Christopher Tudan

810 citations
16 papers · 697 · h-index 12

Impact in

Papers in

    • Protein Kinase Regulation and GTPase Signaling 5
    • Cell death mechanisms and regulation 5
    • Cancer therapeutics and mechanisms 2
    • Melanoma and MAPK Pathways 2
    • Cytokine Signaling Pathways and Interactions 2
    • Peptidase Inhibition and Analysis 1

Christopher Tudan

16 papers receiving 687 citations

Peers

Christopher Tudan
Comparison fields: 5 of 80
  • Developmental Neuroscience 57
  • Cellular and Molecular Neuroscience 155
  • Cell Biology 115
  • Molecular Biology 462
  • Nephrology 45
Replace Arthur G. Balliet with:
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Citations per field
00.5×7.5×
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Citations per year

Countries citing papers authored by Christopher Tudan

Since Specialization
Citations

This map shows the geographic impact of Christopher Tudan's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Christopher Tudan with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Christopher Tudan more than expected).

Fields of papers citing papers by Christopher Tudan

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Christopher Tudan. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Christopher Tudan. The network helps show where Christopher Tudan may publish in the future.

Co-authors

The 25 scholars most cited alongside Christopher Tudan, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Christopher Tudan Line = papers co-authored together Christopher Tudan links everyone, so they are left out of the graph.

All Works

16 of 16 papers shown
#Work
1 1999205
2 1995136
3
Antitumor drug fostriecin inhibits the mitotic entry checkpoint and protein phosphatases 1 and 2A.
199482
4
Suppression of Rac activity induces apoptosis of human glioma cells but not normal human astrocytes.
200282
5 199730
6 200229
7 200028
8
The inhibition of spontaneous and tumor necrosis factor-alpha induced neutrophil apoptosis by crystals of calcium pyrophosphate dihydrate and monosodium urate monohydrate.
200021
9 199821
10
The involvement of phospholipase C in crystal induced human neutrophil activation.
200016
11 200312
12 200311
13 20029
14 19998
15 20096
16 20021

About Christopher Tudan

Christopher Tudan is a scholar working on Molecular Biology, Oncology, Immunology, Pathology and Forensic Medicine and Nutrition and Dietetics, having authored 16 papers that have together received 697 indexed citations. Recurring topics across this work include Protein Kinase Regulation and GTPase Signaling (5 papers), Cell death mechanisms and regulation (5 papers), Neutrophil, Myeloperoxidase and Oxidative Mechanisms (3 papers), Cytokine Signaling Pathways and Interactions (2 papers), Cancer therapeutics and mechanisms (2 papers), Melanoma and MAPK Pathways (2 papers), Vitamin C and Antioxidants Research (2 papers) and Peptidase Inhibition and Analysis (1 paper). The work is most often cited by research in Developmental Neuroscience (57 citations), Cellular and Molecular Neuroscience (155 citations), Cell Biology (115 citations), Molecular Biology (462 citations) and Nephrology (45 citations). Christopher Tudan has collaborated with scholars based in Canada, United States and Czechia. Frequent co-authors include David R. Kaplan, Irene Mazzoni, Andrew Vaillant, Mathieu Boudreau, Freda D. Miller, Hana Anderson, Helen M. Burt, Michel Roberge, John K. Jackson and Steven Pelech. Their work appears in journals such as Inflammation Research, The Journal of Immunology, The EMBO Journal, Immunology and The Journal of Cell Biology.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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