E. Douglas Lewandowski

5.7k total citations · 2 hit papers
81 papers, 3.9k citations indexed

About

E. Douglas Lewandowski is a scholar working on Cardiology and Cardiovascular Medicine, Molecular Biology and Radiology, Nuclear Medicine and Imaging. According to data from OpenAlex, E. Douglas Lewandowski has authored 81 papers receiving a total of 3.9k indexed citations (citations by other indexed papers that have themselves been cited), including 43 papers in Cardiology and Cardiovascular Medicine, 38 papers in Molecular Biology and 32 papers in Radiology, Nuclear Medicine and Imaging. Recurrent topics in E. Douglas Lewandowski's work include Cardiovascular Function and Risk Factors (32 papers), Advanced MRI Techniques and Applications (29 papers) and Mitochondrial Function and Pathology (28 papers). E. Douglas Lewandowski is often cited by papers focused on Cardiovascular Function and Risk Factors (32 papers), Advanced MRI Techniques and Applications (29 papers) and Mitochondrial Function and Pathology (28 papers). E. Douglas Lewandowski collaborates with scholars based in United States, Canada and Germany. E. Douglas Lewandowski's co-authors include J. Michael O’Donnell, Lawrence T. White, Andrew N. Carley, Daniel P. Kelly, Teresa C. Leone, Deborah M. Muoio, Peter A. Crawford, Rick B. Vega, Julie L. Horton and Nathaniel M. Alpert and has published in prestigious journals such as Circulation, Journal of Clinical Investigation and Journal of the American College of Cardiology.

In The Last Decade

E. Douglas Lewandowski

80 papers receiving 3.9k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

The Failing Heart Relies on Ketone Bodies as a Fuel

2016 549 citations Grégory Aubert, Ola J. Martin et al. Circulation profile →
The failing heart utilizes 3-hydroxybutyrate as a metabolic stress defense

2019 260 citations Julie L. Horton, Rick B. Vega et al. profile →

Peers

Countries citing papers authored by E. Douglas Lewandowski

Since Specialization

Citations

This map shows the geographic impact of E. Douglas Lewandowski's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by E. Douglas Lewandowski with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites E. Douglas Lewandowski more than expected).

Fields of papers citing papers by E. Douglas Lewandowski

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by E. Douglas Lewandowski. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by E. Douglas Lewandowski. The network helps show where E. Douglas Lewandowski may publish in the future.

Co-authorship network of co-authors of E. Douglas Lewandowski

This figure shows the co-authorship network connecting the top 25 collaborators of E. Douglas Lewandowski. A scholar is included among the top collaborators of E. Douglas Lewandowski based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with E. Douglas Lewandowski. E. Douglas Lewandowski is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

#	Title	Journal	Authors	Indexed citations
1	CPT1a Expression Is a Critical Cardioprotective Response to Pathological Stress That Enables Rescue by Gene Transfer	Circulation Research	Andrew N. Carley, S. Maurya et al.	0
2	UCP1 ‐dependent brown adipose activation accelerates cardiac metabolic remodeling and reduces initial hypertrophic and fibrotic responses to pathological stress	The FASEB Journal	Azariyas A. Challa, Pablo Vidal et al.	6
3	RIP140 deficiency enhances cardiac fuel metabolism and protects mice from heart failure	Journal of Clinical Investigation	Tsunehisa Yamamoto, Santosh K. Maurya et al.	8
4	The Failing Heart Relies on Ketone Bodies as a Fuel breakdown →	Circulation	Grégory Aubert, Ola J. Martin et al.	549
5	Triacylglycerol turnover in the failing heart	Biochimica et Biophysica Acta (BBA) - Molecular and Cell Biology of Lipids	Andrew N. Carley, E. Douglas Lewandowski	21
6	Acyl CoA synthetase-1 links facilitated long chain fatty acid uptake to intracellular metabolic trafficking differently in hearts of male versus female mice	Journal of Molecular and Cellular Cardiology	Xuerong Wang, E. Douglas Lewandowski et al.	25
7	Impaired cytosolic NADH shuttling and elevated UCP3 contribute to inefficient citric acid cycle flux support of postischemic cardiac work in diabetic hearts	Journal of Molecular and Cellular Cardiology	Natasha H. Banke, E. Douglas Lewandowski	23
8	MITOCHONDRIA-TARGETED ANTIOXIDANT AMELIORATES DIET-INDUCED DIABETES AND DIASTOLIC DYSFUNCTION	Journal of the American College of Cardiology	Jaehoon Chung, Scott Gladstein et al.	5
9	In Vivo, Cardiac-Specific Knockdown of a Target Protein, Malic Enzyme- 1, in Rat via Adenoviral Delivery of DNA for Non-Native miRNA	Current Gene Therapy	J. Michael O’Donnell, Jian Bi et al.	5
10	Multiphasic triacylglycerol dynamics in the intact heart during acute in vivo overexpression of CD36	Journal of Lipid Research	Andrew N. Carley, Jian Bi et al.	29
11	Second window of preconditioning normalizes palmitate use for oxidation and improves function during low-flow ischaemia	Cardiovascular Research	Raymond K. Kudej, Xin Zhao et al.	9
12	Sexual dimorphism in cardiac triacylglyceride dynamics in mice on long term caloric restriction	Journal of Molecular and Cellular Cardiology	Natasha H. Banke, Sunil Dhar et al.	16
13	Substrate–Enzyme Competition Attenuates Upregulated Anaplerotic Flux Through Malic Enzyme in Hypertrophied Rat Heart and Restores Triacylglyceride Content	Circulation Research	Н. И. Сорокина, Kalpana Ballal et al.	121
14	Recruitment of Compensatory Pathways to Sustain Oxidative Flux With Reduced Carnitine Palmitoyltransferase I Activity Characterizes Inefficiency in Energy Metabolism in Hypertrophied Hearts	Circulation	Н. И. Сорокина, J. Michael O’Donnell et al.	151
15	The absence of endogenous lipid oxidation in early stage heart failure exposes limits in lipid storage and turnover	Journal of Molecular and Cellular Cardiology	J. Michael O’Donnell, Н. И. Сорокина et al.	98
16	Coupling of Mitochondrial Fatty Acid Uptake to Oxidative Flux in the Intact Heart	Biophysical Journal	J. Michael O’Donnell, Nathaniel M. Alpert et al.	37
17	Multiplet structure of ¹³C NMR signal from glutamate and direct detection of tricarboxylic acid (TCA) cycle intermediates	Magnetic Resonance in Medicine	E. Douglas Lewandowski, Chris Doumen et al.	37
18	Kinetic analysis of dynamic 13C NMR spectra: metabolic flux, regulation, and compartmentation in hearts	Biophysical Journal	Xin Yu, Lawrence T. White et al.	91
19	Abstracts-part X		M. Harold Laughlin, Joby Taylor et al.	1
20	Metabolic heterogeneity of carbon substrate utilization in mammalian heart: NMR determinations of mitochondrial versus cytosolic compartmentation	Biochemistry	E. Douglas Lewandowski	56

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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