Chi Wai Eric So

4.2k total citations
65 papers, 3.0k citations indexed

About

Chi Wai Eric So is a scholar working on Molecular Biology, Hematology and Oncology. According to data from OpenAlex, Chi Wai Eric So has authored 65 papers receiving a total of 3.0k indexed citations (citations by other indexed papers that have themselves been cited), including 55 papers in Molecular Biology, 38 papers in Hematology and 9 papers in Oncology. Recurrent topics in Chi Wai Eric So's work include Acute Myeloid Leukemia Research (37 papers), Protein Degradation and Inhibitors (20 papers) and Retinoids in leukemia and cellular processes (12 papers). Chi Wai Eric So is often cited by papers focused on Acute Myeloid Leukemia Research (37 papers), Protein Degradation and Inhibitors (20 papers) and Retinoids in leukemia and cellular processes (12 papers). Chi Wai Eric So collaborates with scholars based in United Kingdom, United States and Hong Kong. Chi Wai Eric So's co-authors include Michael L. Cleary, Bernd B. Zeisig, Li Chong Chan, Ngai Cheung, Maria Teresa Esposito, Holger Karsunky, Irving L. Weissman, Tsz Kan Fung, Alexander Thompson and Jenny Yeung and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Nature Medicine and The EMBO Journal.

In The Last Decade

Chi Wai Eric So

64 papers receiving 2.9k citations

Peers

Chi Wai Eric So
Paul M. Ayton United States
Silvia Grisendi United States
Kristin J. Hope Canada
Hanna S. Radomska United States
Jana Krošl Canada
Kalindi Parmar United States
Jasper de Boer United Kingdom
Gary W. Reuther United States
Yutaka Okuno Japan
Marc A. Kerenyi United States
Paul M. Ayton United States
Chi Wai Eric So
Citations per year, relative to Chi Wai Eric So Chi Wai Eric So (= 1×) peers Paul M. Ayton

Countries citing papers authored by Chi Wai Eric So

Since Specialization
Citations

This map shows the geographic impact of Chi Wai Eric So's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Chi Wai Eric So with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Chi Wai Eric So more than expected).

Fields of papers citing papers by Chi Wai Eric So

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Chi Wai Eric So. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Chi Wai Eric So. The network helps show where Chi Wai Eric So may publish in the future.

