Dora Games

17.2k total citations · 2 hit papers
55 papers, 7.0k citations indexed

About

Dora Games is a scholar working on Physiology, Cellular and Molecular Neuroscience and Neurology. According to data from OpenAlex, Dora Games has authored 55 papers receiving a total of 7.0k indexed citations (citations by other indexed papers that have themselves been cited), including 49 papers in Physiology, 23 papers in Cellular and Molecular Neuroscience and 17 papers in Neurology. Recurrent topics in Dora Games's work include Alzheimer's disease research and treatments (49 papers), Neuroinflammation and Neurodegeneration Mechanisms (16 papers) and Neuroscience and Neuropharmacology Research (14 papers). Dora Games is often cited by papers focused on Alzheimer's disease research and treatments (49 papers), Neuroinflammation and Neurodegeneration Mechanisms (16 papers) and Neuroscience and Neuropharmacology Research (14 papers). Dora Games collaborates with scholars based in United States, United Kingdom and Japan. Dora Games's co-authors include Dale Schenk, Peter Seubert, Eliezer Masliah, Bradley T. Hyman, Margaret Mallory, Karen Khan, Lisa McConlogue, Robin Barbour, Stephen B. Freedman and Lennart Mucke and has published in prestigious journals such as Nature, Proceedings of the National Academy of Sciences and Journal of Biological Chemistry.

In The Last Decade

Dora Games

55 papers receiving 6.8k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

A learning deficit related to age and β-amyloid plaques in a mouse model of Alzheimer's disease

2000 609 citations Guiquan Chen, Karen Chen et al. profile →
Amyloid precursor protein processing and Aβ ₄₂ deposition in a transgenic mouse model of Alzheimer disease

1997 553 citations Kelly Johnson‐Wood, Ruth Motter et al. profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Dora Games United States	41	5.4k	2.2k	2.2k	1.9k	1.4k	55	7.0k
Dorothée Abramowski Switzerland	32	5.4k 1.0×	2.5k 1.1×	3.3k 1.5×	1.8k 0.9×	1.1k 0.8×	48	7.8k
Chris Eckman United States	11	6.7k 1.2×	2.2k 1.0×	3.0k 1.3×	1.7k 0.9×	1.8k 1.3×	13	8.0k
Yasuo Harigaya Japan	27	5.5k 1.0×	1.8k 0.8×	2.6k 1.2×	1.3k 0.7×	1.3k 0.9×	77	6.9k
Lisa McConlogue United States	31	6.0k 1.1×	2.2k 1.0×	3.6k 1.6×	1.5k 0.8×	1.7k 1.2×	41	8.2k
M. Paul Murphy United States	43	5.8k 1.1×	2.1k 1.0×	3.4k 1.6×	1.9k 1.0×	1.4k 1.0×	75	8.9k
Antonella Caccamo United States	36	6.4k 1.2×	2.9k 1.3×	3.6k 1.7×	1.8k 1.0×	2.0k 1.5×	56	9.6k
Oliver Wirths Germany	46	4.9k 0.9×	1.9k 0.9×	2.3k 1.1×	1.4k 0.8×	1.1k 0.8×	120	6.7k
Karl‐Heinz Wiederhold Switzerland	32	3.7k 0.7×	2.5k 1.1×	2.1k 1.0×	1.4k 0.8×	817 0.6×	48	6.3k
Gui-Qiu Yu United States	26	4.8k 0.9×	2.9k 1.3×	2.1k 0.9×	1.6k 0.8×	1.1k 0.8×	33	7.0k
Hilda H. Slunt United States	31	5.5k 1.0×	2.3k 1.1×	3.9k 1.8×	960 0.5×	1.5k 1.1×	39	8.0k

Countries citing papers authored by Dora Games

Since Specialization

Citations

This map shows the geographic impact of Dora Games's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Dora Games with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Dora Games more than expected).

Fields of papers citing papers by Dora Games

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Dora Games. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Dora Games. The network helps show where Dora Games may publish in the future.

Co-authorship network of co-authors of Dora Games

This figure shows the co-authorship network connecting the top 25 collaborators of Dora Games. A scholar is included among the top collaborators of Dora Games based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Dora Games. Dora Games is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Zago, Wagner, Manuel Buttini, Thomas A. Comery, et al.. (2012). Neutralization of Soluble, Synaptotoxic Amyloid β Species by Antibodies Is Epitope Specific. Journal of Neuroscience. 32(8). 2696–2702. 53 indexed citations

Schroeter, Sally, Elizabeth Brigham, Ruth Motter, et al.. (2010). P3‐321: APP‐selective gamma secretase inhibitor ELND006 effects on brain parenchymal and vascular amyloid beta in the PDAPP mouse model of Alzheimer's disease. Alzheimer s & Dementia. 6(4S_Part_18). 3 indexed citations

Seubert, Peter, Robin Barbour, Karen Khan, et al.. (2008). Antibody Capture of Soluble Aβ Does Not Reduce Cortical Aβ Amyloidosis in the PDAPP Mouse. Neurodegenerative Diseases. 5(2). 65–71. 55 indexed citations

