Elsje G. Otten
- Molecular Biology top 10%
- Epidemiology top 5%
- Cell Biology top 5%
- Physiology top 10%
- Immunology
- Co-authors
- Viktor I. KorolchukYoana Rabanal‐RuizKeith B. BoyleFelix RandowBernadette CarrollSovan SarkarDorothea MaetzelBalaji Santhanam
- Topics
- Autophagy in Disease and Therapy (9 papers)Toxoplasma gondii Research Studies (3 papers)Mitochondrial Function and Pathology (2 papers)
- Cited by
- PhysiologyAgingEpidemiology
- Partner nations
- United KingdomUnited StatesAustria
In The Last Decade
Elsje G. Otten
15 papers receiving 1.7k citations
Hit Papers
Peers
Comparison fields: 5 of 96
- Molecular Biology 946
- Epidemiology 861
- Cell Biology 312
- Physiology 270
- Immunology 186
Countries citing papers authored by Elsje G. Otten
This map shows the geographic impact of Elsje G. Otten's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Elsje G. Otten with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Elsje G. Otten more than expected).
Fields of papers citing papers by Elsje G. Otten
This network shows the impact of papers produced by Elsje G. Otten. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Elsje G. Otten. The network helps show where Elsje G. Otten may publish in the future.
Co-authorship network of co-authors of Elsje G. Otten
This figure shows the co-authorship network connecting the top 25 collaborators of Elsje G. Otten. A scholar is included among the top collaborators of Elsje G. Otten based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Elsje G. Otten. Elsje G. Otten is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 1 | |
| 2 | 5 | |
| 3 | 4 | |
| 4 | 40 | |
| 5 | Ubiquitylation of lipopolysaccharide by RNF213 during bacterial infectionbreakdown → | 229 |
| 6 | 2 | |
| 7 | The Cargo Receptor NDP52 Initiates Selective Autophagy by Recruiting the ULK Complex to Cytosol-Invading Bacteriabreakdown → | 221 |
| 8 | 147 | |
| 9 | mTORC1 as the main gateway to autophagybreakdown → | 431 |
| 10 | 52 | |
| 11 | 144 | |
| 12 | 37 | |
| 13 | 200 | |
| 14 | 121 | |
| 15 | 119 |
About Elsje G. Otten
Elsje G. Otten is a scholar working on Aging, Parasitology and Epidemiology, having authored 15 papers that have together received 1.8k indexed citations. Recurring topics across this work include Autophagy in Disease and Therapy (9 papers), Toxoplasma gondii Research Studies (3 papers) and Mitochondrial Function and Pathology (2 papers). The work is most often cited by research in Physiology (144 citations), Aging (51 citations) and Epidemiology (861 citations). Elsje G. Otten has collaborated with scholars based in United Kingdom, United States and Austria. Frequent co-authors include Viktor I. Korolchuk, Yoana Rabanal‐Ruiz, Keith B. Boyle, Felix Randow, Bernadette Carroll, Sovan Sarkar, Dorothea Maetzel, Balaji Santhanam, Emma Werner and Ana Crespillo-Casado. Their work appears in journals such as Nature, Nature Communications and The EMBO Journal.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.