Conrad L. Cowan

2.3k total citations · 2 hit papers
21 papers, 1.9k citations indexed

About

Conrad L. Cowan is a scholar working on Molecular Biology, Physiology and Cellular and Molecular Neuroscience. According to data from OpenAlex, Conrad L. Cowan has authored 21 papers receiving a total of 1.9k indexed citations (citations by other indexed papers that have themselves been cited), including 16 papers in Molecular Biology, 9 papers in Physiology and 7 papers in Cellular and Molecular Neuroscience. Recurrent topics in Conrad L. Cowan's work include Receptor Mechanisms and Signaling (15 papers), Neuropeptides and Animal Physiology (7 papers) and Nitric Oxide and Endothelin Effects (5 papers). Conrad L. Cowan is often cited by papers focused on Receptor Mechanisms and Signaling (15 papers), Neuropeptides and Animal Physiology (7 papers) and Nitric Oxide and Endothelin Effects (5 papers). Conrad L. Cowan collaborates with scholars based in United States, France and Italy. Conrad L. Cowan's co-authors include Richard A. Cohen, Jonathan D. Violin, Michael W. Lark, David H. Rominger, Michael Koblish, James Palacino, Soheil Najibi, Catherine Yuan, Dimitar B. Gotchev and Dennis S. Yamashita and has published in prestigious journals such as Journal of Biological Chemistry, Circulation and Journal of the American College of Cardiology.

In The Last Decade

Conrad L. Cowan

21 papers receiving 1.9k citations

Hit Papers

A G Protein-Biased Ligand at the μ-Opioid Receptor Is Po... 2006 2026 2012 2019 2013 2006 100 200 300 400 500

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Conrad L. Cowan United States 18 1.3k 946 495 285 157 21 1.9k
Hervé Paris France 21 1.0k 0.8× 621 0.7× 271 0.5× 105 0.4× 111 0.7× 43 1.5k
David H. Rominger United States 15 1.6k 1.2× 1.2k 1.2× 227 0.5× 193 0.7× 135 0.9× 18 2.0k
Kenneth E.J. Dickinson United States 22 845 0.7× 456 0.5× 324 0.7× 325 1.1× 114 0.7× 61 1.5k
Takafumi Nagatomo Japan 19 733 0.6× 461 0.5× 271 0.5× 265 0.9× 109 0.7× 138 1.4k
Jason E. Savage United States 19 1.3k 1.0× 553 0.6× 377 0.8× 135 0.5× 117 0.7× 38 2.6k
Bryan F. Cox United States 27 1.0k 0.8× 316 0.3× 239 0.5× 702 2.5× 149 0.9× 85 1.8k
Michael J. Forrest United States 21 786 0.6× 367 0.4× 778 1.6× 60 0.2× 111 0.7× 37 2.0k
Trine Meldgaard Lund Denmark 21 459 0.4× 345 0.4× 322 0.7× 132 0.5× 118 0.8× 68 1.3k
Stéphane Schaak France 16 654 0.5× 328 0.3× 114 0.2× 70 0.2× 210 1.3× 32 1.1k
Ricardo Caballero Spain 34 1.6k 1.2× 421 0.4× 180 0.4× 2.0k 7.1× 138 0.9× 103 2.8k

Countries citing papers authored by Conrad L. Cowan

Since Specialization
Citations

This map shows the geographic impact of Conrad L. Cowan's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Conrad L. Cowan with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Conrad L. Cowan more than expected).

Fields of papers citing papers by Conrad L. Cowan

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Conrad L. Cowan. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Conrad L. Cowan. The network helps show where Conrad L. Cowan may publish in the future.

