E.B. Mawer

7.1k total citations
131 papers, 5.3k citations indexed

About

E.B. Mawer is a scholar working on Pathology and Forensic Medicine, Genetics and Molecular Biology. According to data from OpenAlex, E.B. Mawer has authored 131 papers receiving a total of 5.3k indexed citations (citations by other indexed papers that have themselves been cited), including 83 papers in Pathology and Forensic Medicine, 25 papers in Genetics and 22 papers in Molecular Biology. Recurrent topics in E.B. Mawer's work include Vitamin D Research Studies (82 papers), Bone health and treatments (19 papers) and Parathyroid Disorders and Treatments (17 papers). E.B. Mawer is often cited by papers focused on Vitamin D Research Studies (82 papers), Bone health and treatments (19 papers) and Parathyroid Disorders and Treatments (17 papers). E.B. Mawer collaborates with scholars based in United Kingdom, United States and France. E.B. Mawer's co-authors include Michael Davies, G. A. Lumb, Sarah Stanbury, Peter Selby, Jacqueline Berry, Anthony J. Freemont, Nigel Bundred, M.E. Hayes, Ann E. Canfield and A.P. Mee and has published in prestigious journals such as Nature, The Lancet and Circulation Research.

In The Last Decade

E.B. Mawer

131 papers receiving 4.9k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
E.B. Mawer United Kingdom 42 2.8k 1.1k 952 932 812 131 5.3k
J. L. H. O’Riordan United States 34 1.3k 0.5× 476 0.4× 940 1.0× 626 0.7× 494 0.6× 102 4.0k
M Garabédian France 31 1.8k 0.7× 644 0.6× 557 0.6× 572 0.6× 589 0.7× 109 3.4k
Michèle Garabédian France 32 1.1k 0.4× 681 0.6× 924 1.0× 686 0.7× 358 0.4× 73 3.4k
P. Rødbro Denmark 37 1.0k 0.4× 1.7k 1.6× 718 0.8× 377 0.4× 636 0.8× 124 4.3k
Sol Epstein United States 35 1.2k 0.4× 2.0k 1.8× 588 0.6× 437 0.5× 498 0.6× 104 4.4k
Klaus Ølgaard Denmark 38 1.4k 0.5× 857 0.8× 3.6k 3.7× 1.1k 1.2× 496 0.6× 213 6.5k
Adriana Dusso United States 42 2.9k 1.0× 570 0.5× 3.0k 3.1× 1.3k 1.4× 468 0.6× 106 6.0k
M. C. Chapuy France 28 1.6k 0.6× 3.2k 3.0× 482 0.5× 1.0k 1.1× 541 0.7× 51 5.3k
Subhash C. Kukreja United States 32 985 0.4× 974 0.9× 393 0.4× 398 0.4× 511 0.6× 93 3.4k
Harald Dobnig Austria 41 3.0k 1.1× 2.8k 2.6× 692 0.7× 1.6k 1.7× 543 0.7× 88 6.7k

Countries citing papers authored by E.B. Mawer

Since Specialization
Citations

This map shows the geographic impact of E.B. Mawer's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by E.B. Mawer with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites E.B. Mawer more than expected).

Fields of papers citing papers by E.B. Mawer

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by E.B. Mawer. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by E.B. Mawer. The network helps show where E.B. Mawer may publish in the future.

