Bart Lutterbach

4.8k total citations
29 papers, 3.1k citations indexed

About

Bart Lutterbach is a scholar working on Molecular Biology, Hematology and Oncology. According to data from OpenAlex, Bart Lutterbach has authored 29 papers receiving a total of 3.1k indexed citations (citations by other indexed papers that have themselves been cited), including 26 papers in Molecular Biology, 12 papers in Hematology and 6 papers in Oncology. Recurrent topics in Bart Lutterbach's work include Acute Myeloid Leukemia Research (11 papers), Protein Kinase Regulation and GTPase Signaling (5 papers) and Retinoids in leukemia and cellular processes (5 papers). Bart Lutterbach is often cited by papers focused on Acute Myeloid Leukemia Research (11 papers), Protein Kinase Regulation and GTPase Signaling (5 papers) and Retinoids in leukemia and cellular processes (5 papers). Bart Lutterbach collaborates with scholars based in United States, Japan and Malaysia. Bart Lutterbach's co-authors include Scott W. Hiebert, Stephan Hann, Bryan Linggi, Lenora J. Davis, Jennifer J. Westendorf, Joseph M. Amann, Edward Seto, Harold Hatch, James R. Downing and John Nip and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Journal of Biological Chemistry and PLoS ONE.

In The Last Decade

Bart Lutterbach

29 papers receiving 3.1k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Bart Lutterbach United States 23 2.5k 1.0k 653 313 263 29 3.1k
Mikhail L. Gishizky United States 20 1.7k 0.7× 698 0.7× 717 1.1× 384 1.2× 251 1.0× 30 3.0k
Doris Steinemann Germany 31 1.6k 0.7× 445 0.4× 551 0.8× 174 0.6× 505 1.9× 127 2.8k
Cristina E. Tognon United States 26 2.0k 0.8× 691 0.7× 980 1.5× 509 1.6× 705 2.7× 81 3.6k
Nathalie A. Lokker United States 20 1.1k 0.4× 476 0.5× 468 0.7× 164 0.5× 247 0.9× 29 2.1k
Kolja Eppert Canada 16 1.8k 0.7× 985 1.0× 841 1.3× 235 0.8× 491 1.9× 27 3.1k
Richard C. Frank United States 21 1.1k 0.4× 504 0.5× 986 1.5× 495 1.6× 204 0.8× 50 2.4k
Maike Jaworski Germany 14 1.1k 0.5× 1.1k 1.0× 496 0.8× 101 0.3× 424 1.6× 18 2.6k
Kalindi Parmar United States 28 2.1k 0.9× 706 0.7× 862 1.3× 170 0.5× 586 2.2× 60 3.0k
Maria Luisa Sulis United States 19 2.2k 0.9× 764 0.7× 638 1.0× 132 0.4× 367 1.4× 52 3.2k
Lisa A. Moreau United States 28 3.3k 1.3× 448 0.4× 1.3k 2.0× 171 0.5× 782 3.0× 46 4.2k

Countries citing papers authored by Bart Lutterbach

Since Specialization
Citations

This map shows the geographic impact of Bart Lutterbach's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Bart Lutterbach with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Bart Lutterbach more than expected).

Fields of papers citing papers by Bart Lutterbach

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Bart Lutterbach. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Bart Lutterbach. The network helps show where Bart Lutterbach may publish in the future.

