Charles R. Dearolf

3.2k total citations
28 papers, 2.7k citations indexed

About

Charles R. Dearolf is a scholar working on Molecular Biology, Immunology and Oncology. According to data from OpenAlex, Charles R. Dearolf has authored 28 papers receiving a total of 2.7k indexed citations (citations by other indexed papers that have themselves been cited), including 20 papers in Molecular Biology, 9 papers in Immunology and 7 papers in Oncology. Recurrent topics in Charles R. Dearolf's work include Developmental Biology and Gene Regulation (9 papers), Invertebrate Immune Response Mechanisms (8 papers) and Cytokine Signaling Pathways and Interactions (7 papers). Charles R. Dearolf is often cited by papers focused on Developmental Biology and Gene Regulation (9 papers), Invertebrate Immune Response Mechanisms (8 papers) and Cytokine Signaling Pathways and Interactions (7 papers). Charles R. Dearolf collaborates with scholars based in United States, Switzerland and Germany. Charles R. Dearolf's co-authors include William P. Hanratty, Allen Shearn, Hong Luo, Hongbo R. Luo, Riqiang Yan, Joanne Topol, Evelyn Hersperger, Stephen Small, James Darnell and Claude Desplan and has published in prestigious journals such as Nature, Cell and Proceedings of the National Academy of Sciences.

In The Last Decade

Charles R. Dearolf

28 papers receiving 2.6k citations

Peers

Charles R. Dearolf

IMMUN

ONCOL

CMN

Martin P. Zeidler United Kingdom

Allison J. Bardin France

J. Peter Gergen United States

Christof Fellmann United States

D A Withers United States

Hugh W. Brock Canada

Anthony Boureux France

George Pyrowolakis Germany

Mark Stapleton United States

Paul Badenhorst United Kingdom

Martin P. Zeidler United Kingdom

Charles R. Dearolf

1.7k ×1.1

1.2k ×1.0

IMMUN

756 ×1.1

ONCOL

565 ×1.4

CMN

336 ×1.0

Citations per year, relative to Charles R. Dearolf Charles R. Dearolf (= 1×) peers Martin P. Zeidler

Countries citing papers authored by Charles R. Dearolf

Since Specialization

Citations

This map shows the geographic impact of Charles R. Dearolf's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Charles R. Dearolf with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Charles R. Dearolf more than expected).

Fields of papers citing papers by Charles R. Dearolf

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Charles R. Dearolf. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Charles R. Dearolf. The network helps show where Charles R. Dearolf may publish in the future.

Co-authorship network of co-authors of Charles R. Dearolf

This figure shows the co-authorship network connecting the top 25 collaborators of Charles R. Dearolf. A scholar is included among the top collaborators of Charles R. Dearolf based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Charles R. Dearolf. Charles R. Dearolf is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Dearolf, Charles R., et al.. (2024). The National Institutes of Health Lasker Clinical Research Scholars Program: A Decade of Launching Clinician-Scientist Careers. Academic Medicine. 100(1). 57–62. 1 indexed citations

Nairz, Knud, Peder Zipperlen, Charles R. Dearolf, Konrad Basler, & Ernst Hafen. (2004). A reverse genetic screen in Drosophila using a deletion-inducing mutagen. Genome biology. 5(10). R83–R83. 7 indexed citations

Bettencourt, Raúl, Hesarghatta Shyamasunder Asha, Charles R. Dearolf, & Y. Tony Ip. (2004). Hemolymph‐dependent and ‐independent responses in Drosophila immune tissue. Journal of Cellular Biochemistry. 92(4). 849–863. 40 indexed citations

Dobens, Leonard L., et al.. (2002). The Drosophila STAT Protein, Stat92E, Regulates Follicle Cell Differentiation during Oogenesis. Developmental Biology. 243(1). 166–175. 77 indexed citations

Luo, Hong, Paul Rose, Thomas G. Roberts, & Charles R. Dearolf. (2002). The Hopscotch Jak kinase requires the Raf pathway to promote blood cell activation and differentiation in Drosophila. Molecular Genetics and Genomics. 267(1). 57–63. 44 indexed citations

Luo, Hong & Charles R. Dearolf. (2001). The JAK/STAT pathway and Drosophila development. BioEssays. 23(12). 1138–1147. 56 indexed citations

Myrick, Kyl V. & Charles R. Dearolf. (2000). Hyperactivation of the Drosophila Hop Jak kinase causes the preferential overexpression of eIF1A transcripts in larval blood cells. Gene. 244(1-2). 119–125. 14 indexed citations

Dearolf, Charles R.. (1999). JAKs and STATs in invertebrate model organisms. Cellular and Molecular Life Sciences. 55(12). 1578–1584. 58 indexed citations

Luo, Hong, et al.. (1999). The Drosophila Jak Kinase Hopscotch Is Required for Multiple Developmental Processes in the Eye. Developmental Biology. 213(2). 432–441. 35 indexed citations

10.

Dearolf, Charles R.. (1998). Fruit fly “leukemia”. Biochimica et Biophysica Acta (BBA) - Reviews on Cancer. 1377(1). M13–M23. 54 indexed citations

11.

Yasothornsrikul, Sukkid, et al.. (1997). viking: identification and characterization of a second type IV collagen in Drosophila. Gene. 198(1-2). 17–25. 99 indexed citations

12.

Luo, Hong, Paul Rose, Dwayne L. Barber, et al.. (1997). Mutation in the Jak Kinase JH2 Domain Hyperactivates Drosophila and Mammalian Jak-Stat Pathways. Molecular and Cellular Biology. 17(3). 1562–1571. 207 indexed citations

13.

Yan, Riqiang, Stephen Small, Claude Desplan, Charles R. Dearolf, & James Darnell. (1996). Identification of a Stat Gene That Functions in Drosophila Development. Cell. 84(3). 421–430. 325 indexed citations

14.

Luo, Hongbo R., William P. Hanratty, & Charles R. Dearolf. (1995). An amino acid substitution in the Drosophila hopTum-l Jak kinase causes leukemia-like hematopoietic defects.. The EMBO Journal. 14(7). 1412–1420. 256 indexed citations

15.

Zinyk, Dawn, Bethany McGonnigal, & Charles R. Dearolf. (1993). Drosophila awdK–pn, a homologue of the metastasis suppressor gene nm23, suppresses the Tum–I haematopoietic oncogene. Nature Genetics. 4(2). 195–201. 31 indexed citations

16.

Hanratty, William P. & Charles R. Dearolf. (1993). The Drosophila Tumorous lethal hematopoietic oncogene is a dominant mutation in the hopscotch locus. Molecular and General Genetics MGG. 238-238(1-2). 33–37. 130 indexed citations

17.

Dearolf, Charles R., Joanne Topol, & Carl S. Parker. (1990). Transcriptional regulation of the Drosophila segmentation gene fushi tarazu (ftz). BioEssays. 12(3). 109–113. 8 indexed citations

18.

Dearolf, Charles R., Joanne Topol, & C S Parker. (1989). Transcriptional control of Drosophila fushi tarazu zebra stripe expression.. Genes & Development. 3(3). 384–398. 81 indexed citations

19.

Dearolf, Charles R., Evelyn Hersperger, & Allen Shearn. (1988). Developmental consequences of awd, a cell-autonomous lethal mutation of Drosophila induced by hybrid dysgenesis. Developmental Biology. 129(1). 159–168. 148 indexed citations

20.

Dearolf, Charles R., et al.. (1988). Molecular consequences of awd, a cell-autonomous lethal mutation of Drosophila induced by hybrid dysgenesis. Developmental Biology. 129(1). 169–178. 91 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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