Anton Gartner
- Aging top 0.02%
- Genetics, Aging, and Longevity in Model Organisms 58
- Molecular Biology top 0.5%
- DNA Repair Mechanisms 57
- Mitochondrial Function and Pathology 21
- CRISPR and Genetic Engineering 12
- Fungal and yeast genetics research 12
- Photosynthetic Processes and Mechanisms 7
- Cell Biology top 0.5%
- Microtubule and mitosis dynamics 13
- Geriatrics and Gerontology top 2%
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- Cancer Genomics and Diagnostics 7
- Co-authors
- Gustav AmmererMichael O. HengartnerShawn AhmedSimon J. BoultonSebastian GreissBjörn SchumacherKay HofmannKim Nasmyth
- Cited by
- AgingMolecular BiologyCell Biology
- Partner nations
- United KingdomUnited StatesGermany
In The Last Decade
Anton Gartner
105 papers receiving 7.6k citations
Peers
Comparison fields: 5 of 121
- Aging 2.5k
- Molecular Biology 6.0k
- Cell Biology 1.2k
- Endocrine and Autonomic Systems 381
- Geriatrics and Gerontology 143
Countries citing papers authored by Anton Gartner
This map shows the geographic impact of Anton Gartner's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Anton Gartner with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Anton Gartner more than expected).
Fields of papers citing papers by Anton Gartner
This network shows the impact of papers produced by Anton Gartner. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Anton Gartner. The network helps show where Anton Gartner may publish in the future.
Co-authorship network
The 25 scholars most cited alongside Anton Gartner, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2024 | 2 | |
| 2 | 2024 | 3 | |
| 3 | 2024 | 4 | |
| 4 | 2023 | 6 | |
| 5 | 2023 | 2 | |
| 6 | 2021 | 37 | |
| 7 | 2021 | 0 | |
| 8 | 2020 | 137 | |
| 9 | 2018 | 20 | |
| 10 | 2017 | 9 | |
| 11 | 2017 | 81 | |
| 12 | 2017 | 57 | |
| 13 | 2016 | 27 | |
| 14 | 2014 | 126 | |
| 15 | 2012 | 59 | |
| 16 | 2012 | 137 | |
| 17 | 2002 | 229 | |
| 18 | 2002 | 71 | |
| 19 | 2001 | 297 | |
| 20 | 1998 | 78 |
About Anton Gartner
Anton Gartner is a scholar working on Aging, Molecular Biology and Cell Biology, having authored 107 papers that have together received 7.7k indexed citations. Recurring topics across this work include Genetics, Aging, and Longevity in Model Organisms (58 papers), DNA Repair Mechanisms (57 papers), Mitochondrial Function and Pathology (21 papers), Microtubule and mitosis dynamics (13 papers), CRISPR and Genetic Engineering (12 papers), Fungal and yeast genetics research (12 papers), Cancer Genomics and Diagnostics (7 papers) and Photosynthetic Processes and Mechanisms (7 papers). The work is most often cited by research in Aging (2.5k citations), Molecular Biology (6.0k citations) and Cell Biology (1.2k citations). Anton Gartner has collaborated with scholars based in United Kingdom, United States and Germany. Frequent co-authors include Gustav Ammerer, Michael O. Hengartner, Shawn Ahmed, Simon J. Boulton, Sebastian Greiss, Björn Schumacher, Kay Hofmann, Kim Nasmyth, Stuart Milstein and Matthias Peter. Their work appears in journals such as PLoS Genetics, Current Biology, Nucleic Acids Research, Molecular and Cellular Biology and Cell.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.