Bruce L. Kagan

7.7k total citations · 1 hit paper
71 papers, 6.3k citations indexed

About

Bruce L. Kagan is a scholar working on Molecular Biology, Physiology and Pharmacology. According to data from OpenAlex, Bruce L. Kagan has authored 71 papers receiving a total of 6.3k indexed citations (citations by other indexed papers that have themselves been cited), including 43 papers in Molecular Biology, 34 papers in Physiology and 13 papers in Pharmacology. Recurrent topics in Bruce L. Kagan's work include Alzheimer's disease research and treatments (33 papers), Lipid Membrane Structure and Behavior (22 papers) and Cholinesterase and Neurodegenerative Diseases (11 papers). Bruce L. Kagan is often cited by papers focused on Alzheimer's disease research and treatments (33 papers), Lipid Membrane Structure and Behavior (22 papers) and Cholinesterase and Neurodegenerative Diseases (11 papers). Bruce L. Kagan collaborates with scholars based in United States, Israel and Japan. Bruce L. Kagan's co-authors include Meng‐Chin Lin, Yutaka Hirakura, Tajib A. Mirzabekov, Ratnesh Lal, Tomas Ganz, Rustam Azimov, Ruth Nussinov, Hyunbum Jang, Srinivasan Ramachandran and Fernando Terán Arce and has published in prestigious journals such as Nature, Science and Proceedings of the National Academy of Sciences.

In The Last Decade

Bruce L. Kagan

71 papers receiving 6.1k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Amyloid ion channels: A common structural link for protein-misfolding disease

2005 831 citations Bruce L. Kagan, Ratnesh Lal et al. profile →

Peers

Bruce L. Kagan

MB

PHYSI

MICRO

BIOMA

IMMUN

László Ötvös United States

Jean‐Marie Ruysschaert Belgium

Agnes Henschen Germany

Jacques Fantini France

Dieter Willbold Germany

Witold K. Surewicz United States

Marie‐Isabel Aguilar Australia

Mauro Dalla Serra Italy

Heinrich Sticht Germany

Christoph Weise Germany

László Ötvös United States

Bruce L. Kagan

4.1k ×1.1

MB

3.9k ×1.3

PHYSI

755 ×0.9

MICRO

238 ×0.3

BIOMA

1.0k ×1.4

IMMUN

Citations per year, relative to Bruce L. Kagan Bruce L. Kagan (= 1×) peers László Ötvös

Countries citing papers authored by Bruce L. Kagan

Since Specialization

Citations

This map shows the geographic impact of Bruce L. Kagan's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Bruce L. Kagan with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Bruce L. Kagan more than expected).

Fields of papers citing papers by Bruce L. Kagan

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Bruce L. Kagan. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Bruce L. Kagan. The network helps show where Bruce L. Kagan may publish in the future.

Co-authorship network of co-authors of Bruce L. Kagan

This figure shows the co-authorship network connecting the top 25 collaborators of Bruce L. Kagan. A scholar is included among the top collaborators of Bruce L. Kagan based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Bruce L. Kagan. Bruce L. Kagan is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

Sort: Min cites: Since: Top N: Style:

20 of 20 papers shown

1.

Fogelson, David L. & Bruce L. Kagan. (2020). Bipolar spectrum disorder masquerading as treatment resistant unipolar depression. CNS Spectrums. 27(1). 4–6. 3 indexed citations

2.

Glasner, Suzette, Frances Kay‐Lambkin, Alan J. Budney, et al.. (2018). Preliminary Outcomes of a Computerized CBT/MET Intervention for Depressed Cannabis Users in Psychiatry Care. PubMed. 1(2). 36–47. 4 indexed citations

3.

Lee, Joon, Young Hun Kim, Fernando Terán Arce, et al.. (2017). Amyloid β Ion Channels in a Membrane Comprising Brain Total Lipid Extracts. ACS Chemical Neuroscience. 8(6). 1348–1357. 63 indexed citations

4.

Jang, Hyunbum, Laura Connelly, Fernando Terán Arce, et al.. (2013). Alzheimer's disease: which type of amyloid-preventing drug agents to employ?. Physical Chemistry Chemical Physics. 15(23). 8868–8868. 43 indexed citations

5.

