Josep V. Forment

3.9k total citations · 1 hit paper
30 papers, 2.1k citations indexed

About

Josep V. Forment is a scholar working on Molecular Biology, Oncology and Cell Biology. According to data from OpenAlex, Josep V. Forment has authored 30 papers receiving a total of 2.1k indexed citations (citations by other indexed papers that have themselves been cited), including 27 papers in Molecular Biology, 18 papers in Oncology and 4 papers in Cell Biology. Recurrent topics in Josep V. Forment's work include DNA Repair Mechanisms (18 papers), PARP inhibition in cancer therapy (12 papers) and CRISPR and Genetic Engineering (8 papers). Josep V. Forment is often cited by papers focused on DNA Repair Mechanisms (18 papers), PARP inhibition in cancer therapy (12 papers) and CRISPR and Genetic Engineering (8 papers). Josep V. Forment collaborates with scholars based in United Kingdom, Spain and Singapore. Josep V. Forment's co-authors include Stephen P. Jackson, Abderrahmane Kaidi, Mark J. O’Connor, Sébastien Britton, Blerta Xhemalçe, Kyle M. Miller, Mehran Nikan, Shankar Balasubramanian, Charles R. Bradshaw and Raphaël Rodriguez and has published in prestigious journals such as Nucleic Acids Research, Journal of Biological Chemistry and Nature Communications.

In The Last Decade

Josep V. Forment

28 papers receiving 2.0k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Small-molecule–induced DNA damage identifies alternative DNA structures in human genes

2012 589 citations Raphaël Rodriguez, Kyle M. Miller et al. Nature Chemical Biology profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Josep V. Forment United Kingdom	18	1.8k	552	225	225	182	30	2.1k
Joshua C. Saldivar United States	17	1.7k 0.9×	520 0.9×	235 1.0×	187 0.8×	414 2.3×	25	2.0k
Kai J. Neelsen Switzerland	15	2.1k 1.2×	760 1.4×	319 1.4×	247 1.1×	195 1.1×	15	2.4k
Hiroyuki Sasanuma Japan	23	1.6k 0.9×	492 0.9×	173 0.8×	259 1.2×	129 0.7×	67	1.7k
Mitsumasa Hashimoto Japan	20	1.5k 0.8×	510 0.9×	291 1.3×	395 1.8×	186 1.0×	60	2.0k
Timothy C. Hallstrom United States	20	1.2k 0.7×	528 1.0×	148 0.7×	166 0.7×	150 0.8×	28	1.6k
Zheng Pan China	18	1.2k 0.7×	456 0.8×	152 0.7×	168 0.7×	193 1.1×	43	1.6k
Joseph Lee United States	17	1.7k 1.0×	305 0.6×	143 0.6×	144 0.6×	142 0.8×	31	1.9k
Irina I. Dianova United Kingdom	23	1.7k 1.0×	653 1.2×	133 0.6×	375 1.7×	172 0.9×	25	1.9k
Vanesa Gottifredi Argentina	27	1.9k 1.1×	1.1k 2.0×	267 1.2×	384 1.7×	138 0.8×	59	2.2k
Lee Jamieson United States	17	1.6k 0.9×	490 0.9×	385 1.7×	244 1.1×	110 0.6×	21	2.2k

Countries citing papers authored by Josep V. Forment

Since Specialization

Citations

This map shows the geographic impact of Josep V. Forment's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Josep V. Forment with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Josep V. Forment more than expected).

Fields of papers citing papers by Josep V. Forment

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Josep V. Forment. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Josep V. Forment. The network helps show where Josep V. Forment may publish in the future.

Co-authorship network of co-authors of Josep V. Forment

This figure shows the co-authorship network connecting the top 25 collaborators of Josep V. Forment. A scholar is included among the top collaborators of Josep V. Forment based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Josep V. Forment. Josep V. Forment is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Illuzzi, Giuditta, Alessandro Galbiati, Anna D. Staniszewska, et al.. (2025). Androgen receptor inhibition extends PARP inhibitor activity in prostate cancer models beyond BRCA mutations and defects in homologous recombination repair. NAR Cancer. 7(4). zcaf035–zcaf035.

Wang, Zijuan, Deepak Behera, Benjamin M. Foster, et al.. (2025). Site-selective photo-crosslinking for the characterisation of transient ubiquitin-like protein-protein interactions. PLoS ONE. 20(1). e0316321–e0316321.

