Edward S. Ruthazer
- Cellular and Molecular Neuroscience top 0.5%
- Molecular Biology top 5%
- Cognitive Neuroscience top 2%
- Developmental Neuroscience top 1%
- Cell Biology top 5%
- Co-authors
- Hollis T. ClineMichael P. StrykerKurt HaasWun Chey SinMari SildAnne SchohlColin J. AkermanMarion R. Van Horn
- Topics
- Neuroscience and Neuropharmacology Research (43 papers)Retinal Development and Disorders (24 papers)Axon Guidance and Neuronal Signaling (17 papers)
- Partner nations
- CanadaUnited StatesJapan
In The Last Decade
Edward S. Ruthazer
71 papers receiving 3.3k citations
Peers
Comparison fields: 5 of 116
- Cellular and Molecular Neuroscience 2.2k
- Molecular Biology 1.3k
- Cognitive Neuroscience 940
- Developmental Neuroscience 527
- Cell Biology 352
Countries citing papers authored by Edward S. Ruthazer
This map shows the geographic impact of Edward S. Ruthazer's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Edward S. Ruthazer with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Edward S. Ruthazer more than expected).
Fields of papers citing papers by Edward S. Ruthazer
This network shows the impact of papers produced by Edward S. Ruthazer. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Edward S. Ruthazer. The network helps show where Edward S. Ruthazer may publish in the future.
Co-authorship network of co-authors of Edward S. Ruthazer
This figure shows the co-authorship network connecting the top 25 collaborators of Edward S. Ruthazer. A scholar is included among the top collaborators of Edward S. Ruthazer based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Edward S. Ruthazer. Edward S. Ruthazer is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 2 | |
| 2 | 1 | |
| 3 | 7 | |
| 4 | 76 | |
| 5 | 9 | |
| 6 | 64 | |
| 7 | 30 | |
| 8 | 13 | |
| 9 | 54 | |
| 10 | 35 | |
| 11 | 76 | |
| 12 | 45 | |
| 13 | 51 | |
| 14 | 31 | |
| 15 | 51 | |
| 16 | 31 | |
| 17 | 133 | |
| 18 | 156 | |
| 19 | 13 | |
| 20 | 89 |
About Edward S. Ruthazer
Edward S. Ruthazer is a scholar working on Developmental Neuroscience, Cellular and Molecular Neuroscience and Biophysics, having authored 72 papers that have together received 3.4k indexed citations. Recurring topics across this work include Neuroscience and Neuropharmacology Research (43 papers), Retinal Development and Disorders (24 papers) and Axon Guidance and Neuronal Signaling (17 papers). The work is most often cited by research in Developmental Neuroscience (527 citations), Cellular and Molecular Neuroscience (2.2k citations) and Cognitive Neuroscience (940 citations). Edward S. Ruthazer has collaborated with scholars based in Canada, United States and Japan. Frequent co-authors include Hollis T. Cline, Michael P. Stryker, Kurt Haas, Wun Chey Sin, Mari Sild, Anne Schohl, Colin J. Akerman, Marion R. Van Horn, Tony K.Y. Lim and Jianli Li. Their work appears in journals such as Nature, Science and Proceedings of the National Academy of Sciences.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.