Davide Trotti
- Neurology top 0.5%
- Amyotrophic Lateral Sclerosis Research 43
- Parkinson's Disease Mechanisms and Treatments 11
- Cellular and Molecular Neuroscience top 0.5%
- Neuroscience and Neuropharmacology Research 25
- Genetic Neurodegenerative Diseases 10
- Neurology top 0.5%
- Amyotrophic Lateral Sclerosis Research 43
- Parkinson's Disease Mechanisms and Treatments 11
- Genetics top 0.5%
- Neurogenetic and Muscular Disorders Research 22
- Biochemistry top 0.5%
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- Ion channel regulation and function 7
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- Molecular Sensors and Ion Detection 6
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- Drug Transport and Resistance Mechanisms 6
Davide Trotti
69 papers receiving 5.8k citations
Hit Papers
Peers
Comparison fields: 5 of 111
- Neurology 2.3k
- Cellular and Molecular Neuroscience 2.3k
- Neurology 952
- Genetics 1.1k
- Biochemistry 699
Countries citing papers authored by Davide Trotti
This map shows the geographic impact of Davide Trotti's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Davide Trotti with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Davide Trotti more than expected).
Fields of papers citing papers by Davide Trotti
This network shows the impact of papers produced by Davide Trotti. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Davide Trotti. The network helps show where Davide Trotti may publish in the future.
Co-authorship network
The 25 scholars most cited alongside Davide Trotti, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2025 | 2 | |
| 2 | 2024 | 3 | |
| 3 | 2024 | 3 | |
| 4 | 2024 | 2 | |
| 5 | 2023 | 0 | |
| 6 | 2023 | 7 | |
| 7 | 2023 | 4 | |
| 8 | 2022 | 35 | |
| 9 | 2022 | 16 | |
| 10 | 2021 | 2 | |
| 11 | 2019 | 45 | |
| 12 | 2014 | 93 | |
| 13 | 2012 | 154 | |
| 14 | 2006 | 109 | |
| 15 | 2003 | 9 | |
| 16 | 2001 | 101 | |
| 17 | 2001 | 50 | |
| 18 | 1999 | 24 | |
| 19 | 1995 | 4 | |
| 20 | 1994 | 146 |
About Davide Trotti
Davide Trotti is a scholar working on Neurology, Cellular and Molecular Neuroscience and Genetics, having authored 70 papers that have together received 5.9k indexed citations. Recurring topics across this work include Amyotrophic Lateral Sclerosis Research (43 papers), Neuroscience and Neuropharmacology Research (25 papers), Neurogenetic and Muscular Disorders Research (22 papers), Parkinson's Disease Mechanisms and Treatments (11 papers), Genetic Neurodegenerative Diseases (10 papers), Ion channel regulation and function (7 papers), Molecular Sensors and Ion Detection (6 papers) and Drug Transport and Resistance Mechanisms (6 papers). The work is most often cited by research in Neurology (2.3k citations), Cellular and Molecular Neuroscience (2.3k citations) and Neurology (952 citations). Davide Trotti has collaborated with scholars based in United States, Italy and Norway. Frequent co-authors include Andrea Volterra, Piera Pasinelli, Niels C. Danbolt, Giorgio Racagni, Robert H. Brown, Matthias A. Hediger, Emily Foran, Cinzia Tromba, Daniela Rossi and Xinmei Wen. Their work appears in journals such as Journal of Biological Chemistry, Glia, European Journal of Neuroscience, Experimental Neurology and Journal of Neuroscience.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.