Diha Freije

3.1k total citations
19 papers, 1.2k citations indexed

About

Diha Freije is a scholar working on Molecular Biology, Pulmonary and Respiratory Medicine and Genetics. According to data from OpenAlex, Diha Freije has authored 19 papers receiving a total of 1.2k indexed citations (citations by other indexed papers that have themselves been cited), including 12 papers in Molecular Biology, 8 papers in Pulmonary and Respiratory Medicine and 6 papers in Genetics. Recurrent topics in Diha Freije's work include Prostate Cancer Treatment and Research (8 papers), BRCA gene mutations in cancer (5 papers) and Epigenetics and DNA Methylation (5 papers). Diha Freije is often cited by papers focused on Prostate Cancer Treatment and Research (8 papers), BRCA gene mutations in cancer (5 papers) and Epigenetics and DNA Methylation (5 papers). Diha Freije collaborates with scholars based in United States, Sweden and Italy. Diha Freije's co-authors include William B. Isaacs, G. Steven Bova, Deborah Nusskern, P Cairns, Hidenori Suzuki, Kenji Okami, David Sidransky, Helen Donis-Keller, Cynthia Helms and Michael S. Watson and has published in prestigious journals such as Science, The Journal of Urology and American Journal Of Pathology.

In The Last Decade

Diha Freije

19 papers receiving 1.2k citations

Peers

Diha Freije

PRM

GENET

ONCOL

L.W. Chu United States

Debora Rasio Italy

Christiane Hader Germany

Hisako Ochi United States

Cacilda Casartelli Brazil

Alexandr V. Togo Russia

Toshinori Hinoue United States

Alina Levy United States

Ángel Martínez‐Ramírez Spain

Małgorzata Jarmuż‐Szymczak Poland

L.W. Chu United States

Diha Freije

357 ×0.5

147 ×0.3

PRM

368 ×1.2

GENET

349 ×1.5

252 ×1.6

ONCOL

Citations per year, relative to Diha Freije Diha Freije (= 1×) peers L.W. Chu

Countries citing papers authored by Diha Freije

Since Specialization

Citations

This map shows the geographic impact of Diha Freije's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Diha Freije with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Diha Freije more than expected).

Fields of papers citing papers by Diha Freije

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Diha Freije. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Diha Freije. The network helps show where Diha Freije may publish in the future.

Co-authorship network of co-authors of Diha Freije

This figure shows the co-authorship network connecting the top 25 collaborators of Diha Freije. A scholar is included among the top collaborators of Diha Freije based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Diha Freije. Diha Freije is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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19 of 19 papers shown

Brikun, Igor, Deborah Nusskern, & Diha Freije. (2019). An expanded biomarker panel for the detection of prostate cancer from urine DNA. Experimental Hematology and Oncology. 8(1). 13–13. 19 indexed citations

Brikun, Igor, et al.. (2018). A panel of DNA methylation markers for the detection of prostate cancer from FV and DRE urine DNA. Clinical Epigenetics. 10(1). 91–91. 36 indexed citations

Brikun, Igor, Deborah Nusskern, Daniel L. Gillen, et al.. (2014). A panel of DNA methylation markers reveals extensive methylation in histologically benign prostate biopsy cores from cancer patients. Biomarker Research. 2(1). 25–25. 15 indexed citations

Ho, Gloria, Michael Knapp, Diha Freije, et al.. (2002). Transmission/disequilibrium tests of androgen receptor and glutathione S‐transferase pi variants in prostate cancer families. International Journal of Cancer. 98(6). 938–942. 6 indexed citations

Lin, Xiaohui, Metin Tascilar, Wen-Hsiang Lee, et al.. (2001). GSTP1 CpG Island Hypermethylation Is Responsible for the Absence of GSTP1 Expression in Human Prostate Cancer Cells. American Journal Of Pathology. 159(5). 1815–1826. 179 indexed citations