Co-authorship network of co-authors of Chi Wai Eric So

This figure shows the co-authorship network connecting the top 25 collaborators of Chi Wai Eric So. A scholar is included among the top collaborators of Chi Wai Eric So based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Chi Wai Eric So. Chi Wai Eric So is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Daniels, Ruth, Wim Van der Elst, Chi Wai Eric So, et al.. (2024). Fit for purpose testing and independent GMP validation of the monocyte activation test. Current Research in Toxicology. 8. 100206–100206. 1 indexed citations
2.
So, Chi Wai Eric, L. Di Silvio, Ruby Quartey‐Papafio, et al.. (2023). High Throughput Fluorescence-Based In Vitro Experimental Platform for the Identification of Effective Therapies to Overcome Tumour Microenvironment-Mediated Drug Resistance in AML. Cancers. 15(7). 1988–1988. 4 indexed citations
3.
Luo, Huacheng, Ganqian Zhu, Melanie A. Eshelman, et al.. (2022). HOTTIP-dependent R-loop formation regulates CTCF boundary activity and TAD integrity in leukemia. Molecular Cell. 82(4). 833–851.e11. 76 indexed citations
4.
So, Chi Wai Eric, et al.. (2020). Novel therapeutic strategies for MLL-rearranged leukemias. Biochimica et Biophysica Acta (BBA) - Gene Regulatory Mechanisms. 1863(9). 194584–194584. 14 indexed citations
5.
He, Bai‐Liang, Ning Yang, Cheuk Him Man, et al.. (2020). Follistatin is a novel therapeutic target and biomarker in FLT 3/ ITD acute myeloid leukemia. EMBO Molecular Medicine. 12(4). e10895–e10895. 14 indexed citations
6.
Zhao, Lu & Chi Wai Eric So. (2016). PARP-inhibitor-induced synthetic lethality for acute myeloid leukemia treatment. Experimental Hematology. 44(10). 902–907. 26 indexed citations
7.
Esposito, Maria Teresa & Chi Wai Eric So. (2014). DNA damage accumulation and repair defects in acute myeloid leukemia: implications for pathogenesis, disease progression, and chemotherapy resistance. Chromosoma. 123(6). 545–561. 59 indexed citations
8.
Arteaga, Marı́a Francisca, Jan‐Henrik Mikesch, Jihui Qiu, et al.. (2013). The Histone Demethylase PHF8 Governs Retinoic Acid Response in Acute Promyelocytic Leukemia. Cancer Cell. 23(3). 376–389. 69 indexed citations
9.
Jiang, Xueqing, Lin Sun, Jihui Qiu, et al.. (2013). A Novel Application of Furazolidone: Anti-Leukemic Activity in Acute Myeloid Leukemia. PLoS ONE. 8(8). e72335–e72335. 26 indexed citations
10.
Zeisig, Bernd B. & Chi Wai Eric So. (2013). Linking MLL Leukemia with Integrin Signaling. Cancer Cell. 24(1). 5–7. 1 indexed citations
11.
Fung, Tsz Kan, Arnaud Gandillet, & Chi Wai Eric So. (2012). Selective treatment of mixed-lineage leukemia leukemic stem cells through targeting glycogen synthase kinase 3 and the canonical Wnt/β-catenin pathway. Current Opinion in Hematology. 19(4). 280–286. 22 indexed citations
12.
Smith, Lan‐Lan, Jenny Yeung, Bernd B. Zeisig, et al.. (2011). Functional Crosstalk between Bmi1 and MLL/Hoxa9 Axis in Establishment of Normal Hematopoietic and Leukemic Stem Cells. Cell stem cell. 8(6). 649–662. 89 indexed citations
13.
Alvares, Caroline, Tino Schenk, Toon Min, et al.. (2011). Tyrosine kinase inhibitor insensitivity of non‐cycling CD34+ human acute myeloid leukaemia cells with FMS‐like tyrosine kinase 3 mutations. British Journal of Haematology. 154(4). 457–465. 6 indexed citations
14.
Yeung, Jenny, Maria Teresa Esposito, Arnaud Gandillet, et al.. (2010). β-Catenin Mediates the Establishment and Drug Resistance of MLL Leukemic Stem Cells. Cancer Cell. 18(6). 606–618. 218 indexed citations
15.
So, Chi Wai Eric. (2009). An Overview: From Discovery of Candidate Mutations to Disease Modeling and Transformation Mechanisms of Acute Leukemia. Methods in molecular biology. 538. 1–5. 4 indexed citations
16.
Zeisig, Bernd B., Colin Kwok, Arthur Zelent, et al.. (2007). Recruitment of RXR by Homotetrameric RARα Fusion Proteins Is Essential for Transformation. Cancer Cell. 12(1). 36–51. 76 indexed citations
17.
Cheung, Ngai, Li Chong Chan, Alexander Thompson, Michael L. Cleary, & Chi Wai Eric So. (2007). Protein arginine-methyltransferase-dependent oncogenesis. Nature Cell Biology. 9(10). 1208–1215. 235 indexed citations
18.
19.
Kwok, Colin, Bernd B. Zeisig, Shuo Dong, & Chi Wai Eric So. (2006). Forced homo-oligomerization of RARα leads to transformation of primary hematopoietic cells. Cancer Cell. 9(2). 95–108. 55 indexed citations
20.
So, Chi Wai Eric, Holger Karsunky, Emmanuelle Passegué, et al.. (2003). MLL-GAS7 transforms multipotent hematopoietic progenitors and induces mixed lineage leukemias in mice. Cancer Cell. 3(2). 161–171. 153 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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