Co-authorship network of co-authors of E.B. Mawer

This figure shows the co-authorship network connecting the top 25 collaborators of E.B. Mawer. A scholar is included among the top collaborators of E.B. Mawer based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with E.B. Mawer. E.B. Mawer is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Mawer, E.B. & Michael Davies. (2001). Vitamin D Nutrition and Bone Disease in Adults. Reviews in Endocrine and Metabolic Disorders. 2(2). 153–164. 54 indexed citations
2.
Tetlow, L. C., S J Smith, E.B. Mawer, & David E. Woolley. (1999). Vitamin D receptors in the rheumatoid lesion: expression by chondrocytes, macrophages, and synoviocytes. Annals of the Rheumatic Diseases. 58(2). 118–121. 78 indexed citations
3.
Haworth, Charles, Peter Selby, A.K. Webb, et al.. (1999). Low bone mineral density in adults with cystic fibrosis. Thorax. 54(11). 961–967. 171 indexed citations
4.
Smith, S J, M.E. Hayes, Peter Selby, & E.B. Mawer. (1999). Autocrine control of vitamin D metabolism in synovial cells from arthritic patients. Annals of the Rheumatic Diseases. 58(6). 372–378. 14 indexed citations
5.
Haworth, Charles, Anthony J. Freemont, A.K. Webb, et al.. (1999). Hip fracture and bone histomorphometry in a young adult with cystic fibrosis. European Respiratory Journal. 14(2). 478–479. 18 indexed citations
6.
Farquharson, Colin, J. L. Berry, E.B. Mawer, Elaine Seawright, & C. C. Whitehead. (1998). Ascorbic acid-induced chondrocyte terminal differentiation. European Journal of Cell Biology. 76(2). 1 indexed citations
7.
Fraser, W.D., Benjamin H. Durham, J. L. Berry, & E.B. Mawer. (1997). Measurement of Plasma 1,25 Dihydroxyvitamin D Using a Novel Immunoextraction Technique and Immunoassay with Iodine Labelled Vitamin D Tracer. Annals of Clinical Biochemistry International Journal of Laboratory Medicine. 34(6). 632–637. 59 indexed citations
8.
Mawer, E.B., J. Walls, Anthony Howell, et al.. (1997). Serum 1,25-Dihydroxyvitamin D May Be Related Inversely to Disease Activity in Breast Cancer Patients with Bone Metastases1. The Journal of Clinical Endocrinology & Metabolism. 82(1). 118–122. 73 indexed citations
9.
Davies, Michael, et al.. (1997). Increased Catabolism of 25-Hydroxyvitamin D in Patients with Partial Gastrectomy and Elevated 1,25-Dihydroxyvitamin D Levels. Implications for Metabolic Bone Disease1. The Journal of Clinical Endocrinology & Metabolism. 82(1). 209–212. 82 indexed citations
10.
Mee, A.P., et al.. (1996). Novel and sensitive detection systems for the vitamin D receptor — in situ-reverse transcriptase-polymerase chain reaction and immunogold cytochemistry. Journal of Molecular Endocrinology. 16(2). 183–195. 19 indexed citations
11.
Farquharson, Colin, J. L. Berry, E.B. Mawer, Elaine Seawright, & C. C. Whitehead. (1995). Regulators of chondrocyte differentiation in tibial dyschondroplasia. Bone. 17(3). 2 indexed citations
12.
13.
Kumar, Sudhesh, A O Olukoga, Caroline Gordon, et al.. (1994). Impaired glucose tolerance and insulin insensitivity in primary hyperparathyroidism. Clinical Endocrinology. 40(1). 47–53. 93 indexed citations
14.
Clements, M. R., Michael Davies, M.E. Hayes, et al.. (1992). The role of 1,25‐dihydroxyvitamin D in the mechanism of acquired vitamin D deficiency. Clinical Endocrinology. 37(1). 17–27. 148 indexed citations
15.
16.
Davies, Michael, et al.. (1990). Synthesis of 1,25-dihydroxyvitamin D3 by spleen cells isolated from two patients with myelofibrosis and a normal subject. Journal of Steroid Biochemistry. 35(2). 191–194. 3 indexed citations
17.
Shany, Shraga, et al.. (1984). Feto-maternal relationships between vitamin D metabolites in Israeli Bedouins and Jews. American Journal of Clinical Nutrition. 40(6). 1290–1294. 11 indexed citations
18.
Stephens, W P, et al.. (1983). Vitamin D state of Asians living in Pakistan.. BMJ. 286(6360). 182–184. 32 indexed citations
19.
Stamp, T. C. B., et al.. (1983). Hypercalcaemic sarcoidosis in hypoparathyroidism.. BMJ. 286(6367). 764–765. 9 indexed citations
20.
Mawer, E.B., et al.. (1972). Rickets in Glasgow Pakistanis.. BMJ. 3(5819). 177.4–178. 4 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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