Forment, Josep V., Dimitris Polychronopoulos, Andreas Bender, et al.. (2024). Understanding tumour growth variability in breast cancer xenograft models identifies PARP inhibition resistance biomarkers. npj Precision Oncology. 8(1). 266–266. 2 indexed citations

Sharma, Abhishek, Muhammad Khairul Ramlee, Martin R. Higgs, et al.. (2023). C16orf72/HAPSTR1/TAPR1 functions with BRCA1/Senataxin to modulate replication-associated R-loops and confer resistance to PARP disruption. Nature Communications. 14(1). 5003–5003. 5 indexed citations

Galbiati, Alessandro, Joshua Armenia, Giuditta Illuzzi, et al.. (2022). Drug–gene Interaction Screens Coupled to Tumor Data Analyses Identify the Most Clinically Relevant Cancer Vulnerabilities Driving Sensitivity to PARP Inhibition. Cancer Research Communications. 2(10). 1244–1254. 11 indexed citations

Muñoz‐Martínez, Francisco, Christelle de Renty, Mark J. O’Connor, et al.. (2021). Loss of Cyclin C or CDK8 provides ATR inhibitor resistance by suppressing transcription-associated replication stress. Nucleic Acids Research. 49(15). 8665–8683. 35 indexed citations

Tobalina, Luis, et al.. (2020). A meta-analysis of reversion mutations in BRCA genes identifies signatures of DNA end-joining repair mechanisms driving therapy resistance. Annals of Oncology. 32(1). 103–112. 114 indexed citations

Elling, Ulrich, Michael Woods, Josep V. Forment, et al.. (2019). Derivation and maintenance of mouse haploid embryonic stem cells. Nature Protocols. 14(7). 1991–2014. 15 indexed citations

Forment, Josep V. & Mark J. O’Connor. (2018). Targeting the replication stress response in cancer. Pharmacology & Therapeutics. 188. 155–167. 112 indexed citations

10.

Herzog, Mareike, et al.. (2018). Detection of functional protein domains by unbiased genome-wide forward genetic screening. Scientific Reports. 8(1). 6161–6161. 12 indexed citations

11.

Forment, Josep V., Mareike Herzog, Julia Coates, et al.. (2016). Genome-wide genetic screening with chemically mutagenized haploid embryonic stem cells. Nature Chemical Biology. 13(1). 12–14. 30 indexed citations

12.

Forment, Josep V. & Stephen P. Jackson. (2015). A flow cytometry–based method to simplify the analysis and quantification of protein association to chromatin in mammalian cells. Nature Protocols. 10(9). 1297–1307. 56 indexed citations

13.

Forment, Josep V., et al.. (2014). High-Affinity Glucose Transport in Aspergillus nidulans Is Mediated by the Products of Two Related but Differentially Expressed Genes. PLoS ONE. 9(4). e94662–e94662. 21 indexed citations

14.

Rodriguez, Raphaël, Kyle M. Miller, Josep V. Forment, et al.. (2012). Small-molecule–induced DNA damage identifies alternative DNA structures in human genes. Nature Chemical Biology. 8(3). 301–310. 589 indexed citations breakdown →

15.

Forment, Josep V., Rachael Walker, & Stephen P. Jackson. (2012). A high‐throughput, flow cytometry‐based method to quantify DNA‐end resection in mammalian cells. Cytometry Part A. 81A(10). 922–928. 63 indexed citations

16.

Forment, Josep V., Melanie Blasius, Ilaria Guerini, & Stephen P. Jackson. (2011). Structure-Specific DNA Endonuclease Mus81/Eme1 Generates DNA Damage Caused by Chk1 Inactivation. PLoS ONE. 6(8). e23517–e23517. 90 indexed citations

17.

Blasius, Melanie, et al.. (2011). A phospho-proteomic screen identifies substrates of the checkpoint kinase Chk1. Genome biology. 12(8). R78–R78. 113 indexed citations

18.

Fattouch, Sami, José Vicente Gil, Josep V. Forment, et al.. (2010). Concentration dependent effects of commonly used pesticides on activation versus inhibition of the quince (Cydonia Oblonga) polyphenol oxidase. Food and Chemical Toxicology. 48(3). 957–963. 15 indexed citations

19.

Forment, Josep V., Daniel Ramón, & Andrew MacCabe. (2006). Consecutive gene deletions in Aspergillus nidulans: application of the Cre/loxP system. Current Genetics. 50(3). 217–224. 41 indexed citations

20.

Forment, Josep V., et al.. (2006). Identification of the mstE Gene Encoding a Glucose-inducible, Low Affinity Glucose Transporter in Aspergillus nidulans. Journal of Biological Chemistry. 281(13). 8339–8346. 36 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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