Cheryll Sanchez-Irizarry

3.8k total citations · 1 hit paper
8 papers, 2.8k citations indexed

About

Cheryll Sanchez-Irizarry is a scholar working on Molecular Biology, Neurology and Genetics. According to data from OpenAlex, Cheryll Sanchez-Irizarry has authored 8 papers receiving a total of 2.8k indexed citations (citations by other indexed papers that have themselves been cited), including 6 papers in Molecular Biology, 3 papers in Neurology and 2 papers in Genetics. Recurrent topics in Cheryll Sanchez-Irizarry's work include Cerebrovascular and genetic disorders (3 papers), Developmental Biology and Gene Regulation (3 papers) and Acute Myeloid Leukemia Research (2 papers). Cheryll Sanchez-Irizarry is often cited by papers focused on Cerebrovascular and genetic disorders (3 papers), Developmental Biology and Gene Regulation (3 papers) and Acute Myeloid Leukemia Research (2 papers). Cheryll Sanchez-Irizarry collaborates with scholars based in United States and Netherlands. Cheryll Sanchez-Irizarry's co-authors include Jon C. Aster, Stephen C. Blacklow, Andrew P. Weng, Adolfo A. Ferrando, John P. Morris, A. Thomas Look, Lewis B. Silverman, Woojoong Lee, Gavin Histen and Didem Vardar‐Ulu and has published in prestigious journals such as Science, Blood and PLoS ONE.

In The Last Decade

Cheryll Sanchez-Irizarry

8 papers receiving 2.7k citations

Hit Papers

Activating Mutations of NOTCH1 in Human T Cell Acute Lymp... 2004 2026 2011 2018 2004 500 1000 1.5k 2.0k

Peers

Cheryll Sanchez-Irizarry
Woojoong Lee South Korea
Blanca Scheijen Netherlands
Lee N. Lawton United States
Bart Lutterbach United States
Thomas Trimarchi United States
Ellen van Drunen Netherlands
Susan Winandy United States
Fredrick G. Karnell United States
Jami McLaughlin United States
Woojoong Lee South Korea
Cheryll Sanchez-Irizarry
Citations per year, relative to Cheryll Sanchez-Irizarry Cheryll Sanchez-Irizarry (= 1×) peers Woojoong Lee

Countries citing papers authored by Cheryll Sanchez-Irizarry

Since Specialization
Citations

This map shows the geographic impact of Cheryll Sanchez-Irizarry's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Cheryll Sanchez-Irizarry with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Cheryll Sanchez-Irizarry more than expected).

Fields of papers citing papers by Cheryll Sanchez-Irizarry

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Cheryll Sanchez-Irizarry. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Cheryll Sanchez-Irizarry. The network helps show where Cheryll Sanchez-Irizarry may publish in the future.

Co-authorship network of co-authors of Cheryll Sanchez-Irizarry

This figure shows the co-authorship network connecting the top 25 collaborators of Cheryll Sanchez-Irizarry. A scholar is included among the top collaborators of Cheryll Sanchez-Irizarry based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Cheryll Sanchez-Irizarry. Cheryll Sanchez-Irizarry is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

8 of 8 papers shown
1.
Hagenbeek, Thijs J., Xiumin Wu, Lisa Choy, et al.. (2014). Murine Pten−/− T-ALL requires non-redundant PI3K/mTOR and DLL4/Notch1 signals for maintenance and γc/TCR signals for thymic exit. Cancer Letters. 346(2). 237–248. 10 indexed citations
2.
Gordon, Wendy R., Didem Vardar‐Ulu, Todd Ashworth, et al.. (2009). Effects of S1 Cleavage on the Structure, Surface Export, and Signaling Activity of Human Notch1 and Notch2. PLoS ONE. 4(8). e6613–e6613. 82 indexed citations
3.
Gordon, Wendy R., Didem Vardar‐Ulu, Gavin Histen, et al.. (2007). Structural basis for autoinhibition of Notch. Nature Structural & Molecular Biology. 14(4). 295–300. 272 indexed citations
4.
Malecki, Michael, Cheryll Sanchez-Irizarry, Jennifer Mitchell, et al.. (2006). Leukemia-Associated Mutations within the NOTCH1 Heterodimerization Domain Fall into at Least Two Distinct Mechanistic Classes. Molecular and Cellular Biology. 26(12). 4642–4651. 192 indexed citations
5.
Sanchez-Irizarry, Cheryll, Michael Malecki, Woojoong Lee, et al.. (2005). Functional Analysis of Leukemia-Associated Mutations Involving the Heterodimerization Domain of NOTCH1.. Blood. 106(11). 843–843. 1 indexed citations
6.
Weng, Andrew P., Adolfo A. Ferrando, Woojoong Lee, et al.. (2004). Activating Mutations of NOTCH1 in Human T Cell Acute Lymphoblastic Leukemia. Science. 306(5694). 269–271. 2013 indexed citations breakdown →
7.
Sanchez-Irizarry, Cheryll, Andrea C. Carpenter, Andrew P. Weng, et al.. (2004). Notch Subunit Heterodimerization and Prevention of Ligand-Independent Proteolytic Activation Depend, Respectively, on a Novel Domain and the LNR Repeats. Molecular and Cellular Biology. 24(21). 9265–9273. 163 indexed citations
8.
Vardar‐Ulu, Didem, Christopher L. North, Cheryll Sanchez-Irizarry, Jon C. Aster, & Stephen C. Blacklow. (2003). Nuclear Magnetic Resonance Structure of a Prototype Lin12-Notch Repeat Module from Human Notch1. Biochemistry. 42(23). 7061–7067. 36 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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