Christine Kretz
- Genetics top 0.5%
- Genetics and Neurodevelopmental Disorders 8
- Cell Biology top 1%
- Cellular transport and secretion 24
- Microtubule and mitosis dynamics 5
- Cognitive Neuroscience top 2%
- Molecular Biology top 2%
- Ubiquitin and proteasome pathways 10
- Muscle Physiology and Disorders 6
- RNA modifications and cancer 5
- Mitochondrial Function and Pathology 3
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- Genetic Neurodegenerative Diseases 6
- Co-authors
- Jean‐Louis MandelI. OberléDominique HeitzJocelyn LaporteDidier DevysJ BouéAndré HanauerM. F. Bertheas
- Partner nations
- FranceUnited StatesSweden
In The Last Decade
Christine Kretz
39 papers receiving 4.6k citations
Hit Papers
Peers
Comparison fields: 5 of 94
- Genetics 2.3k
- Cell Biology 1.1k
- Cognitive Neuroscience 1.1k
- Molecular Biology 3.4k
- Cellular and Molecular Neuroscience 810
Countries citing papers authored by Christine Kretz
This map shows the geographic impact of Christine Kretz's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Christine Kretz with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Christine Kretz more than expected).
Fields of papers citing papers by Christine Kretz
This network shows the impact of papers produced by Christine Kretz. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Christine Kretz. The network helps show where Christine Kretz may publish in the future.
Co-authorship network
The 25 scholars most cited alongside Christine Kretz, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2023 | 8 | |
| 2 | 2021 | 25 | |
| 3 | 2021 | 13 | |
| 4 | 2020 | 25 | |
| 5 | 2020 | 21 | |
| 6 | 2018 | 35 | |
| 7 | 2013 | 43 | |
| 8 | 2012 | 47 | |
| 9 | 2010 | 26 | |
| 10 | 2009 | 142 | |
| 11 | 2009 | 17 | |
| 12 | 2009 | 2 | |
| 13 | 2007 | 284 | |
| 14 | 2006 | 108 | |
| 15 | 1998 | 97 | |
| 16 | 1996 | 25 | |
| 17 | 1994 | 65 | |
| 18 | 1994 | 202 | |
| 19 | 1993 | 168 | |
| 20 | 1989 | 20 |
About Christine Kretz
Christine Kretz is a scholar working on Cell Biology, Cellular and Molecular Neuroscience and Genetics, having authored 41 papers that have together received 4.7k indexed citations. Recurring topics across this work include Cellular transport and secretion (24 papers), Ubiquitin and proteasome pathways (10 papers), Genetics and Neurodevelopmental Disorders (8 papers), Muscle Physiology and Disorders (6 papers), Genetic Neurodegenerative Diseases (6 papers), Microtubule and mitosis dynamics (5 papers), RNA modifications and cancer (5 papers) and Mitochondrial Function and Pathology (3 papers). The work is most often cited by research in Genetics (2.3k citations), Cell Biology (1.1k citations) and Cognitive Neuroscience (1.1k citations). Christine Kretz has collaborated with scholars based in France, United States and Sweden. Frequent co-authors include Jean‐Louis Mandel, I. Oberlé, Dominique Heitz, Jocelyn Laporte, Didier Devys, J Boué, André Hanauer, M. F. Bertheas, F. Rousseau and Niklas Dahl. Their work appears in journals such as Human Molecular Genetics, Nature Genetics, Molecular Therapy, Neuromuscular Disorders and Proceedings of the National Academy of Sciences.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.