Daniel E. Gottschling

16.1k total citations · 6 hit papers
74 papers, 12.6k citations indexed

About

Daniel E. Gottschling is a scholar working on Molecular Biology, Physiology and Aging. According to data from OpenAlex, Daniel E. Gottschling has authored 74 papers receiving a total of 12.6k indexed citations (citations by other indexed papers that have themselves been cited), including 64 papers in Molecular Biology, 14 papers in Physiology and 14 papers in Aging. Recurrent topics in Daniel E. Gottschling's work include DNA Repair Mechanisms (23 papers), Fungal and yeast genetics research (23 papers) and Genomics and Chromatin Dynamics (19 papers). Daniel E. Gottschling is often cited by papers focused on DNA Repair Mechanisms (23 papers), Fungal and yeast genetics research (23 papers) and Genomics and Chromatin Dynamics (19 papers). Daniel E. Gottschling collaborates with scholars based in United States, South Africa and Netherlands. Daniel E. Gottschling's co-authors include Oscar M. Aparicio, Barbara Billington, Miriam Singer, Virginia A. Zakian, Fred van Leeuwen, Thomas R. Cech, Arthur J. Zaug, Kelly Kruger, Paula J. Grabowski and Adam L. Hughes and has published in prestigious journals such as Nature, Science and Cell.

In The Last Decade

Daniel E. Gottschling

72 papers receiving 12.3k citations

Hit Papers

5 papers align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Self-splicing RNA: Autoexcision and autocyclization of the ribosomal RNA intervening sequence of tetrahymena

1982 1.6k citations Daniel E. Gottschling et al. Cell profile →
Position effect at S. cerevisiae telomeres: Reversible repression of Pol II transcription

1990 1.1k citations Daniel E. Gottschling, Oscar M. Aparicio et al. Cell profile →
Dot1p Modulates Silencing in Yeast by Methylation of the Nucleosome Core

2002 661 citations Fred van Leeuwen, Philip R. Gafken et al. Cell profile →
The histone modification pattern of active genes revealed through genome-wide chromatin analysis of a higher eukaryote

2004 634 citations Fred van Leeuwen, Daniel E. Gottschling et al. Genes & Development profile →
TLC1 : Template RNA Component of Saccharomyces cerevisiae Telomerase

1994 632 citations Daniel E. Gottschling et al. Science profile →
Modifiers of position effect are shared between telomeric and silent mating-type loci in S. cerevisiae

1991 604 citations Oscar M. Aparicio, Barbara Billington et al. Cell profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Daniel E. Gottschling United States	48	11.1k	2.5k	1.9k	1.4k	943	74	12.6k
David Shore Switzerland	60	9.7k 0.9×	2.8k 1.1×	1.6k 0.8×	1.2k 0.8×	661 0.7×	111	10.7k
Robbie Loewith Switzerland	41	12.2k 1.1×	1.2k 0.5×	1.3k 0.7×	734 0.5×	627 0.7×	67	15.4k
Ronald A. Butow United States	57	9.8k 0.9×	685 0.3×	773 0.4×	563 0.4×	536 0.6×	137	10.7k
Anton Gartner United Kingdom	51	6.0k 0.5×	484 0.2×	943 0.5×	2.5k 1.8×	598 0.6×	107	7.7k
Simon J. Boulton United Kingdom	66	13.8k 1.2×	1.6k 0.6×	1.3k 0.7×	1.6k 1.1×	1.5k 1.6×	152	15.7k
Meng‐Qiu Dong China	52	7.3k 0.7×	822 0.3×	733 0.4×	766 0.5×	545 0.6×	186	9.8k
Brian F.C. Clark Denmark	52	8.3k 0.7×	741 0.3×	685 0.4×	417 0.3×	1.4k 1.5×	218	10.4k
Michael P. Myers United States	44	7.3k 0.7×	828 0.3×	1.5k 0.8×	393 0.3×	594 0.6×	98	10.2k
Cynthia Wolberger United States	55	9.0k 0.8×	1.5k 0.6×	595 0.3×	267 0.2×	1.1k 1.1×	121	12.8k
Ingrid Grummt Germany	80	15.2k 1.4×	608 0.2×	1.3k 0.7×	150 0.1×	1.6k 1.7×	175	17.3k

Countries citing papers authored by Daniel E. Gottschling

Since Specialization

Citations

This map shows the geographic impact of Daniel E. Gottschling's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Daniel E. Gottschling with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Daniel E. Gottschling more than expected).

Fields of papers citing papers by Daniel E. Gottschling

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Daniel E. Gottschling. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Daniel E. Gottschling. The network helps show where Daniel E. Gottschling may publish in the future.