Kagan, Bruce L.. (2012). Membrane Pores in the Pathogenesis of Neurodegenerative Disease. Progress in molecular biology and translational science. 107. 295–325. 35 indexed citations

6.

Kagan, Bruce L., et al.. (2010). Amyloid Peptide Pores and the Beta Sheet Conformation. Advances in experimental medicine and biology. 677. 150–167. 53 indexed citations

7.

Epand, Richard M., Yanliang Zhang, Tajib A. Mirzabekov, et al.. (2007). Membrane activity of an amphiphilic α-Helical membrane-proximal cytoplasmic domain of the MoMuLV envelope glycoprotein. Experimental and Molecular Pathology. 84(1). 9–17. 6 indexed citations

8.

Kagan, Bruce L., et al.. (2004). Amyloid Peptide Channels. The Journal of Membrane Biology. 202(1). 1–10. 148 indexed citations

9.

Hirakura, Yutaka, Isabel Carreras, Jean D. Sipe, & Bruce L. Kagan. (2002). Channel formation by serum amyloid A: a potential mechanism for amyloid pathogenesis and host defense. Amyloid. 9(1). 13–23. 92 indexed citations

10.

Lin, Meng‐Chin & Bruce L. Kagan. (2002). Electrophysiologic properties of channels induced by Aβ25–35 in planar lipid bilayers. Peptides. 23(7). 1215–1228. 73 indexed citations

11.

Kagan, Bruce L., et al.. (2001). The channel hypothesis of Huntington’s disease. Brain Research Bulletin. 56(3-4). 281–284. 30 indexed citations

12.

Kagan, Bruce L., et al.. (2001). Pore formation by beta-2-microglobulin: A mechanism for the pathogenesis of dialysis associated amyloidosis. Amyloid. 8(2). 94–100. 57 indexed citations

13.

Hirakura, Yutaka, et al.. (2000). Amyloid peptide channels: Blockade by zinc and inhibition by Congo red (amyloid channel block). Amyloid. 7(3). 194–199. 65 indexed citations

14.

Chen, Qi-Sheng, Bruce L. Kagan, Yutaka Hirakura, & Cui-Wei Xie. (2000). Impairment of hippocampal long-term potentiation by Alzheimer amyloid ?-peptides. Journal of Neuroscience Research. 60(1). 65–72. 227 indexed citations

15.

Sokolov, Yuri, Tajib A. Mirzabekov, Daniel W. Martin, Robert I. Lehrer, & Bruce L. Kagan. (1999). Membrane channel formation by antimicrobial protegrins. Biochimica et Biophysica Acta (BBA) - Biomembranes. 1420(1-2). 23–29. 102 indexed citations

16.

Schendel, Sharon L., Rustam Azimov, Krzysztof Pawłowski, et al.. (1999). Ion Channel Activity of the BH3 Only Bcl-2 Family Member, BID. Journal of Biological Chemistry. 274(31). 21932–21936. 163 indexed citations

17.

Lin, Meng‐Chin, Tajib A. Mirzabekov, & Bruce L. Kagan. (1997). Channel Formation by a Neurotoxic Prion Protein Fragment. Journal of Biological Chemistry. 272(1). 44–47. 155 indexed citations

18.

Mirzabekov, Tajib A., Meng‐Chin Lin, & Bruce L. Kagan. (1996). Pore Formation by the Cytotoxic Islet Amyloid Peptide Amylin. Journal of Biological Chemistry. 271(4). 1988–1992. 376 indexed citations

19.

Kagan, Bruce L., et al.. (1994). Defensins: a family of antimicrobial and cytotoxic peptides. Toxicology. 87(1-3). 131–149. 160 indexed citations

20.

Ballard, Jimmy D., Yuri Sokolov, Wu Yuan, Bruce L. Kagan, & Rodney K. Tweten. (1993). Activation and mechanism of Clostridium septicum alpha toxin. Molecular Microbiology. 10(3). 627–634. 71 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

Explore authors with similar magnitude of impact