Co-authorship network of co-authors of Bart Lutterbach

This figure shows the co-authorship network connecting the top 25 collaborators of Bart Lutterbach. A scholar is included among the top collaborators of Bart Lutterbach based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Bart Lutterbach. Bart Lutterbach is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Ware, Christopher, et al.. (2014). FGFR2 Is Amplified in the NCI-H716 Colorectal Cancer Cell Line and Is Required for Growth and Survival. PLoS ONE. 9(6). e98515–e98515. 33 indexed citations
2.
Benedettini, Elisa, Lynette M. Sholl, Michael Peyton, et al.. (2010). Met Activation in Non-Small Cell Lung Cancer Is Associated with de Novo Resistance to EGFR Inhibitors and the Development of Brain Metastasis. American Journal Of Pathology. 177(1). 415–423. 139 indexed citations
3.
Lu, Wei, Lenora J. Davis, Gaozhen Hang, et al.. (2009). Abstract #3714: In vitro and in vivo antitumor activities of MK-2206, a new allosteric Akt inhibitor. Cancer Research. 69. 3714–3714. 11 indexed citations
4.
Kunii, Kaiko, Lenora J. Davis, Julie Gorenstein, et al.. (2008). FGFR2 -Amplified Gastric Cancer Cell Lines Require FGFR2 and Erbb3 Signaling for Growth and Survival. Cancer Research. 68(7). 2340–2348. 230 indexed citations
5.
Lutterbach, Bart, et al.. (2008). High-Throughput Analysis of HGF-Stimulated Cell Scattering. SLAS DISCOVERY. 13(9). 847–854. 11 indexed citations
6.
Lutterbach, Bart, Qinwen Zeng, Lenora J. Davis, et al.. (2007). Lung Cancer Cell Lines Harboring MET Gene Amplification Are Dependent on Met for Growth and Survival. Cancer Research. 67(5). 2081–2088. 260 indexed citations
7.
Moreno–Miralles, Isabel, Bart Lutterbach, K. Scott Luce, et al.. (2006). RUNX1 associates with histone deacetylases and SUV39H1 to repress transcription. Oncogene. 25(42). 5777–5786. 60 indexed citations
8.
Lutterbach, Bart, Scott W. Hiebert, Jeffrey A. Nickerson, et al.. (2002). Multiple subnuclear targeting signals of the leukemia-related AML1/ETO and ETO repressor proteins. Proceedings of the National Academy of Sciences. 99(24). 15434–15439. 51 indexed citations
9.
Amann, Joseph M., John Nip, David K. Strom, et al.. (2001). ETO, a Target of t(8;21) in Acute Leukemia, Makes Distinct Contacts with Multiple Histone Deacetylases and Binds mSin3A through Its Oligomerization Domain. Molecular and Cellular Biology. 21(19). 6470–6483. 270 indexed citations
10.
Hiebert, Scott W., Bart Lutterbach, & Joseph M. Amann. (2001). Role of co-repressors in transcriptional repression mediated by the t(8;21), t(16;21), t(12;21), and inv(16) fusion proteins. Current Opinion in Hematology. 8(4). 197–200. 44 indexed citations
11.
Hiebert, Scott W., Bart Lutterbach, Lilin Wang, et al.. (2001). Mechanisms of transcriptional repression by the t(8;21)-, t(12;21)-, and inv(16)-encoded fusion proteins. Cancer Chemotherapy and Pharmacology. 48(0). S31–S34. 23 indexed citations
12.
Lutterbach, Bart & Scott W. Hiebert. (2000). Role of the transcription factor AML-1 in acute leukemia and hematopoietic differentiation. Gene. 245(2). 223–235. 170 indexed citations
13.
Gregory, Mark A., Qiurong Xiao, Gail A. Cornwall, Bart Lutterbach, & Stephan Hann. (2000). B-Myc is preferentially expressed in hormonally-controlled tissues and inhibits cellular proliferation. Oncogene. 19(42). 4886–4895. 16 indexed citations
14.
Strom, David K., John Nip, Jennifer J. Westendorf, et al.. (2000). Expression of the AML-1 Oncogene Shortens the G1Phase of the Cell Cycle. Journal of Biological Chemistry. 275(5). 3438–3445. 91 indexed citations
15.
Lutterbach, Bart & Stephan Hann. (1999). c-Myc transactivation domain-associated kinases: Questionable role for map kinases in c-Myc phosphorylation. Journal of Cellular Biochemistry. 72(4). 483–491. 18 indexed citations
16.
Lutterbach, Bart, Daxi Sun, John D. Schuetz, & Scott W. Hiebert. (1998). The MYND Motif Is Required for Repression of Basal Transcription from the Multidrug Resistance 1 Promoter by the t(8;21) Fusion Protein. Molecular and Cellular Biology. 18(6). 3604–3611. 156 indexed citations
17.
Lutterbach, Bart & Stephan Hann. (1997). Overexpression of c-Myc and cell immortalization alters c-Myc phosphorylation. Oncogene. 14(8). 967–975. 29 indexed citations
18.
Hoang, Arthur T., Bart Lutterbach, Brian C. Lewis, et al.. (1995). A Link between Increased Transforming Activity of Lymphoma-Derived MYC Mutant Alleles, Their Defective Regulation by p107, and Altered Phosphorylation of the c-Myc Transactivation Domain. Molecular and Cellular Biology. 15(8). 4031–4042. 123 indexed citations
19.
Lutterbach, Bart & Stephan Hann. (1994). Hierarchical Phosphorylation at N-Terminal Transformation-Sensitive Sites in c-Myc Protein Is Regulated by Mitogens and in Mitosis. Molecular and Cellular Biology. 14(8). 5510–5522. 77 indexed citations
20.
Coulson, Alan, et al.. (1991). YACs and the C. elegans genome. BioEssays. 13(8). 413–417. 97 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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