Isabelle Marty

4.8k total citations
106 papers, 3.5k citations indexed

About

Isabelle Marty is a scholar working on Molecular Biology, Cardiology and Cardiovascular Medicine and Cellular and Molecular Neuroscience. According to data from OpenAlex, Isabelle Marty has authored 106 papers receiving a total of 3.5k indexed citations (citations by other indexed papers that have themselves been cited), including 90 papers in Molecular Biology, 34 papers in Cardiology and Cardiovascular Medicine and 27 papers in Cellular and Molecular Neuroscience. Recurrent topics in Isabelle Marty's work include Ion channel regulation and function (46 papers), Muscle Physiology and Disorders (27 papers) and Cardiomyopathy and Myosin Studies (20 papers). Isabelle Marty is often cited by papers focused on Ion channel regulation and function (46 papers), Muscle Physiology and Disorders (27 papers) and Cardiomyopathy and Myosin Studies (20 papers). Isabelle Marty collaborates with scholars based in France, United States and Italy. Isabelle Marty's co-authors include Michel Villaz, Yves Meyer, Joël Lunardi, Julien Fauré, Michel Ronjat, Julie Brocard, Jane‐Lise Samuel, Christophe Heymes, Yvette Chartier and Nicole Monnier and has published in prestigious journals such as Proceedings of the National Academy of Sciences, The Lancet and Nucleic Acids Research.

In The Last Decade

Isabelle Marty

99 papers receiving 3.5k citations

Peers

Countries citing papers authored by Isabelle Marty

Since Specialization

Citations

This map shows the geographic impact of Isabelle Marty's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Isabelle Marty with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Isabelle Marty more than expected).

Fields of papers citing papers by Isabelle Marty

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Isabelle Marty. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Isabelle Marty. The network helps show where Isabelle Marty may publish in the future.

Co-authorship network of co-authors of Isabelle Marty

This figure shows the co-authorship network connecting the top 25 collaborators of Isabelle Marty. A scholar is included among the top collaborators of Isabelle Marty based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Isabelle Marty. Isabelle Marty is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

#	Title	Journal	Authors	Indexed citations
1	Limited pre-clinical relevance of the heterozygous RYR1-I4895T/+ mouse model due to its mild phenotype	Journal of Neuromuscular Diseases	Caroline Benstaali, Julie Brocard et al.	0
2	Functional benefit of CRISPR-Cas9-induced allele deletion for RYR1 dominant mutation	Molecular Therapy — Nucleic Acids	Julie Brocard, Kamel Mamchaoui et al.	1
3	Caveolae and Bin1 form ring-shaped platforms for T-tubule initiation	eLife	Jeanne Lainé, Marion Benoist et al.	13
4	Gene therapies for RyR1-related myopathies	Current Opinion in Pharmacology	Isabelle Marty, Julien Fauré et al.	2
5	Huntingtin regulates calcium fluxes in skeletal muscle	The Journal of General Physiology	Mathilde Chivet, Anne‐Sophie Nicot et al.	7
6	Therapies for RYR1-Related Myopathies: Where We Stand and the Perspectives	Current Pharmaceutical Design	John Rendu, Isabelle Marty et al.	8
7	SPEG binds with desmin and its deficiency causes defects in triad and focal adhesion proteins	Human Molecular Genetics	Shiyu Luo, Qifei Li et al.	7
8	In vivo RyR1 reduction in muscle triggers a core-like myopathy	Acta Neuropathologica Communications	Laurent Pelletier, Anne Petiot et al.	15
9	Interplay between Triadin and Calsequestrin in the Pathogenesis of CPVT in the Mouse	Molecular Therapy	Marine Cacheux, Jérémy Fauconnier et al.	20
10	Deletion of the microtubule-associated protein 6 (MAP6) results in skeletal muscle dysfunction	Skeletal Muscle	Benoı̂t Giannesini, Julie Brocard et al.	19
11	217th ENMC International Workshop: RYR1-related myopathies, Naarden, The Netherlands, 29–31 January 2016	Neuromuscular Disorders	Heinz Jungbluth, James J. Dowling et al.	32
12	Triadin and CLIMP-63 form a link between triads and microtubules in muscle cells	Journal of Cell Science	Mathieu Baudet, Yohann Couté et al.	25
13	An Integrated Diagnosis Strategy for Congenital Myopathies	PLoS ONE	Johann Böhm, Nasim Vasli et al.	43
14	Recessive RYR1 mutations cause unusual congenital myopathy with prominent nuclear internalization and large areas of myofibrillar disorganization	Neuropathology and Applied Neurobiology	Jorge A. Bevilacqua, Nicole Monnier et al.	75
15	Triadin Function In Sarcoplasmic Reticulum Structure?	Biophysical Journal	Julien Fauré, Anne Fourest‐Lieuvin et al.	1
16	Triadin Deletion Induces Impaired Skeletal Muscle Function	Journal of Biological Chemistry	Sarah Oddoux, Julie Brocard et al.	71
17	C.P.1.05 Congenital fibre type disproportion associated with de novo mutations in TPM3 and ACTA1 genes	Neuromuscular Disorders	Nicole Monnier, Annick Labarre‐Vila et al.	1
18	C.P.1.13 A homozygous null mutation in TPM2 gene causes autosomal recessive nemaline myopathy associated with multiple pterygia	Neuromuscular Disorders	Pierre‐Simon Jouk, Annick Labarre‐Vila et al.	2
19	Triadin Binding to the C-Terminal Luminal Loop of the Ryanodine Receptor is Important for Skeletal Muscle Excitation–Contraction Coupling	The Journal of General Physiology	Sanjeewa A. Goonasekera, Nicole A. Beard et al.	65
20	Growth-related gene expression in Nicotiana tabacum mesophyll protoplasts	HAL (Le Centre pour la Communication Scientifique Directe)	Isabelle Marty, Christophe Brugidou et al.	1

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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