Boris Klementiev

507 total citations
16 papers, 431 citations indexed

About

Boris Klementiev is a scholar working on Cellular and Molecular Neuroscience, Developmental Neuroscience and Molecular Biology. According to data from OpenAlex, Boris Klementiev has authored 16 papers receiving a total of 431 indexed citations (citations by other indexed papers that have themselves been cited), including 6 papers in Cellular and Molecular Neuroscience, 5 papers in Developmental Neuroscience and 3 papers in Molecular Biology. Recurrent topics in Boris Klementiev's work include Neurogenesis and neuroplasticity mechanisms (3 papers), Alzheimer's disease research and treatments (2 papers) and Neuroscience and Neuropharmacology Research (2 papers). Boris Klementiev is often cited by papers focused on Neurogenesis and neuroplasticity mechanisms (3 papers), Alzheimer's disease research and treatments (2 papers) and Neuroscience and Neuropharmacology Research (2 papers). Boris Klementiev collaborates with scholars based in Denmark, Russia and Estonia. Boris Klementiev's co-authors include Elisabeth Bock, Vladimir Berezin, Peter S. Walmod, Oksana Dmytriyeva, Bente Pakkenberg, Vera Novitskaya, Birte Glenthøj, Thomas Secher, Elisabetta Vaudano and Irina Korshunova and has published in prestigious journals such as PLoS ONE, Neuroscience and European Journal of Neuroscience.

In The Last Decade

Boris Klementiev

16 papers receiving 420 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Boris Klementiev Denmark 10 179 172 122 97 66 16 431
Fleur Davey United Kingdom 10 217 1.2× 194 1.1× 97 0.8× 96 1.0× 47 0.7× 13 470
Maria Angeliki S. Pavlou Germany 12 283 1.6× 157 0.9× 64 0.5× 87 0.9× 86 1.3× 16 539
Andrew Shum United States 9 247 1.4× 236 1.4× 57 0.5× 100 1.0× 49 0.7× 10 525
Noboru Iwakami Japan 9 174 1.0× 118 0.7× 66 0.5× 154 1.6× 47 0.7× 15 390
Shun Shimohama Japan 12 373 2.1× 337 2.0× 106 0.9× 80 0.8× 54 0.8× 14 728
K. Murase Japan 10 132 0.7× 242 1.4× 70 0.6× 90 0.9× 73 1.1× 17 479
Nobutaka Doe Japan 15 190 1.1× 82 0.5× 104 0.9× 80 0.8× 172 2.6× 28 503
Laurie Robak United States 7 266 1.5× 270 1.6× 137 1.1× 59 0.6× 78 1.2× 11 583
Tatyana Kareva United States 12 297 1.7× 373 2.2× 84 0.7× 68 0.7× 72 1.1× 17 665
Christian Lesuisse United States 6 232 1.3× 243 1.4× 55 0.5× 156 1.6× 53 0.8× 6 550

Countries citing papers authored by Boris Klementiev

Since Specialization
Citations

This map shows the geographic impact of Boris Klementiev's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Boris Klementiev with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Boris Klementiev more than expected).

Fields of papers citing papers by Boris Klementiev

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Boris Klementiev. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Boris Klementiev. The network helps show where Boris Klementiev may publish in the future.

Co-authorship network of co-authors of Boris Klementiev

This figure shows the co-authorship network connecting the top 25 collaborators of Boris Klementiev. A scholar is included among the top collaborators of Boris Klementiev based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Boris Klementiev. Boris Klementiev is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

16 of 16 papers shown
1.
Klementiev, Boris, Shizhong Li, Irina Korshunova, et al.. (2014). Anti-inflammatory properties of a novel peptide interleukin 1 receptor antagonist. Journal of Neuroinflammation. 11(1). 27–27. 24 indexed citations
2.
Corbett, Nicola J., P.L.A. Gabbott, Boris Klementiev, et al.. (2013). Amyloid-Beta Induced CA1 Pyramidal Cell Loss in Young Adult Rats Is Alleviated by Systemic Treatment with FGL, a Neural Cell Adhesion Molecule-Derived Mimetic Peptide. PLoS ONE. 8(8). e71479–e71479. 17 indexed citations
3.
Klementiev, Boris, et al.. (2013). BASP1 and Its N-end Fragments (BNEMFs) Dynamics in Rat Brain During Development. Neurochemical Research. 38(6). 1278–1284. 3 indexed citations
4.
Klementiev, Boris, Shizhong Li, R. Carlsson, et al.. (2013). Antiinflammatory properties of a peptide derived from interleukin-4. Cytokine. 64(1). 112–121. 11 indexed citations
5.
Jürgenson, Monika, Külli Jaako, Oksana Dmytriyeva, et al.. (2012). Neuroprotective and memory enhancing properties of a dual agonist of the FGF receptor and NCAM. Neurobiology of Disease. 48(3). 533–545. 16 indexed citations
6.
Dmytriyeva, Oksana, Boris Klementiev, Vladimir Berezin, & Elisabeth Bock. (2012). Prenatal prochloraz treatment significantly increases pregnancy length and reduces offspring weight but does not affect social-olfactory memory in rats. Experimental and Toxicologic Pathology. 65(5). 591–593. 4 indexed citations
7.
Klementiev, Boris, et al.. (2008). The NCAM‐derived P2 peptide facilitates recovery of cognitive and motor function and ameliorates neuropathology following traumatic brain injury. European Journal of Neuroscience. 27(11). 2885–2896. 23 indexed citations
8.
Klementiev, Boris, Vera Novitskaya, Peter S. Walmod, et al.. (2007). A neural cell adhesion molecule–derived peptide reduces neuropathological signs and cognitive impairment induced by Aβ25-35. Neuroscience. 145(1). 209–224. 145 indexed citations
9.
Corbett, Nicola J., P.L.A. Gabbott, Michael Stewart, et al.. (2007). A neural cell adhesion molecule mimetic, FG Loop (FGL), alleviates spine loss induced by beta-amyloid(25-35) in the rat hippocampus. Open Research Online (The Open University). 1 indexed citations
10.
Klementiev, Boris, Karine Cambon, César Venero, et al.. (2007). Effects of P2, a peptide derived from a homophilic binding site in the neural cell adhesion molecule on learning and memory in rats. Neuroscience. 149(4). 931–942. 18 indexed citations
11.
Gotfryd, Kamil, Sylwia Owczarek, Katrin Hoffmann, et al.. (2006). Multiple effects of pentyl-4-yn-VPA enantiomers: From toxicity to short-term memory enhancement. Neuropharmacology. 52(3). 764–778. 9 indexed citations
13.
Skibo, G. G., Iryna Lushnikova, Boris Klementiev, et al.. (2005). A synthetic NCAM‐derived peptide, FGL, protects hippocampal neurons from ischemic insult both in vitro and in vivo. European Journal of Neuroscience. 22(7). 1589–1596. 56 indexed citations
14.
Klementiev, Boris, et al.. (2002). A peptide agonist of the neural cell adhesion molecule (NCAM), C3, protects against developmental defects induced by a teratogen pyrimethamine. International Journal of Developmental Neuroscience. 20(7). 527–536. 14 indexed citations
15.
Mosevitsky, Mark I., et al.. (2001). Not growth associated protein GAP-43 (B-50), but its fragment GAP-43-3 (B-60) predominates in rat brain during development. Neuroscience Letters. 297(1). 49–52. 5 indexed citations
16.
Mosevitsky, Mark I., et al.. (2000). Enhanced level of site-specific proteolysis of GAP-43 protein during early stages of brain development.. PubMed. 65(10). 1153–6. 3 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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