Axel Niemann
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- Hereditary Neurological Disorders 12
- Genetic Neurodegenerative Diseases 6
- Nerve injury and regeneration 2
- Neurology top 5%
- Neurological diseases and metabolism 3
- Clinical Biochemistry top 5%
- Cell Biology top 5%
- Cellular transport and secretion 2
- Molecular Biology top 10%
- Mitochondrial Function and Pathology 4
- RNA Research and Splicing 3
- RNA regulation and disease 2
- Co-authors
- Ueli SuterPhilipp BergerHans‐Peter ElsässerAngelo SchenoneVeronica La PadulaSawa KostinWolfgang MeißnerAkira Takatsuki
- Journals
- Journal of Biological Chemistry (2 papers)The Journal of Cell Biology (1 paper)PLoS ONE (1 paper)
- Partner nations
- SwitzerlandGermanyUnited States
In The Last Decade
Axel Niemann
19 papers receiving 1.6k citations
Peers
Comparison fields: 5 of 103
- Cellular and Molecular Neuroscience 641
- Neurology 244
- Clinical Biochemistry 125
- Cell Biology 292
- Molecular Biology 1.0k
Countries citing papers authored by Axel Niemann
This map shows the geographic impact of Axel Niemann's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Axel Niemann with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Axel Niemann more than expected).
Fields of papers citing papers by Axel Niemann
This network shows the impact of papers produced by Axel Niemann. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Axel Niemann. The network helps show where Axel Niemann may publish in the future.
Co-authorship network
The 25 scholars most cited alongside Axel Niemann, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2016 | 27 | |
| 2 | 2015 | 1 | |
| 3 | 2014 | 175 | |
| 4 | 2014 | 58 | |
| 5 | 2014 | 4 | |
| 6 | 2013 | 70 | |
| 7 | 2012 | 54 | |
| 8 | 2011 | 15 | |
| 9 | 2011 | 61 | |
| 10 | 2009 | 39 | |
| 11 | 2009 | 110 | |
| 12 | 2006 | 135 | |
| 13 | 2006 | 142 | |
| 14 | 2006 | 19 | |
| 15 | 2005 | 312 | |
| 16 | 2002 | 287 | |
| 17 | 2001 | 46 | |
| 18 | 2000 | 96 | |
| 19 | 1990 | 8 |
About Axel Niemann
Axel Niemann is a scholar working on Cellular and Molecular Neuroscience, Neurology and Biophysics, having authored 19 papers that have together received 1.7k indexed citations. Recurring topics across this work include Hereditary Neurological Disorders (12 papers), Genetic Neurodegenerative Diseases (6 papers), Mitochondrial Function and Pathology (4 papers), Neurological diseases and metabolism (3 papers), RNA Research and Splicing (3 papers), RNA regulation and disease (2 papers), Cellular transport and secretion (2 papers) and Nerve injury and regeneration (2 papers). The work is most often cited by research in Cellular and Molecular Neuroscience (641 citations), Neurology (244 citations) and Clinical Biochemistry (125 citations). Axel Niemann has collaborated with scholars based in Switzerland, Germany and United States. Frequent co-authors include Ueli Suter, Philipp Berger, Hans‐Peter Elsässer, Angelo Schenone, Veronica La Padula, Sawa Kostin, Wolfgang Meißner, Akira Takatsuki, Nina Huber and Milica Bugarski. Their work appears in journals such as Journal of Biological Chemistry, The Journal of Cell Biology and PLoS ONE.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.