Co-authorship network of co-authors of Daniel E. Gottschling

This figure shows the co-authorship network connecting the top 25 collaborators of Daniel E. Gottschling. A scholar is included among the top collaborators of Daniel E. Gottschling based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Daniel E. Gottschling. Daniel E. Gottschling is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

Sort: Min cites: Since: Top N: Style:

20 of 20 papers shown

Thayer, Nathaniel H., et al.. (2026). Single-cell analyses identify independent aging processes that compete to determine cellular fate in budding yeast. Proceedings of the National Academy of Sciences. 123(11). e2534452123–e2534452123.

Shabestary, Kiyan, Reinhard Dechant, & Daniel E. Gottschling. (2025). Along the Trajectory to Understanding Cellular Aging: More Lessons from Yeast. Annual Review of Cell and Developmental Biology. 41(1). 353–373.

Waite, Adam James, et al.. (2025). Potassium ion homeostasis modulates mitochondrial function. The Journal of Cell Biology. 225(4). 1 indexed citations

Thayer, Nathaniel H., et al.. (2014). Identification of long-lived proteins retained in cells undergoing repeated asymmetric divisions. Proceedings of the National Academy of Sciences. 111(39). 14019–14026. 60 indexed citations

Dillin, Andrew, Daniel E. Gottschling, & Thomas Nyström. (2014). The good and the bad of being connected: the integrons of aging. Current Opinion in Cell Biology. 26. 107–112. 104 indexed citations

Dymond, Jessica S., Sarah M. Richardson, Candice Coombes, et al.. (2011). Synthetic chromosome arms function in yeast and generate phenotypic diversity by design. Nature. 477(7365). 471–476. 327 indexed citations

Lindstrom, Derek L. & Daniel E. Gottschling. (2009). The Mother Enrichment Program: A Genetic System for Facile Replicative Life Span Analysis in Saccharomyces cerevisiae. Genetics. 183(2). 413–422. 135 indexed citations

Welsem, Tibor van, Floor Frederiks, Kitty F. Verzijlbergen, et al.. (2008). Synthetic Lethal Screens Identify Gene Silencing Processes in Yeast and Implicate the Acetylated Amino Terminus of Sir3 in Recognition of the Nucleosome Core. Molecular and Cellular Biology. 28(11). 3861–3872. 49 indexed citations

Gottschling, Daniel E.. (2006). DNA Repair: Corrections in the Golden Years. Current Biology. 16(22). R956–R958. 4 indexed citations

10.

Gardner, Richard G., et al.. (2005). Ubp10/Dot4p Regulates the Persistence of Ubiquitinated Histone H2B: Distinct Roles in Telomeric Silencing and General Chromatin. Molecular and Cellular Biology. 25(14). 6123–6139. 130 indexed citations

11.

McMurray, Michael A. & Daniel E. Gottschling. (2003). An Age-Induced Switch to a Hyper-Recombinational State. Science. 301(5641). 1908–1911. 166 indexed citations

12.

Smith, Christine M., Catherine O. Johnson, Philip R. Gafken, et al.. (2002). Heritable chromatin structure: Mapping “memory” in histones H3 and H4. Proceedings of the National Academy of Sciences. 99(suppl_4). 16454–16461. 69 indexed citations

13.

Leeuwen, Fred van & Daniel E. Gottschling. (2002). Assays for gene silencing in yeast. Methods in enzymology on CD-ROM/Methods in enzymology. 350. 165–186. 63 indexed citations

14.

Diede, Scott J. & Daniel E. Gottschling. (2001). Exonuclease activity is required for sequence addition and Cdc13p loading at a de novo telomere. Current Biology. 11(17). 1336–1340. 121 indexed citations

15.

Qin, Song, et al.. (2000). Type B Histone Acetyltransferase Hat1p Participates in Telomeric Silencing. Molecular and Cellular Biology. 20(19). 7051–7058. 103 indexed citations

16.

Gottschling, Daniel E.. (2000). Gene silencing: Two faces of SIR2. Current Biology. 10(19). R708–R711. 36 indexed citations

17.

Diede, Scott J., et al.. (2000). All Things Must End: Telomere Dynamics in Yeast. Cold Spring Harbor Symposia on Quantitative Biology. 65(0). 281–296. 5 indexed citations

18.

Wright, James H., Daniel E. Gottschling, & Virginia A. Zakian. (1992). Saccharomyces telomeres assume a non-nucleosomal chromatin structure.. Genes & Development. 6(2). 197–210. 233 indexed citations

19.

Gottschling, Daniel E., Oscar M. Aparicio, Barbara Billington, & Virginia A. Zakian. (1990). Position effect at S. cerevisiae telomeres: Reversible repression of Pol II transcription. Cell. 63(4). 751–762. 1135 indexed citations breakdown →

20.

Zühlke, H, et al.. (1974). Catabolism of proinsulin and insulin. Proteolytic activities in Langerhans islets of rat and mice pancreas in vitro.. PubMed. 33(4). 407–18. 7 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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