Charles Massie

24.4k total citations · 1 hit paper
42 papers, 4.0k citations indexed

About

Charles Massie is a scholar working on Molecular Biology, Pulmonary and Respiratory Medicine and Cancer Research. According to data from OpenAlex, Charles Massie has authored 42 papers receiving a total of 4.0k indexed citations (citations by other indexed papers that have themselves been cited), including 25 papers in Molecular Biology, 19 papers in Pulmonary and Respiratory Medicine and 14 papers in Cancer Research. Recurrent topics in Charles Massie's work include Prostate Cancer Treatment and Research (14 papers), Cancer Genomics and Diagnostics (8 papers) and Epigenetics and DNA Methylation (7 papers). Charles Massie is often cited by papers focused on Prostate Cancer Treatment and Research (14 papers), Cancer Genomics and Diagnostics (8 papers) and Epigenetics and DNA Methylation (7 papers). Charles Massie collaborates with scholars based in United Kingdom, United States and Norway. Charles Massie's co-authors include Nitzan Rosenfeld, Florent Moulière, Carlos Caldas, James D. Brenton, Javier García-Corbacho, Jonathan C. M. Wan, Richard D. Baird, Simon Pacey, Ian G. Mills and David E. Neal and has published in prestigious journals such as Proceedings of the National Academy of Sciences, The Lancet and Nucleic Acids Research.

In The Last Decade

Charles Massie

39 papers receiving 4.0k citations

Hit Papers

Liquid biopsies come of age: towards implementation of ci... 2017 2026 2020 2023 2017 500 1000 1.5k

Peers

Charles Massie
Richard D. Baird United Kingdom
Luca Roz Italy
Mark R. Lackner United States
Robert J. Lonigro United States
Leander Van Neste United States
Gaia Schiavon United Kingdom
Richard D. Baird United Kingdom
Charles Massie
Citations per year, relative to Charles Massie Charles Massie (= 1×) peers Richard D. Baird

Countries citing papers authored by Charles Massie

Since Specialization
Citations

This map shows the geographic impact of Charles Massie's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Charles Massie with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Charles Massie more than expected).

Fields of papers citing papers by Charles Massie

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Charles Massie. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Charles Massie. The network helps show where Charles Massie may publish in the future.

Co-authorship network of co-authors of Charles Massie

This figure shows the co-authorship network connecting the top 25 collaborators of Charles Massie. A scholar is included among the top collaborators of Charles Massie based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Charles Massie. Charles Massie is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Rossi, Sabrina H., Gahee Park, Christopher G. Smith, et al.. (2025). Evidence of DNA methylation heterogeneity and epipolymorphism in kidney cancer tissue samples. Oncogene. 44(15). 1024–1036.
2.
Illuzzi, Giuditta, Alessandro Galbiati, Anna D. Staniszewska, et al.. (2025). Androgen receptor inhibition extends PARP inhibitor activity in prostate cancer models beyond BRCA mutations and defects in homologous recombination repair. NAR Cancer. 7(4). zcaf035–zcaf035.
4.
Rossi, Sabrina H., Kevin Brennan, Gahee Park, et al.. (2022). Accurate detection of benign and malignant renal tumor subtypes with MethylBoostER: An epigenetic marker–driven learning framework. Science Advances. 8(39). eabn9828–eabn9828. 7 indexed citations
5.
Mair, Richard, Florent Moulière, Christopher G. Smith, et al.. (2018). Measurement of Plasma Cell-Free Mitochondrial Tumor DNA Improves Detection of Glioblastoma in Patient-Derived Orthotopic Xenograft Models. Cancer Research. 79(1). 220–230. 68 indexed citations
6.
Patel, Keval, Kristan E. van der Vos, Christopher G. Smith, et al.. (2017). Association Of Plasma And Urinary Mutant DNA With Clinical Outcomes In Muscle Invasive Bladder Cancer. Scientific Reports. 7(1). 5554–5554. 84 indexed citations
7.
Möck, Andreas, Suzanne Murphy, James Morris, et al.. (2017). CVE: an R package for interactive variant prioritisation in precision oncology. BMC Medical Genomics. 10(1). 37–37. 8 indexed citations
8.
Wan, Jonathan C. M., Charles Massie, Javier García-Corbacho, et al.. (2017). Liquid biopsies come of age: towards implementation of circulating tumour DNA. Nature reviews. Cancer. 17(4). 223–238. 1766 indexed citations breakdown →
9.
Massie, Charles, Inmaculada Spiteri, Helen Ross‐Adams, et al.. (2015). HES5 silencing is an early and recurrent change in prostate tumourigenesis. Endocrine Related Cancer. 22(2). 131–144. 10 indexed citations
10.
Zecchini, Vincent, Basetti Madhu, Roslin Russell, et al.. (2014). Nuclear ARRB 1 induces pseudohypoxia and cellular metabolism reprogramming in prostate cancer. The EMBO Journal. 33(12). 1365–1382. 51 indexed citations
11.
Whitaker, Hayley C., Jasmine Kay, Henrik Grönberg, et al.. (2013). N-acetyl-L-aspartyl-L-glutamate peptidase-like 2 is overexpressed in cancer and promotes a pro-migratory and pro-metastatic phenotype. Oncogene. 33(45). 5274–5287. 30 indexed citations
12.
Wong, Chi Chun, Sancha Martin, Alistair G. Rust, et al.. (2013). Inactivating CUX1 mutations promote tumorigenesis. Nature Genetics. 46(1). 33–38. 96 indexed citations
13.
Sharma, Naomi L., Charles Massie, Antonio Ramos‐Montoya, et al.. (2012). The Androgen Receptor Induces a Distinct Transcriptional Program in Castration-Resistant Prostate Cancer in Man. Cancer Cell. 23(1). 35–47. 284 indexed citations
14.
Massie, Charles & Ian G. Mills. (2011). Mapping Protein–DNA Interactions Using ChIP-Sequencing. Methods in molecular biology. 809. 157–173. 12 indexed citations
15.
Ross-Innes, Caryn S., Rory Stark, Kelly A. Holmes, et al.. (2010). Cooperative interaction between retinoic acid receptor-α and estrogen receptor in breast cancer. Genes & Development. 24(2). 171–182. 216 indexed citations
16.
Vias, Maria, Charles Massie, Philip East, et al.. (2008). Pro-neural transcription factors as cancer markers. BMC Medical Genomics. 1(1). 17–17. 29 indexed citations
17.
Ahmed, Ahmed A., Anthony D. Mills, Ashraf E.K. Ibrahim, et al.. (2007). The Extracellular Matrix Protein TGFBI Induces Microtubule Stabilization and Sensitizes Ovarian Cancers to Paclitaxel. Cancer Cell. 12(6). 514–527. 180 indexed citations
18.
Massie, Charles & Ian G. Mills. (2006). The developing role of receptors and adaptors. Nature reviews. Cancer. 6(5). 403–409. 57 indexed citations
19.
Cameron, David, Charles Massie, G.R. Kerr, & Robert Leonard. (2003). Moderate neutropenia with adjuvant CMF confers improved survival in early breast cancer. British Journal of Cancer. 89(10). 1837–1842. 99 indexed citations
20.
Sellar, Grant C., Genevieve J. Rabiasz, Euan A. Stronach, et al.. (2003). OPCML at 11q25 is epigenetically inactivated and has tumor-suppressor function in epithelial ovarian cancer. Nature Genetics. 34(3). 337–343. 150 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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