Joseph Collin
- Molecular Biology top 10%
- Ophthalmology top 2%
- Cellular and Molecular Neuroscience top 5%
- Radiology, Nuclear Medicine and Imaging top 10%
- Cancer Research
- Co-authors
- Majlinda LakoDarin ZertiBirthe DorgauLyle ArmstrongDavid SteelRachel QueenCarla MelloughEvelyne Sernagor
- Topics
- Retinal Development and Disorders (24 papers)CRISPR and Genetic Engineering (10 papers)Photoreceptor and optogenetics research (8 papers)
- Partner nations
- United KingdomSaudi ArabiaSpain
In The Last Decade
Joseph Collin
35 papers receiving 1.3k citations
Peers
Comparison fields: 5 of 78
- Molecular Biology 1.0k
- Ophthalmology 347
- Cellular and Molecular Neuroscience 305
- Radiology, Nuclear Medicine and Imaging 186
- Cancer Research 96
Countries citing papers authored by Joseph Collin
This map shows the geographic impact of Joseph Collin's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Joseph Collin with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Joseph Collin more than expected).
Fields of papers citing papers by Joseph Collin
This network shows the impact of papers produced by Joseph Collin. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Joseph Collin. The network helps show where Joseph Collin may publish in the future.
Co-authorship network of co-authors of Joseph Collin
This figure shows the co-authorship network connecting the top 25 collaborators of Joseph Collin. A scholar is included among the top collaborators of Joseph Collin based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Joseph Collin. Joseph Collin is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 3 | |
| 2 | 16 | |
| 3 | 13 | |
| 4 | 21 | |
| 5 | 19 | |
| 6 | 22 | |
| 7 | 39 | |
| 8 | 27 | |
| 9 | 101 | |
| 10 | 65 | |
| 11 | 65 | |
| 12 | Using hESC-derived retinal organoids to investigate the transcriptional profile of emerging photoreceptors | 1 |
| 13 | 76 | |
| 14 | 38 | |
| 15 | 38 | |
| 16 | 22 | |
| 17 | 34 | |
| 18 | 52 | |
| 19 | 114 | |
| 20 | 13 |
About Joseph Collin
Joseph Collin is a scholar working on Ophthalmology, Cellular and Molecular Neuroscience and Molecular Biology, having authored 35 papers that have together received 1.3k indexed citations. Recurring topics across this work include Retinal Development and Disorders (24 papers), CRISPR and Genetic Engineering (10 papers) and Photoreceptor and optogenetics research (8 papers). The work is most often cited by research in Ophthalmology (347 citations), Cellular and Molecular Neuroscience (305 citations) and Molecular Biology (1.0k citations). Joseph Collin has collaborated with scholars based in United Kingdom, Saudi Arabia and Spain. Frequent co-authors include Majlinda Lako, Darin Zerti, Birthe Dorgau, Lyle Armstrong, David Steel, Rachel Queen, Carla Mellough, Evelyne Sernagor, Rafiqul Hussain and Jonathan Coxhead. Their work appears in journals such as Nature Communications, Development and Scientific Reports.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.