeline Bon

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Émeline Bon. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Émeline Bon. The network helps show where Émeline Bon may publish in the future.

Co-authorship network of co-authors of Émeline Bon

This figure shows the co-authorship network connecting the top 25 collaborators of Émeline Bon. A scholar is included among the top collaborators of Émeline Bon based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Émeline Bon. Émeline Bon is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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9 of 9 papers shown

Bon, Émeline, et al.. (2024). Understanding the Role of the SMN Complex Component GEMIN5 and Its Functional Relationship with Demethylase KDM6B in the Flunarizine-Mediated Neuroprotection of Motor Neuron Disease Spinal Muscular Atrophy. International Journal of Molecular Sciences. 25(18). 10039–10039. 2 indexed citations

Chadet, Stéphanie, Émeline Bon, Roxane Lemoine, et al.. (2020). Rock inhibition promotes NaV1.5 sodium channel-dependent SW620 colon cancer cell invasiveness. Scientific Reports. 10(1). 13350–13350. 11 indexed citations

Greco, Maria Raffaella, Émeline Bon, Rosa Rubino, et al.. (2018). Phosphorylation of NHERF1 S279 and S301 differentially regulates breast cancer cell phenotype and metastatic organotropism. Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease. 1865(1). 26–37. 8 indexed citations

Bon, Émeline, Lucie Brisson, Stéphan Chevalier, Pierre Besson, & Sébastien Roger. (2017). Navβ4 : un-suppresseur de métastases et un nouveau biomarqueur des cancers agressifs. médecine/sciences. 33(6–7). 596–599. 2 indexed citations

Bon, Émeline, Virginie Driffort, Carlos Martínez, et al.. (2016). SCN4B acts as a metastasis-suppressor gene preventing hyperactivation of cell migration in breast cancer. Nature Communications. 7(1). 13648–13648. 53 indexed citations

Besson, Pierre, et al.. (2015). How do voltage-gated sodium channels enhance migration and invasiveness in cancer cells?. Biochimica et Biophysica Acta (BBA) - Biomembranes. 1848(10). 2493–2501. 78 indexed citations

Driffort, Virginie, Ludovic Gillet, Émeline Bon, et al.. (2014). Ranolazine inhibits NaV1.5-mediated breast cancer cell invasiveness and lung colonization. Molecular Cancer. 13(1). 264–264. 113 indexed citations

Bon, Émeline, Ludovic Gillet, Günther Weber, et al.. (2014). Suppression of PPARβ, and DHA treatment, inhibit NaV1.5 and NHE-1 pro-invasive activities. Pflügers Archiv - European Journal of Physiology. 467(6). 1249–1259. 25 indexed citations

Bon, Émeline, Karine Mahéo, Caroline Goupille, et al.. (2013). PPARβ mRNA expression, reduced by n−3 PUFA diet in mammary tumor, controls breast cancer cell growth. Biochimica et Biophysica Acta (BBA) - Molecular and Cell Biology of Lipids. 1831(11). 1618–1625. 28 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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