Jillian K. Cooper

3.2k total citations · 1 hit paper
16 papers, 2.5k citations indexed

About

Jillian K. Cooper is a scholar working on Molecular Biology, Cellular and Molecular Neuroscience and Clinical Biochemistry. According to data from OpenAlex, Jillian K. Cooper has authored 16 papers receiving a total of 2.5k indexed citations (citations by other indexed papers that have themselves been cited), including 15 papers in Molecular Biology, 10 papers in Cellular and Molecular Neuroscience and 2 papers in Clinical Biochemistry. Recurrent topics in Jillian K. Cooper's work include Genetic Neurodegenerative Diseases (10 papers), Mitochondrial Function and Pathology (8 papers) and Muscle Physiology and Disorders (5 papers). Jillian K. Cooper is often cited by papers focused on Genetic Neurodegenerative Diseases (10 papers), Mitochondrial Function and Pathology (8 papers) and Muscle Physiology and Disorders (5 papers). Jillian K. Cooper collaborates with scholars based in United States, United Kingdom and Japan. Jillian K. Cooper's co-authors include Matthew F. Peters, Christopher A. Ross, Frederick C. Nucifora, Christopher A. Ross, Valina L. Dawson, Ted M. Dawson, Hitoshi Takahashi, Shoji Tsuji, Mitsunori Yamada and Hui Huang and has published in prestigious journals such as Science, Nature Medicine and Neuron.

In The Last Decade

Jillian K. Cooper

16 papers receiving 2.5k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Interference by Huntingtin and Atrophin-1 with CBP-Mediated Transcription Leading to Cellular Toxicity

2001 880 citations Frederick C. Nucifora, Masayuki Sasaki et al. Science profile →

Peers

Jillian K. Cooper

CMN

NEURO

GENET

Michael A. Kalchman Canada

Shuichi Igarashi Japan

Christopher J. Cummings United States

Tammy Gillis United States

Makoto Minamiyama Japan

Eric N. Burright United States

Bénédicte C. Charrin France

Crystal N. Doty Canada

Jong‐Min Lee United States

Jim Dompierre France

Michael A. Kalchman Canada

Jillian K. Cooper

2.1k ×1.0

2.0k ×1.0

CMN

790 ×1.2

NEURO

147 ×0.6

GENET

208 ×1.3

Citations per year, relative to Jillian K. Cooper Jillian K. Cooper (= 1×) peers Michael A. Kalchman

Countries citing papers authored by Jillian K. Cooper

Since Specialization

Citations

This map shows the geographic impact of Jillian K. Cooper's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Jillian K. Cooper with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Jillian K. Cooper more than expected).

Fields of papers citing papers by Jillian K. Cooper

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Jillian K. Cooper. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Jillian K. Cooper. The network helps show where Jillian K. Cooper may publish in the future.

Co-authorship network of co-authors of Jillian K. Cooper

This figure shows the co-authorship network connecting the top 25 collaborators of Jillian K. Cooper. A scholar is included among the top collaborators of Jillian K. Cooper based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Jillian K. Cooper. Jillian K. Cooper is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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16 of 16 papers shown

Salamat, Muhammad Khalid Farooq, et al.. (2024). Longitudinal detection of prion infection in preclinical sheep blood samples compared using 3 assays. Blood. 144(18). 1962–1973. 3 indexed citations

Dong, Michael, Frances Mae West, Jillian K. Cooper, Jonathan D. Foster, & Rebecca Davis. (2023). A Guide to Point of Care Ultrasound Examination of a Pericardial Effusion. The Medicine Forum. 24(1). 3 indexed citations

Wilde, Lindsay, et al.. (2019). Clinical, Cytogenetic, and Molecular Findings in Two Cases of Variant t(8;21) Acute Myeloid Leukemia (AML). Frontiers in Oncology. 9. 4 indexed citations

Cooper, Jillian K., et al.. (2013). Evaluation of a test for its suitability in the diagnosis of variant Creutzfeldt–Jakob disease. Vox Sanguinis. 105(3). 196–204. 7 indexed citations

Cooper, Jillian K., et al.. (2011). Comparison of candidate vCJD in vitro diagnostic assays using identical sample sets. Vox Sanguinis. 102(2). 100–109. 7 indexed citations

Schilling, Gabriele, Alexandra M. Klevytska, Andrew T.N. Tebbenkamp, et al.. (2007). Characterization of Huntingtin Pathologic Fragments in Human Huntington Disease, Transgenic Mice, and Cell Models. Journal of Neuropathology & Experimental Neurology. 66(4). 313–320. 74 indexed citations

Cooper, Jillian K., et al.. (2007). Reference materials for the evaluation of pre‐mortem variant Creutzfeldt–Jakob disease diagnostic assays. Vox Sanguinis. 92(4). 302–310. 8 indexed citations

Igarashi, Shuichi, Hokuto Morita, Yuji Tanaka, et al.. (2003). Inducible PC12 cell model of Huntington’s disease shows toxicity and decreased histone acetylation. Neuroreport. 14(4). 565–568. 58 indexed citations

Nucifora, Frederick C., Masayuki Sasaki, Matthew F. Peters, et al.. (2001). Interference by Huntingtin and Atrophin-1 with CBP-Mediated Transcription Leading to Cellular Toxicity. Science. 291(5512). 2423–2428. 880 indexed citations breakdown →

10.

Rigamonti, Dorotea, Johannes H. Bauer, Claudio De‐Fraja, et al.. (2000). Wild-Type Huntingtin Protects from Apoptosis Upstream of Caspase-3. Journal of Neuroscience. 20(10). 3705–3713. 312 indexed citations

11.

Schilling, Gabriele, Jonathan Wood, Kui Duan, et al.. (1999). Nuclear Accumulation of Truncated Atrophin-1 Fragments in a Transgenic Mouse Model of DRPLA. Neuron. 24(1). 275–286. 143 indexed citations

12.

Sawa, Akira, Gordon Wiegand, Jillian K. Cooper, et al.. (1999). Increased apoptosis of Huntington disease lymphoblasts associated with repeat length-dependent mitochondrial depolarization. Nature Medicine. 5(10). 1194–1198. 468 indexed citations

13.

Peters, Matthew F., Frederick C. Nucifora, Jillian K. Cooper, et al.. (1999). Nuclear Targeting of Mutant Huntingtin Increases Toxicity. Molecular and Cellular Neuroscience. 14(2). 121–128. 154 indexed citations

14.

Ross, Christopher A., Jonathan Wood, Gabriele Schilling, et al.. (1999). Polyglutamine pathogenesis. Philosophical Transactions of the Royal Society B Biological Sciences. 354(1386). 1005–1011. 60 indexed citations

15.

Cooper, Jillian K.. (1998). Truncated N-terminal fragments of huntingtin with expanded glutamine repeats form nuclear and cytoplasmic aggregates in cell culture. Human Molecular Genetics. 7(5). 783–790. 312 indexed citations

16.

Margolis, Russell L., et al.. (1998). Chapter 28 Pathogenesis of neurodegenerative diseases associated with expanded glutamine repeats: New answers, new questions. Progress in brain research. 117. 397–419. 31 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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