McConlogue, Lisa, Manuel Buttini, John P. Anderson, et al.. (2007). Partial Reduction of BACE1 Has Dramatic Effects on Alzheimer Plaque and Synaptic Pathology in APP Transgenic Mice. Journal of Biological Chemistry. 282(36). 26326–26334. 231 indexed citations

Chen, Guiquan, Karen Chen, Dione Kobayashi, et al.. (2007). Active β-Amyloid Immunization Restores Spatial Learning in PDAPP Mice Displaying Very Low Levels of β-Amyloid. Journal of Neuroscience. 27(10). 2654–2662. 41 indexed citations

Buckwalter, Marion S., Manuel Buttini, Robin Barbour, et al.. (2006). Increased T Cell Recruitment to the CNS after Amyloid β_1–42Immunization in Alzheimer's Mice Overproducing Transforming Growth Factor-β1. Journal of Neuroscience. 26(44). 11437–11441. 41 indexed citations

German, Dwight C., et al.. (2005). The PDAPP mouse model of Alzheimer's disease: Locus coeruleus neuronal shrinkage. The Journal of Comparative Neurology. 492(4). 469–476. 43 indexed citations

Masliah, Eliezer, Edward Rockenstein, Anthony Adame, et al.. (2005). Effects of α-Synuclein Immunization in a Mouse Model of Parkinson’s Disease. Neuron. 46(6). 857–868. 423 indexed citations

Valla, J., Kewei Chen, Jason D. Berndt, et al.. (2002). Effects of Image Resolution on Autoradiographic Measurements of Posterior Cingulate Activity in PDAPP Mice: Implications for Functional Brain Imaging Studies of Transgenic Mouse Models of Alzheimer's Disease. NeuroImage. 16(1). 1–6. 24 indexed citations

10.

Huitrón‐Reséndiz, Salvador, Manuel Sánchez-Alavez, Roger A. Gallegos, et al.. (2002). Age-independent and age-related deficits in visuospatial learning, sleep–wake states, thermoregulation and motor activity in PDAPP mice. Brain Research. 928(1-2). 126–137. 85 indexed citations

11.

Oshima, Noriko, Maho Morishima‐Kawashima, Haruyasu Yamaguchi, et al.. (2001). Accumulation of Amyloid β-Protein in the Low-Density Membrane Domain Accurately Reflects the Extent of β-Amyloid Deposition in the Brain. American Journal Of Pathology. 158(6). 2209–2218. 41 indexed citations

12.

Masliah, Eliezer, et al.. (2001). Neurofibrillary Pathology in Transgenic Mice Overexpressing V717F β-Amyloid Precursor Protein. Journal of Neuropathology & Experimental Neurology. 60(4). 357–368. 99 indexed citations

13.

Thomas, Elizabeth A., et al.. (2001). Apolipoprotein D mRNA expression is elevated in PDAPP transgenic mice. Journal of Neurochemistry. 79(5). 1059–1064. 23 indexed citations

14.

Schenk, Dale, Dora Games, & Peter Seubert. (2001). Potential Treatment Opportunities for Alzheimer's Disease Through Inhibition of Secretases and Aβ Immunization. Journal of Molecular Neuroscience. 17(2). 259–267. 31 indexed citations

15.

Gonzalez‐Lima, F., Jason D. Berndt, J. Valla, Dora Games, & Eric M. Reiman. (2001). Reduced corpus callosum, fornix and hippocampus in PDAPP transgenic mouse model of Alzheimer's disease. Neuroreport. 12(11). 2375–2379. 54 indexed citations

16.

Diez, Margarita, et al.. (2000). Neuropeptides in hippocampus and cortex in transgenic mice overexpressing V717F β-amyloid precursor protein — initial observations. Neuroscience. 100(2). 259–286. 69 indexed citations

17.

Morishima‐Kawashima, Maho, Masahiro Yoshimura, Kaoru Kusui, et al.. (1999). Appearance of Sodium Dodecyl Sulfate-Stable Amyloid β-Protein (Aβ) Dimer in the Cortex During Aging. American Journal Of Pathology. 154(1). 271–279. 86 indexed citations

18.

Masliah, Eliezer, et al.. (1996). Comparison of Neurodegenerative Pathology in Transgenic Mice Overexpressing V717F β-Amyloid Precursor Protein and Alzheimer’s Disease. Journal of Neuroscience. 16(18). 5795–5811. 274 indexed citations

19.

Games, Dora, Karen Khan, Ferdie Soriano, et al.. (1992). Lack of alzheimer pathology after β-amyloid protein injections in rat brain. Neurobiology of Aging. 13(5). 569–576. 112 indexed citations

20.

Games, Dora, Catharine Joachim, Pamela J. Ward, et al.. (1989). ANTIBODIES TO DISTINCT REGIONS OF β-AMYLOID PRECURSOR PROTEIN (βAPP) DIFFERENTIALLY LABEL ALZHEIMER DISEASE (AD) SENILE PLAQUES. Journal of Neuropathology & Experimental Neurology. 48(3). 330–330. 3 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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