Dalila Iannotta

618 citations
14 papers · 380 indexed · h-index 10
Topics
Extracellular vesicles in disease (8 papers)MicroRNA in disease regulation (6 papers)Peroxisome Proliferator-Activated Receptors (2 papers)

In The Last Decade

Dalila Iannotta

14 papers receiving 374 citations

Peers

Dalila Iannotta
Comparison fields: 5 of 86
  • Molecular Biology 251
  • Cancer Research 107
  • Biomedical Engineering 47
  • Immunology 40
  • Physiology 36
Replace Rita Chakrabarti with:
Rita Chakrabarti United States
Marie‐Sophie Narzt Austria
Wannita Klinngam United States
Rosa Mistica C. Ignacio South Korea
Zhenfei Bi China
Laura Cornaghi Italy
Jin Sun Hwang South Korea
Alžběta Filipová Czechia
Krystyna Stalińska Poland
Yu Su China
Dalila Iannotta relative to Rita Chakrabarti United States Rita Chakrabarti's profile →
Citations per field
00.5×10×16×
Rita Chakrabarti · 1×
Citations per year

Countries citing papers authored by Dalila Iannotta

Since Specialization
Citations

This map shows the geographic impact of Dalila Iannotta's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Dalila Iannotta with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Dalila Iannotta more than expected).

Fields of papers citing papers by Dalila Iannotta

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Dalila Iannotta. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Dalila Iannotta. The network helps show where Dalila Iannotta may publish in the future.

Co-authorship network of co-authors of Dalila Iannotta

This figure shows the co-authorship network connecting the top 25 collaborators of Dalila Iannotta. A scholar is included among the top collaborators of Dalila Iannotta based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Dalila Iannotta. Dalila Iannotta is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

14 of 14 papers shown
#WorkIndexed citations
1 1
2 9
3 17
4 1
5 14
6 70
7 4
8 41
9 19
10 46
11 57
12 28
13 24
14 49

About Dalila Iannotta

Dalila Iannotta is a scholar working on Cancer Research, Immunology and Allergy and Pharmaceutical Science, having authored 14 papers that have together received 380 indexed citations. Recurring topics across this work include Extracellular vesicles in disease (8 papers), MicroRNA in disease regulation (6 papers) and Peroxisome Proliferator-Activated Receptors (2 papers). The work is most often cited by research in Cancer Research (107 citations), Pharmaceutical Science (33 citations) and Immunology and Allergy (23 citations). Dalila Iannotta has collaborated with scholars based in United States, Australia and Italy. Frequent co-authors include Joy Wolfram, Luisa Di Marzio, Christian Celia, Amanda W. Kijas, Alan E. Rowan, Sierra A. Walker, Mariano Catanesi, Elisabetta Benedetti, Vanessa Castelli and Michele d’Angelo. Their work appears in journals such as Nature Nanotechnology, Advanced Drug Delivery Reviews and Langmuir.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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