Ben G. Wen

1.7k total citations
13 papers, 702 citations indexed

About

Ben G. Wen is a scholar working on Molecular Biology, Immunology and Oncology. According to data from OpenAlex, Ben G. Wen has authored 13 papers receiving a total of 702 indexed citations (citations by other indexed papers that have themselves been cited), including 6 papers in Molecular Biology, 6 papers in Immunology and 4 papers in Oncology. Recurrent topics in Ben G. Wen's work include Immune Cell Function and Interaction (3 papers), T-cell and B-cell Immunology (3 papers) and Chemical Synthesis and Analysis (2 papers). Ben G. Wen is often cited by papers focused on Immune Cell Function and Interaction (3 papers), T-cell and B-cell Immunology (3 papers) and Chemical Synthesis and Analysis (2 papers). Ben G. Wen collaborates with scholars based in United States, Switzerland and United Kingdom. Ben G. Wen's co-authors include Stephen Μ. Hedrick, Craig M. Walsh, Vishva M. Dixit, Arul M. Chinnaiyan, Karen O’Rourke, Naoki Mochizuki, Paul A. Insel, Maureen A. McGargill, Frank Niessen and Richard A. Lerner and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Immunity and The Journal of Immunology.

In The Last Decade

Ben G. Wen

13 papers receiving 690 citations

Peers

Ben G. Wen
Byungje Sung South Korea
Mi Sook Chang United States
Dawne M. Page United States
Marie Wehenkel United States
Sandhya Kumaraswamy United States
Elena Friedmann Switzerland
Byungje Sung South Korea
Ben G. Wen
Citations per year, relative to Ben G. Wen Ben G. Wen (= 1×) peers Byungje Sung

Countries citing papers authored by Ben G. Wen

Since Specialization
Citations

This map shows the geographic impact of Ben G. Wen's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Ben G. Wen with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Ben G. Wen more than expected).

Fields of papers citing papers by Ben G. Wen

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Ben G. Wen. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Ben G. Wen. The network helps show where Ben G. Wen may publish in the future.

Co-authorship network of co-authors of Ben G. Wen

This figure shows the co-authorship network connecting the top 25 collaborators of Ben G. Wen. A scholar is included among the top collaborators of Ben G. Wen based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Ben G. Wen. Ben G. Wen is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

13 of 13 papers shown
1.
Velcicky, Juraj, Casey J. N. Mathison, V. I. Nikulin, et al.. (2019). Discovery of Orally Active Hydroxyethylamine Based SPPL2a Inhibitors. ACS Medicinal Chemistry Letters. 10(6). 887–892. 9 indexed citations
2.
Velcicky, Juraj, Ursula Bodendorf, Pascal Rigollier, et al.. (2018). Discovery of the First Potent, Selective, and Orally Bioavailable Signal Peptide Peptidase-Like 2a (SPPL2a) Inhibitor Displaying Pronounced Immunomodulatory Effects In Vivo. Journal of Medicinal Chemistry. 61(3). 865–880. 20 indexed citations
3.
Miller, Andrew T., Carol Dahlberg, Mark L. Sandberg, et al.. (2015). Inhibition of the Inositol Kinase Itpkb Augments Calcium Signaling in Lymphocytes and Reveals a Novel Strategy to Treat Autoimmune Disease. PLoS ONE. 10(6). e0131071–e0131071. 15 indexed citations
4.
Khare, Shilpi, Xianzhong Liu, Monique Stinson, et al.. (2015). Antitrypanosomal Treatment with Benznidazole Is Superior to Posaconazole Regimens in Mouse Models of Chagas Disease. Antimicrobial Agents and Chemotherapy. 59(10). 6385–6394. 45 indexed citations
5.
Wen, Ben G., et al.. (2014). Correlation Analysis of Rolling Resistance Test Results from SAE J1269 and J2452. SAE technical papers on CD-ROM/SAE technical paper series. 1. 9 indexed citations
6.
Dong, Liqun, Frank Niessen, Ben G. Wen, et al.. (2009). Mechanistic studies of the immunochemical termination of self-tolerance with unnatural amino acids. Proceedings of the National Academy of Sciences. 106(11). 4337–4342. 53 indexed citations
7.
McGargill, Maureen A., et al.. (2008). Drak2 Regulates the Survival of Activated T Cells and Is Required for Organ-Specific Autoimmune Disease. The Journal of Immunology. 181(11). 7593–7605. 26 indexed citations
8.
Perera, Roshan, Liqun Dong, Frank Niessen, et al.. (2008). Immunochemical termination of self-tolerance. Proceedings of the National Academy of Sciences. 105(32). 11276–11280. 60 indexed citations
9.
Wen, Ben G., Gretchen Bain, Barbara L. Kee, et al.. (2005). DRAK2, a lymphoid-enriched DAP kinase, regulates the TCR activation threshold during thymocyte selection. International Immunology. 17(11). 1379–1390. 25 indexed citations
10.
McGargill, Maureen A., Ben G. Wen, Craig M. Walsh, & Stephen Μ. Hedrick. (2004). A Deficiency in Drak2 Results in a T Cell Hypersensitivity and an Unexpected Resistance to Autoimmunity. Immunity. 21(6). 781–791. 62 indexed citations
11.
Wen, Ben G., Mathew T. Pletcher, Masaki Warashina, et al.. (2004). Inositol (1,4,5) trisphosphate 3 kinase B controls positive selection of T cells and modulates Erk activity. Proceedings of the National Academy of Sciences. 101(15). 5604–5609. 66 indexed citations
12.
Walsh, Craig M., Ben G. Wen, Arul M. Chinnaiyan, et al.. (1998). A Role for FADD in T Cell Activation and Development. Immunity. 8(4). 439–449. 215 indexed citations
13.
Mochizuki, Naoki, et al.. (1996). Identification and cDNA cloning of a novel human mosaic protein, LGN, based on interaction with Gαi2. Gene. 181(1-2). 39–43. 97 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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