S. Guimarães

1.5k total citations
63 papers, 1.2k citations indexed

About

S. Guimarães is a scholar working on Molecular Biology, Physiology and Cellular and Molecular Neuroscience. According to data from OpenAlex, S. Guimarães has authored 63 papers receiving a total of 1.2k indexed citations (citations by other indexed papers that have themselves been cited), including 33 papers in Molecular Biology, 20 papers in Physiology and 11 papers in Cellular and Molecular Neuroscience. Recurrent topics in S. Guimarães's work include Receptor Mechanisms and Signaling (15 papers), Nitric Oxide and Endothelin Effects (9 papers) and Eicosanoids and Hypertension Pharmacology (6 papers). S. Guimarães is often cited by papers focused on Receptor Mechanisms and Signaling (15 papers), Nitric Oxide and Endothelin Effects (9 papers) and Eicosanoids and Hypertension Pharmacology (6 papers). S. Guimarães collaborates with scholars based in Portugal, United Kingdom and Germany. S. Guimarães's co-authors include M. Q. Paiva, Walter Osswald, Michael Schrader, D. Moura, Martin Schuster, Gero Steinberg, Sreedhar Kilaru, António Albino‐Teixeira, Rui Vitorino and Ana R. Freitas and has published in prestigious journals such as Nature Communications, The Journal of Cell Biology and The EMBO Journal.

In The Last Decade

S. Guimarães

59 papers receiving 1.2k citations

Peers

S. Guimarães
Marilyn James-Kracke United States
S. Guimarães
Citations per year, relative to S. Guimarães S. Guimarães (= 1×) peers Marilyn James-Kracke

Countries citing papers authored by S. Guimarães

Since Specialization
Citations

This map shows the geographic impact of S. Guimarães's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by S. Guimarães with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites S. Guimarães more than expected).

Fields of papers citing papers by S. Guimarães

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by S. Guimarães. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by S. Guimarães. The network helps show where S. Guimarães may publish in the future.

Co-authorship network of co-authors of S. Guimarães

This figure shows the co-authorship network connecting the top 25 collaborators of S. Guimarães. A scholar is included among the top collaborators of S. Guimarães based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with S. Guimarães. S. Guimarães is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Guimarães, S., et al.. (2025). Amphiphilic Poly(γ‐glutamic acid) Derivatives for Delivering Doxorubicin in Cancer Cells. Macromolecular Bioscience. 25(10). e00090–e00090.
2.
Xavier, Miguel, Ariel Ionescu, María Gómez-Lázaro, et al.. (2025). Engineered Chitosan‐Derived Nanocarrier for Efficient siRNA Delivery to Peripheral and Central Neurons. Advanced Healthcare Materials. 14(15). e2500107–e2500107. 1 indexed citations
3.
Guimarães, S., et al.. (2022). A paradigm shift: Bioengineering meets mechanobiology towards overcoming remyelination failure. Biomaterials. 283. 121427–121427. 8 indexed citations
4.
Freitas, Ana R., et al.. (2017). Biological Implications of Differential Expression of Mitochondrial-Shaping Proteins in Parkinson’s Disease. Antioxidants. 7(1). 1–1. 81 indexed citations
5.
Guimarães, S., Martin Schuster, Ewa Bielska, et al.. (2015). Peroxisomes, lipid droplets, and endoplasmic reticulum “hitchhike” on motile early endosomes. The Journal of Cell Biology. 211(5). 945–954. 97 indexed citations
6.
Islinger, Markus, S. Guimarães, Sreedhar Kilaru, et al.. (2014). New insights into the peroxisomal protein inventory: Acyl-CoA oxidases and -dehydrogenases are an ancient feature of peroxisomes. Biochimica et Biophysica Acta (BBA) - Molecular Cell Research. 1853(1). 111–125. 56 indexed citations
7.
Conde, Carlos, Mariana Osswald, Diana Pinheiro, et al.. (2013). Drosophila Polo regulates the spindle assembly checkpoint through Mps1‐dependent BubR1 phosphorylation. The EMBO Journal. 32(12). 1761–1777. 41 indexed citations
8.
Carvalho, Catarina, Sérgio Gaião, S. Guimarães, et al.. (2009). Cerebral coccidioidomycosis after renal transplantation in a non-endemic area. