Ce Zhang

3.4k total citations
42 papers, 877 citations indexed

About

Ce Zhang is a scholar working on Cellular and Molecular Neuroscience, Molecular Biology and Physiology. According to data from OpenAlex, Ce Zhang has authored 42 papers receiving a total of 877 indexed citations (citations by other indexed papers that have themselves been cited), including 21 papers in Cellular and Molecular Neuroscience, 18 papers in Molecular Biology and 15 papers in Physiology. Recurrent topics in Ce Zhang's work include Neuroscience and Neuropharmacology Research (15 papers), Neuroinflammation and Neurodegeneration Mechanisms (7 papers) and GDF15 and Related Biomarkers (6 papers). Ce Zhang is often cited by papers focused on Neuroscience and Neuropharmacology Research (15 papers), Neuroinflammation and Neurodegeneration Mechanisms (7 papers) and GDF15 and Related Biomarkers (6 papers). Ce Zhang collaborates with scholars based in China, United States and Hong Kong. Ce Zhang's co-authors include Gerard J. Marek, Jianguo Li, Xiaorong Yang, Jian‐Tian Qiao, Yu Zhang, Rui Wang, Ya Ke, Yanli Li, Cun‐Gen Ma and Liang‐Jun Yan and has published in prestigious journals such as Journal of Biological Chemistry, Brain Research and The FASEB Journal.

In The Last Decade

Ce Zhang

39 papers receiving 868 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Ce Zhang China 18 434 253 236 85 85 42 877
Annika Vaarmann Estonia 14 581 1.3× 345 1.4× 328 1.4× 46 0.5× 141 1.7× 24 1.2k
S Freude Germany 15 327 0.8× 118 0.5× 387 1.6× 37 0.4× 90 1.1× 23 856
Chi Zhao-fu China 20 442 1.0× 274 1.1× 199 0.8× 18 0.2× 98 1.2× 45 951
Takahiro Yaguchi Japan 20 536 1.2× 214 0.8× 154 0.7× 34 0.4× 82 1.0× 50 1.0k
Gaoshang Chai China 17 320 0.7× 162 0.6× 318 1.3× 97 1.1× 148 1.7× 39 898
Fabrício Simão Brazil 19 339 0.8× 191 0.8× 264 1.1× 30 0.4× 241 2.8× 30 1.0k
Lianteng Zhi United States 13 365 0.8× 232 0.9× 216 0.9× 18 0.2× 115 1.4× 20 761
Jin‐Jing Pei Sweden 9 467 1.1× 132 0.5× 423 1.8× 30 0.4× 66 0.8× 12 849
Tapas K. Makar United States 18 452 1.0× 330 1.3× 238 1.0× 29 0.3× 299 3.5× 38 1.3k
Sindhu K. Madathil United States 11 336 0.8× 143 0.6× 123 0.5× 20 0.2× 108 1.3× 15 799

Countries citing papers authored by Ce Zhang

Since Specialization
Citations

This map shows the geographic impact of Ce Zhang's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Ce Zhang with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Ce Zhang more than expected).

Fields of papers citing papers by Ce Zhang

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Ce Zhang. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Ce Zhang. The network helps show where Ce Zhang may publish in the future.

Co-authorship network of co-authors of Ce Zhang

This figure shows the co-authorship network connecting the top 25 collaborators of Ce Zhang. A scholar is included among the top collaborators of Ce Zhang based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Ce Zhang. Ce Zhang is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Song, Lijuan, Jing Hua, Maohua Rong, et al.. (2025). Hydroxysafflower yellow A alleviates the inflammatory response in astrocytes following cerebral ischemia by inhibiting the LCN2/STAT3 feedback loop. Metabolic Brain Disease. 40(4). 161–161. 1 indexed citations
2.
Zhang, Xiaoming, Wanxiang Niu, Ben Xu, et al.. (2025). CircPRKD3-loaded exosomes concomitantly elicit tumor growth inhibition and glioblastoma microenvironment remodeling via inhibiting STAT3 signaling. Neuro-Oncology. 27(8). 1987–2005. 4 indexed citations
3.
Ma, Li, et al.. (2025). Mechanical Fault Diagnosis of High‐Voltage Circuit Breakers Based on IPSO‐VMD and KFCM‐SVM. IEEJ Transactions on Electrical and Electronic Engineering. 20(8). 1195–1202.
4.
Yu, Liming, Luying Zhu, Hua He, et al.. (2020). Cytokines Induce Monkey Neural Stem Cell Differentiation through Notch Signaling. BioMed Research International. 2020(1). 1308526–1308526. 12 indexed citations
5.
Yang, Xiaorong, Xuefei Sun, Jinzi Wu, et al.. (2020). Regulation of the SIRT1 signaling pathway in NMDA-induced Excitotoxicity. Toxicology Letters. 322. 66–76. 15 indexed citations
6.
Li, Jianguo, et al.. (2019). Transient Forebrain Ischemia Induces Differential Bdnf Transcript Expression and Histone Acetylation Patterns in the Rat Hippocampus. Journal of Molecular Neuroscience. 70(4). 568–575. 5 indexed citations
7.
Zhao, Xin, et al.. (2018). Fragile X mental retardation protein participates in non-coding RNA pathways.. PubMed. 40(2). 87–94.
8.
Yang, Xiaorong, Hongmei Zhang, Jinzi Wu, et al.. (2018). Humanin Attenuates NMDA-Induced Excitotoxicity by Inhibiting ROS-dependent JNK/p38 MAPK Pathway. International Journal of Molecular Sciences. 19(10). 2982–2982. 32 indexed citations
9.
Zhang, Yinghua, Jianzhong Li, Tianbin Song, et al.. (2017). Humanin rescues cultured rat cortical neurons from NMDA-induced toxicity through the alleviation of mitochondrial dysfunction. Drug Design Development and Therapy. Volume 11. 1243–1253. 23 indexed citations
10.
Yang, Xiaorong, et al.. (2017). The Neuroprotective Effects of SIRT1 on NMDA‐Induced Excitotoxicity. Oxidative Medicine and Cellular Longevity. 2017(1). 2823454–2823454. 26 indexed citations
11.
Wang, Rui, Yu Zhang, Jianguo Li, & Ce Zhang. (2016). Resveratrol ameliorates spatial learning memory impairment induced by Aβ 1–42 in rats. Neuroscience. 344. 39–47. 85 indexed citations
12.
13.
Zhang, Qinli, et al.. (2013). Necrostatin-1 inhibits the degeneration of neural cells induced by aluminum exposure. Restorative Neurology and Neuroscience. 31(5). 543–555. 61 indexed citations
14.
Wu, Xia, Zheng Wu, Ce Zhang, et al.. (2013). Homeostatic Competition between Phasic and Tonic Inhibition. Journal of Biological Chemistry. 288(35). 25053–25065. 21 indexed citations
15.
Zhao, Shen‐Ting, Xiaotian Huang, Ce Zhang, & Ya Ke. (2011). Humanin Protects Cortical Neurons from Ischemia and Reperfusion Injury by the Increased Activity of Superoxide Dismutase. Neurochemical Research. 37(1). 153–160. 38 indexed citations
16.
Zhang, Yu, et al.. (2010). Humanin protects neurons against apoptosis induced by Abeta31-35 through suppression of intrinsic pathway.. PubMed. 62(2). 93–100. 4 indexed citations
17.
Liu, Qinghua, et al.. (2009). Aβ31–35-induced neuronal apoptosis is mediated by JNK-dependent extrinsic apoptosis pathway. Neuroscience Bulletin. 25(6). 361–366. 10 indexed citations
18.
19.
Li, Yanli, Xiaorong Yang, Cun‐Gen Ma, Jian‐Tian Qiao, & Ce Zhang. (2008). Necroptosis contributes to the NMDA-induced excitotoxicity in rat’s cultured cortical neurons. Neuroscience Letters. 447(2-3). 120–123. 70 indexed citations
20.
Zhao, Li, Zhong‐Ming Qian, Ce Zhang, et al.. (2007). Amyloid β‐peptide 31–35‐induced neuronal apoptosis is mediated by caspase‐dependent pathways via cAMP‐dependent protein kinase A activation. Aging Cell. 7(1). 47–57. 42 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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