Tchou, Julia, Xue Lin, Diha Freije, et al.. (2000). GSTP1 CpG island DNA hypermethylation in hepatocellular carcinomas.. International Journal of Oncology. 16(4). 663–76. 91 indexed citations

Tchou, Julia, Xihong Lin, Diha Freije, et al.. (2000). Tchou, J.C. et al. GSTP1 CpG island DNA hypermethylation in hepatocellular carcinoma. Int. J. Oncol. 16, 663−676. 5 indexed citations

Shepard, Timothy F., Elizabeth A. Platz, Philip W. Kantoff, et al.. (2000). No association between the I105V polymorphism of the glutathione S-transferase P1 gene (GSTP1) and prostate cancer risk: a prospective study.. PubMed. 9(11). 1267–8. 29 indexed citations

Wilkens, Eric P., Diha Freije, Jianfeng Xu, et al.. (1999). No evidence for a role ofBRCA1 orBRCA2 mutations in Ashkenazi Jewish families with hereditary prostate cancer. The Prostate. 39(4). 280–284. 45 indexed citations

10.

Wilkens, Eric P., Diha Freije, Jianfeng Xu, et al.. (1999). No evidence for a role of BRCA1 or BRCA2 mutations in Ashkenazi Jewish families with hereditary prostate cancer. The Prostate. 39(4). 280–284. 10 indexed citations

11.

Kibel, Adam S., Diha Freije, William B. Isaacs, & G. Steven Bova. (1999). Deletion mapping at 12p12-13 in metastatic prostate cancer. Genes Chromosomes and Cancer. 25(3). 270–276. 35 indexed citations

12.

Kibel, Adam S., Diha Freije, William B. Isaacs, & G. Steven Bova. (1999). Deletion mapping at 12p12–13 in metastatic prostate cancer. Genes Chromosomes and Cancer. 25(3). 270–276. 1 indexed citations

13.

Suzuki, Hidenori, Diha Freije, Deborah Nusskern, et al.. (1998). Interfocal heterogeneity of PTEN/MMAC1 gene alterations in multiple metastatic prostate cancer tissues.. PubMed. 58(2). 204–9. 361 indexed citations

14.

Grönberg, Henrik, Jianfeng Xu, Jeffrey R. Smith, et al.. (1998). Early Age at Diagnosis in Families Providing Evidence of Linkage to the Hereditary Prostate Cancer Locus (HPC1) on Chromosome 1. The Journal of Urology. 160(1). 265–266. 24 indexed citations

15.

Grönberg, Henrik, Sarah D. Isaacs, Jeffrey R. Smith, et al.. (1998). Characteristics of Prostate Cancer in Families Potentially Linked to the Hereditary Prostate Cancer 1 (HPC1) Locus. The Journal of Urology. 159(4). 1399–1400. 10 indexed citations

16.

Grönberg, Henrik, Jinping Xu, Jeffrey R. Smith, et al.. (1998). Early Age at Diagnosis in Families Providing Evidence of Linkage to the Hereditary Prostate Cancer Locus (HPC1) on Chromosome 1. The Journal of Urology. 57(21). 265–266. 80 indexed citations

17.

Freije, Diha & David Schlessinger. (1992). A 1.6-Mb contig of yeast artificial chromosomes around the human factor VIII gene reveals three regions homologous to probes for the DXS115 locus and two for the DXYS64 locus.. PubMed. 51(1). 66–80. 44 indexed citations

18.

Freije, Diha, Cynthia Helms, Michael S. Watson, & Helen Donis-Keller. (1992). Identification of a Second Pseudoautosomal Region Near the Xq and Yq Telomeres. Science. 258(5089). 1784–1787. 133 indexed citations

19.

Schlessinger, David, Diha Freije, Fatima Abidi, et al.. (1991). Yeast artificial chromosome-based genome mapping: Some lessons from Xq24–q28. Genomics. 11(4). 783–793. 58 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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