Benjamin J. Murdock
- Neurology top 5%
- Molecular Biology
- Physiology top 10%
- Epidemiology top 10%
- Infectious Diseases top 10%
- Co-authors
- Eva L. FeldmanJohn J. OsterholzerStephen A. GoutmanMichal A. OlszewskiGary B. HuffnagleDiane E. BenderEric S. WhiteAndrew B. Shreiner
- Topics
- Amyotrophic Lateral Sclerosis Research (11 papers)Antifungal resistance and susceptibility (6 papers)Neuroinflammation and Neurodegeneration Mechanisms (5 papers)
- Partner nations
- United StatesJapanMexico
In The Last Decade
Benjamin J. Murdock
27 papers receiving 1.2k citations
Peers
Comparison fields: 5 of 88
- Neurology 412
- Molecular Biology 298
- Physiology 264
- Epidemiology 262
- Infectious Diseases 240
Countries citing papers authored by Benjamin J. Murdock
This map shows the geographic impact of Benjamin J. Murdock's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Benjamin J. Murdock with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Benjamin J. Murdock more than expected).
Fields of papers citing papers by Benjamin J. Murdock
This network shows the impact of papers produced by Benjamin J. Murdock. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Benjamin J. Murdock. The network helps show where Benjamin J. Murdock may publish in the future.
Co-authorship network of co-authors of Benjamin J. Murdock
This figure shows the co-authorship network connecting the top 25 collaborators of Benjamin J. Murdock. A scholar is included among the top collaborators of Benjamin J. Murdock based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Benjamin J. Murdock. Benjamin J. Murdock is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 1 | |
| 2 | 0 | |
| 3 | 2 | |
| 4 | 24 | |
| 5 | 21 | |
| 6 | 32 | |
| 7 | 70 | |
| 8 | 40 | |
| 9 | 47 | |
| 10 | 41 | |
| 11 | 64 | |
| 12 | 60 | |
| 13 | 26 | |
| 14 | 47 | |
| 15 | 96 | |
| 16 | 60 | |
| 17 | 31 | |
| 18 | 87 | |
| 19 | 57 | |
| 20 | Regulation of the TRAIL receptor DR5 expression by the transcription repressor Yin Yang 1 (YY1) | 2 |
About Benjamin J. Murdock
Benjamin J. Murdock is a scholar working on Neurology, Neurology and Genetics, having authored 28 papers that have together received 1.2k indexed citations. Recurring topics across this work include Amyotrophic Lateral Sclerosis Research (11 papers), Antifungal resistance and susceptibility (6 papers) and Neuroinflammation and Neurodegeneration Mechanisms (5 papers). The work is most often cited by research in Neurology (412 citations), Neurology (215 citations) and Genetics (195 citations). Benjamin J. Murdock has collaborated with scholars based in United States, Japan and Mexico. Frequent co-authors include Eva L. Feldman, John J. Osterholzer, Stephen A. Goutman, Michal A. Olszewski, Gary B. Huffnagle, Diane E. Bender, Eric S. White, Andrew B. Shreiner, Roderick J. A. Little and Galen B. Toews. Their work appears in journals such as The Journal of Immunology, Brain and Scientific Reports.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.