Co-authorship network of co-authors of Conrad L. Cowan

This figure shows the co-authorship network connecting the top 25 collaborators of Conrad L. Cowan. A scholar is included among the top collaborators of Conrad L. Cowan based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Conrad L. Cowan. Conrad L. Cowan is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Ryba, David M., Jieli Li, Conrad L. Cowan, et al.. (2017). Long-Term Biased β-Arrestin Signaling Improves Cardiac Structure and Function in Dilated Cardiomyopathy. Circulation. 135(11). 1056–1070. 72 indexed citations
2.
Koblish, Michael, Richard W. Carr, Edward R. Siuda, et al.. (2017). TRV0109101, a G Protein-Biased Agonist of the µ-Opioid Receptor, Does Not Promote Opioid-Induced Mechanical Allodynia following Chronic Administration. Journal of Pharmacology and Experimental Therapeutics. 362(2). 254–262. 27 indexed citations
3.
Davis, Robert T., Paul T. Mungai, David M. Ryba, et al.. (2015). Cardiac myosin light chain phosphorylation and inotropic effects of a biased ligand, TRV120023, in a dilated cardiomyopathy model. Cardiovascular Research. 107(2). 226–234. 28 indexed citations
4.
Rominger, David H., Conrad L. Cowan, William Gowen-MacDonald, & Jonathan D. Violin. (2014). Biased ligands: pathway validation for novel GPCR therapeutics. Current Opinion in Pharmacology. 16. 108–115. 25 indexed citations
5.
Soergel, David G., Ruth Ann Subach, Ian E. James, et al.. (2013). TRVO27, A BETA-ARRESTIN BIASED LIGAND AT THE ANGIOTENSIN 2 TYPE 1 RECEPTOR, PRODUCES RAPID, REVERSIBLE CHANGES IN HEMODYNAMICS IN PATIENTS WITH STABLE SYSTOLIC HEART FAILURE. Journal of the American College of Cardiology. 61(10). E683–E683. 12 indexed citations
6.
DeWire, Scott M., Dennis S. Yamashita, David H. Rominger, et al.. (2013). A G Protein-Biased Ligand at the μ-Opioid Receptor Is Potently Analgesic with Reduced Gastrointestinal and Respiratory Dysfunction Compared with Morphine. Journal of Pharmacology and Experimental Therapeutics. 344(3). 708–717. 502 indexed citations breakdown →
7.
Soergel, David G., Ruth Ann Subach, Conrad L. Cowan, Jonathan D. Violin, & Michael W. Lark. (2013). First Clinical Experience with TRV027: Pharmacokinetics and Pharmacodynamics in Healthy Volunteers. The Journal of Clinical Pharmacology. 53(9). 892–899. 68 indexed citations
9.
Soergel, David G., Ruth Ann Subach, Brian M. Sadler, et al.. (2013). First Clinical Experience With TRV130: Pharmacokinetics and Pharmacodynamics in Healthy Volunteers. The Journal of Clinical Pharmacology. 54(3). 351–357. 103 indexed citations
10.
Shearer, Barry G., Esther Y. Chao, David Uehling, et al.. (2007). Synthesis and evaluation of potent and selective β3 adrenergic receptor agonists containing heterobiaryl carboxylic acids. Bioorganic & Medicinal Chemistry Letters. 17(16). 4670–4677. 15 indexed citations
11.
Uehling, David, Barry G. Shearer, Esther Y. Chao, et al.. (2006). Biarylaniline Phenethanolamines as Potent and Selective β3Adrenergic Receptor Agonists. Journal of Medicinal Chemistry. 49(9). 2758–2771. 33 indexed citations
12.
Gesty‐Palmer, Diane, Minyong Chen, Éric Reiter, et al.. (2006). Distinct β-Arrestin- and G Protein-dependent Pathways for Parathyroid Hormone Receptor-stimulated ERK1/2 Activation. Journal of Biological Chemistry. 281(16). 10856–10864. 372 indexed citations breakdown →
13.
Ghosh, Richik N., et al.. (2005). Quantitative Cell-Based High-Content Screening for Vasopressin Receptor Agonists Using Transfluor®Technology. SLAS DISCOVERY. 10(5). 476–484. 57 indexed citations
14.
Uehling, David, David N. Deaton, Elizabeth E. Sugg, et al.. (2001). Synthesis and Evaluation of Potent and Selective β3 Adrenergic Receptor Agonists Containing Acylsulfonamide, Sulfonylsulfonamide, and Sulfonylurea Carboxylic Acid Isosteres. Journal of Medicinal Chemistry. 45(3). 567–583. 48 indexed citations
15.
Norman, Mark H., et al.. (1995). 4-(Heteroarylthio)-2-biphenylyltetrazoles as Nonpeptide Angiotensin II Antagonists. Journal of Medicinal Chemistry. 38(23). 4670–4678. 7 indexed citations
16.
Cowan, Conrad L. & Robert Steffen. (1995). Lysophosphatidylcholine Inhibits Relaxation of Rabbit Abdominal Aorta Mediated by Endothelium-Derived Nitric Oxide and Endothelium-Derived Hyperpolarizing Factor Independent of Protein Kinase C Activation. Arteriosclerosis Thrombosis and Vascular Biology. 15(12). 2290–2297. 46 indexed citations
17.
Najibi, Soheil, Conrad L. Cowan, James Palacino, & Richard A. Cohen. (1994). Enhanced role of potassium channels in relaxations to acetylcholine in hypercholesterolemic rabbit carotid artery. American Journal of Physiology-Heart and Circulatory Physiology. 266(5). H2061–H2067. 111 indexed citations
18.
Cowan, Conrad L., James Palacino, Soheil Najibi, & Richard A. Cohen. (1993). Potassium channel-mediated relaxation to acetylcholine in rabbit arteries.. Journal of Pharmacology and Experimental Therapeutics. 266(3). 1482–1489. 104 indexed citations
19.
Cowan, Conrad L. & Richard A. Cohen. (1991). Two mechanisms mediate relaxation by bradykinin of pig coronary artery: NO-dependent and -independent responses. American Journal of Physiology-Heart and Circulatory Physiology. 261(3). H830–H835. 79 indexed citations
20.
Cowan, Conrad L. & Jack E. McKenzie. (1990). Cholinergic regulation of resting coronary blood flow in domestic swine. American Journal of Physiology-Heart and Circulatory Physiology. 259(1). H109–H115. 23 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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