Christopher J. Hupfeld

980 total citations
15 papers, 765 citations indexed

About

Christopher J. Hupfeld is a scholar working on Molecular Biology, Endocrinology, Diabetes and Metabolism and Cellular and Molecular Neuroscience. According to data from OpenAlex, Christopher J. Hupfeld has authored 15 papers receiving a total of 765 indexed citations (citations by other indexed papers that have themselves been cited), including 11 papers in Molecular Biology, 5 papers in Endocrinology, Diabetes and Metabolism and 4 papers in Cellular and Molecular Neuroscience. Recurrent topics in Christopher J. Hupfeld's work include Protein Kinase Regulation and GTPase Signaling (7 papers), Receptor Mechanisms and Signaling (6 papers) and Metabolism, Diabetes, and Cancer (5 papers). Christopher J. Hupfeld is often cited by papers focused on Protein Kinase Regulation and GTPase Signaling (7 papers), Receptor Mechanisms and Signaling (6 papers) and Metabolism, Diabetes, and Cancer (5 papers). Christopher J. Hupfeld collaborates with scholars based in United States. Christopher J. Hupfeld's co-authors include Jerrold M. Olefsky, Jin Zhang, Susan S. Taylor, Roger Y. Tsien, Takeshi Imamura, Isao Usui, Hiroaki Satoh, Jennie L. Babendure, Stéphane Dalle and Satoshi Ugi and has published in prestigious journals such as Nature, Proceedings of the National Academy of Sciences and Journal of Biological Chemistry.

In The Last Decade

Christopher J. Hupfeld

14 papers receiving 760 citations

Peers

Countries citing papers authored by Christopher J. Hupfeld

Since Specialization

Citations

This map shows the geographic impact of Christopher J. Hupfeld's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Christopher J. Hupfeld with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Christopher J. Hupfeld more than expected).

Fields of papers citing papers by Christopher J. Hupfeld

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Christopher J. Hupfeld. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Christopher J. Hupfeld. The network helps show where Christopher J. Hupfeld may publish in the future.

Co-authorship network of co-authors of Christopher J. Hupfeld

This figure shows the co-authorship network connecting the top 25 collaborators of Christopher J. Hupfeld. A scholar is included among the top collaborators of Christopher J. Hupfeld based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Christopher J. Hupfeld. Christopher J. Hupfeld is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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15 of 15 papers shown

#	Work	Indexed citations
1	SGLT2 Inhibitor–Induced Low-Grade Ketonemia Ameliorates Retinal Hypoxia in Diabetic Retinopathy—A Novel Hypothesis 2021 ·The Journal of Clinical Endocrinology & Metabolism ·Sunder Mudaliar, Christopher J. Hupfeld, Daniel L. Chao	29
2	Sodium-Glucose Cotransporter Type 2 (SGLT-2) Inhibitors and Ketogenesis: the Good and the Bad 2020 ·Current Diabetes Reports ·(unknown), Christopher J. Hupfeld, Sunder Mudaliar	23
3	Navigating the “MACE” in Cardiovascular Outcomes Trials and decoding the relevance of Atherosclerotic Cardiovascular Disease benefits versus Heart Failure benefits 2019 ·Diabetes Obesity and Metabolism ·Christopher J. Hupfeld, Sunder Mudaliar	27
4	Laktatwerte im Blut gesunder und kranker Kaninchen 2009 ·Tierärztliche Praxis Ausgabe K Kleintiere / Heimtiere ·Christopher J. Hupfeld	2
5	Regulation of Receptor Tyrosine Kinase Signaling by GRKs and β-Arrestins 2006 ·Annual Review of Physiology ·Christopher J. Hupfeld, Jerrold M. Olefsky	65
6	Insulin disrupts β-adrenergic signalling to protein kinase A in adipocytes 2005 ·Nature ·Jin Zhang, Christopher J. Hupfeld, Susan S. Taylor, Jerrold M. Olefsky, Roger Y. Tsien	250
7	G Protein-Coupled Receptor Kinase 2 Mediates Endothelin-1-Induced Insulin Resistance via the Inhibition of Both Gαq/11 and Insulin Receptor Substrate-1 Pathways in 3T3-L1 Adipocytes 2005 ·Molecular Endocrinology ·Isao Usui, Takeshi Imamura, Jennie L. Babendure, Hiroaki Satoh, (unknown), Christopher J. Hupfeld, Jerrold M. Olefsky	71
8	GRK2 is an endogenous protein inhibitor of the insulin signaling pathway for glucose transport stimulation 2004 ·The EMBO Journal ·Isao Usui, Takeshi Imamura, Hiroaki Satoh, Jie Huang, Jennie L. Babendure, Christopher J. Hupfeld, Jerrold M. Olefsky	75
9	Insulin-induced β-Arrestin1 Ser-412 Phosphorylation Is a Mechanism for Desensitization of ERK Activation by Gαi-coupled Receptors 2004 ·Journal of Biological Chemistry ·Christopher J. Hupfeld, Jamie L. Resnik, Satoshi Ugi, Jerrold M. Olefsky	39
10	β-Arrestin-1 Competitively Inhibits Insulin-Induced Ubiquitination and Degradation of Insulin Receptor Substrate 1 2004 ·Molecular and Cellular Biology ·Isao Usui, Takeshi Imamura, Jie Huang, Hiroaki Satoh, Sudha K. Shenoy, Robert J. Lefkowitz, Christopher J. Hupfeld, Jerrold M. Olefsky	57
11	Current Recommendations for Prevention of Cardiovascular Disease in Patients With Diabetes Mellitus 2003 ·Preventive Cardiology ·Christopher J. Hupfeld, Gail A. Wong	0
12	Molecular Mechanisms of Diabetic Cardiovascular Disease 2002 ·Preventive Cardiology ·Christopher J. Hupfeld, Gail A. Wong	1
13	β-Arrestin 1 down-regulation after insulin treatment is associated with supersensitization of β2 adrenergic receptor Gαs signaling in 3T3-L1 adipocytes 2002 ·Proceedings of the National Academy of Sciences ·Christopher J. Hupfeld, Stéphane Dalle, Jerrold M. Olefsky	35
14	Insulin Induces Heterologous Desensitization of G Protein-Coupled Receptor and Insulin-Like Growth Factor I Signaling by Downregulating β-Arrestin-1 2002 ·Molecular and Cellular Biology ·Stéphane Dalle, Takeshi Imamura, David W. Rose, Dorothy Sears Worrall, Satoshi Ugi, Christopher J. Hupfeld, Jerrold M. Olefsky	67
15	TZDs inhibit vascular smooth muscle cell growth independently of the cyclin kinase inhibitors p21 and p27 2001 ·American Journal of Physiology-Endocrinology and Metabolism ·Christopher J. Hupfeld, Robert H. Weiss	24

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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