Dittmar Labeit

690 total citations
10 papers, 559 citations indexed

About

Dittmar Labeit is a scholar working on Cardiology and Cardiovascular Medicine, Molecular Biology and Cellular and Molecular Neuroscience. According to data from OpenAlex, Dittmar Labeit has authored 10 papers receiving a total of 559 indexed citations (citations by other indexed papers that have themselves been cited), including 6 papers in Cardiology and Cardiovascular Medicine, 5 papers in Molecular Biology and 2 papers in Cellular and Molecular Neuroscience. Recurrent topics in Dittmar Labeit's work include Muscle Physiology and Disorders (5 papers), Cardiomyopathy and Myosin Studies (3 papers) and Genetic Neurodegenerative Diseases (2 papers). Dittmar Labeit is often cited by papers focused on Muscle Physiology and Disorders (5 papers), Cardiomyopathy and Myosin Studies (3 papers) and Genetic Neurodegenerative Diseases (2 papers). Dittmar Labeit collaborates with scholars based in Germany, United States and United Kingdom. Dittmar Labeit's co-authors include Siegfried Labeit, Alexander Gasch, Muzamil Majid Khan, Rüdiger Rudolf, Christian Witt, Franziska Wild, Markus Reischl, Henk Granzier, Christine Kohl and S. Strack and has published in prestigious journals such as Proceedings of the National Academy of Sciences, SHILAP Revista de lepidopterología and Journal of Molecular Biology.

In The Last Decade

Dittmar Labeit

10 papers receiving 554 citations

Peers

Dittmar Labeit

PHYSI

CMN

CCM

Tiziana Mongini Italy

Sara Bonato Italy

Adel Amirouche France

Elisabetta Tasca Italy

Raffaella Willmann Switzerland

Sean Germain United States

Satoshi Kuru Japan

Marie‐Louise Sveen Denmark

Léonard Feasson France

Emanuela Longa Italy

Tiziana Mongini Italy

Dittmar Labeit

399 ×1.0

111 ×0.7

PHYSI

141 ×1.2

CMN

117 ×1.0

CCM

103 ×1.1

Citations per year, relative to Dittmar Labeit Dittmar Labeit (= 1×) peers Tiziana Mongini

Countries citing papers authored by Dittmar Labeit

Since Specialization

Citations

This map shows the geographic impact of Dittmar Labeit's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Dittmar Labeit with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Dittmar Labeit more than expected).

Fields of papers citing papers by Dittmar Labeit

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Dittmar Labeit. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Dittmar Labeit. The network helps show where Dittmar Labeit may publish in the future.

Co-authorship network of co-authors of Dittmar Labeit

This figure shows the co-authorship network connecting the top 25 collaborators of Dittmar Labeit. A scholar is included among the top collaborators of Dittmar Labeit based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Dittmar Labeit. Dittmar Labeit is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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10 of 10 papers shown

Ujihara, Yoshihiro, Ken Hashimoto, Masato Hoshino, et al.. (2025). CCDC141 is a Connectin/Titin and Nesprin-1 binding protein that adapts cardiomyocytes to mechanical stress. Communications Biology. 8(1). 1693–1693. 1 indexed citations

Adams, Volker, T. Scott Bowen, Sarah Werner, et al.. (2019). Small‐molecule‐mediated chemical knock‐down of MuRF1/MuRF2 and attenuation of diaphragm dysfunction in chronic heart failure. Journal of Cachexia Sarcopenia and Muscle. 10(5). 1102–1115. 42 indexed citations

Bowen, T. Scott, Volker Adams, Sarah Werner, et al.. (2017). Small‐molecule inhibition of MuRF1 attenuates skeletal muscle atrophy and dysfunction in cardiac cachexia. Journal of Cachexia Sarcopenia and Muscle. 8(6). 939–953. 79 indexed citations

Khan, Muzamil Majid, Danilo Lustrino, Wilian A. Silveira, et al.. (2016). Sympathetic innervation controls homeostasis of neuromuscular junctions in health and disease. Proceedings of the National Academy of Sciences. 113(3). 746–750. 127 indexed citations

Bogomolovas, Julius, et al.. (2016). Cardiac specific titin N2B exon is a novel sensitive serological marker for cardiac injury. International Journal of Cardiology. 212. 232–234. 11 indexed citations

Bogomolovas, Julius, Egidijus Šimoliūnas, Daiva Bironaitė, et al.. (2016). A Novel Murine Model of Parvovirus Associated Dilated Cardiomyopathy Induced by Immunization with VP1-Unique Region of Parvovirus B19. BioMed Research International. 2016. 1–9. 10 indexed citations

Khan, Muzamil Majid, S. Strack, Franziska Wild, et al.. (2013). Role of autophagy, SQSTM1, SH3GLB1, and TRIM63 in the turnover of nicotinic acetylcholine receptors. Autophagy. 10(1). 123–136. 80 indexed citations

Rudolf, Rüdiger, Julius Bogomolovas, S. Strack, et al.. (2012). Regulation of nicotinic acetylcholine receptor turnover by MuRF1 connects muscle activity to endo/lysosomal and atrophy pathways. AGE. 35(5). 1663–1674. 55 indexed citations

Labeit, Siegfried, et al.. (2010). Modulation of Muscle Atrophy, Fatigue and MLC Phosphorylation by MuRF1 as Indicated by Hindlimb Suspension Studies on MuRF1-KO Mice. SHILAP Revista de lepidopterología. 2010. 1–9. 77 indexed citations

10.

Hirner, Stephanie, Alexander Schuster, Sigrid Hoffmann, et al.. (2008). MuRF1-dependent Regulation of Systemic Carbohydrate Metabolism as Revealed from Transgenic Mouse Studies. Journal of Molecular Biology. 379(4). 666–677. 77 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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