David G. Parkes

7.6k total citations
112 papers, 6.0k citations indexed

About

David G. Parkes is a scholar working on Endocrinology, Diabetes and Metabolism, Surgery and Endocrine and Autonomic Systems. According to data from OpenAlex, David G. Parkes has authored 112 papers receiving a total of 6.0k indexed citations (citations by other indexed papers that have themselves been cited), including 44 papers in Endocrinology, Diabetes and Metabolism, 34 papers in Surgery and 26 papers in Endocrine and Autonomic Systems. Recurrent topics in David G. Parkes's work include Diabetes Treatment and Management (32 papers), Pancreatic function and diabetes (28 papers) and Regulation of Appetite and Obesity (24 papers). David G. Parkes is often cited by papers focused on Diabetes Treatment and Management (32 papers), Pancreatic function and diabetes (28 papers) and Regulation of Appetite and Obesity (24 papers). David G. Parkes collaborates with scholars based in Australia, United States and United Kingdom. David G. Parkes's co-authors include Jonathan D. Roth, Andrew A. Young, Loretta L. Nielsen, James L. Trevaskis, Carolyn M. Jodka, Clive N. May, Alain Baron, Andrew Young, Steve Chen and Wylie Vale and has published in prestigious journals such as New England Journal of Medicine, Proceedings of the National Academy of Sciences and The Lancet.

In The Last Decade

David G. Parkes

111 papers receiving 5.6k citations

Peers

David G. Parkes
Andrew M. Allen Australia
Gerald J. Taborsky United States
Robin L. Davisson United States
D. Porte United States
Niels Vrang Denmark
Colin Sumners United States
Diego Pérez–Tilve United States
Andrew M. Allen Australia
David G. Parkes
Citations per year, relative to David G. Parkes David G. Parkes (= 1×) peers Andrew M. Allen

Countries citing papers authored by David G. Parkes

Since Specialization
Citations

This map shows the geographic impact of David G. Parkes's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by David G. Parkes with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites David G. Parkes more than expected).

Fields of papers citing papers by David G. Parkes

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by David G. Parkes. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by David G. Parkes. The network helps show where David G. Parkes may publish in the future.

Co-authorship network of co-authors of David G. Parkes

This figure shows the co-authorship network connecting the top 25 collaborators of David G. Parkes. A scholar is included among the top collaborators of David G. Parkes based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with David G. Parkes. David G. Parkes is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Rodríguez, Rubén, Andrew Y. Lee, Bridget Martinez, et al.. (2021). Chronic AT1 blockade improves hyperglycemia by decreasing adipocyte inflammation and decreasing hepatic PCK1 and G6PC1 expression in obese rats. American Journal of Physiology-Endocrinology and Metabolism. 321(5). E714–E727. 5 indexed citations
2.
Hay, Debbie L., Steve Chen, Thomas A. Lutz, David G. Parkes, & Jonathan D. Roth. (2015). Amylin: Pharmacology, Physiology, and Clinical Potential. Pharmacological Reviews. 67(3). 564–600. 299 indexed citations
3.
DeYoung, Mary Beth, et al.. (2012). GLP ‐1 receptor activated insulin secretion from pancreatic β‐cells: mechanism and glucose dependence. Diabetes Obesity and Metabolism. 15(1). 15–27. 292 indexed citations
4.
Mack, Christine M., Porsha Smith, Jennifer Athanacio, et al.. (2011). Glucoregulatory effects and prolonged duration of action of davalintide: a novel amylinomimetic peptide. Diabetes Obesity and Metabolism. 13(12). 1105–1113. 43 indexed citations
5.
Trevaskis, James L., Victoria F. Turek, Carrie Wittmer, et al.. (2010). Enhanced Amylin-Mediated Body Weight Loss in Estradiol-Deficient Diet-Induced Obese Rats. Endocrinology. 151(12). 5657–5668. 55 indexed citations
6.
Laugero, Kevin D., et al.. (2009). Exenatide Improves Hypertension in a Rat Model of the Metabolic Syndrome. Metabolic Syndrome and Related Disorders. 7(4). 327–334. 37 indexed citations
7.
Trevaskis, James L., Todd Coffey, Rebecca L. Cole, et al.. (2008). Amylin-Mediated Restoration of Leptin Responsiveness in Diet-Induced Obesity: Magnitude and Mechanisms. Endocrinology. 149(11). 5679–5687. 125 indexed citations
8.
Roth, Jonathan D., Barbara Roland, Rebecca L. Cole, et al.. (2008). Leptin responsiveness restored by amylin agonism in diet-induced obesity: Evidence from nonclinical and clinical studies. Proceedings of the National Academy of Sciences. 105(20). 7257–7262. 348 indexed citations
9.
Hargrove, Diane M., James M. Reynolds, John Herich, et al.. (2007). Biological activity of AC3174, a peptide analog of exendin-4. Regulatory Peptides. 141(1-3). 113–119. 33 indexed citations
10.
Mack, Christine M., Candace X. Moore, Carolyn M. Jodka, et al.. (2006). Antiobesity action of peripheral exenatide (exendin-4) in rodents: effects on food intake, body weight, metabolic status and side-effect measures. International Journal of Obesity. 30(9). 1332–1340. 119 indexed citations
11.
Pittner, Richard, Candace X. Moore, Sunil Bhavsar, et al.. (2004). Effects of PYY[3–36] in rodent models of diabetes and obesity. International Journal of Obesity. 28(8). 963–971. 186 indexed citations
13.
Parkes, David G.. (1996). Diuretic and Natriuretic Actions of Melanin Concentrating Hormone in Conscious Sheep. Journal of Neuroendocrinology. 8(1). 57–63. 26 indexed citations
14.
Parkes, David G., et al.. (1995). Pyrogenic stimulation of vascular resistance in conscious sheep. Life Sciences. 57(6). 599–608. 3 indexed citations
15.
Kasckow, John, David G. Parkes, Michael J. Owens, et al.. (1994). The BE (2)-M17 neuroblastoma cell line synthesizes and secretes corticotropin-releasing factor. Brain Research. 654(1). 159–162. 12 indexed citations
16.
Parkes, David G. & Wylie Vale. (1993). Contrasting Actions of Melanin‐Concentrating Hormone and Neuropeptide‐E‐I on Posterior Pituitary Function. Annals of the New York Academy of Sciences. 680(1). 588–590. 25 indexed citations
17.
Parkes, David G., et al.. (1991). Effects of Calcium Channel Blockade on the Hemodynamic Responses to Endothelin Infusion in Conscious Sheep. American Journal of Hypertension. 4(8). 719–722. 1 indexed citations
18.
Parkes, David G., J. P. Coghlan, John Lewicki, Robert M. Scarborough, & B. A. Scoggins. (1990). Hemodynamic Effects of Atrial Natriuretic Factor Clearance Receptor Occupancy in Conscious Sheep. American Journal of Hypertension. 3(11). 829–832. 2 indexed citations
19.
Parkes, David G., John P. Coghlan, & Bruce A. Scoggins. (1989). Comparative hemodynamic actions of ANF-related peptides in conscious sheep. Life Sciences. 45(24). 2303–2312. 4 indexed citations
20.
Parkes, David G., et al.. (1987). Hemodynamic Effects of Atrial Natriuretic Peptide in Conscious Sheep. Clinical and Experimental Hypertension Part A Theory and Practice. 9(sup1). 2143–2155. 17 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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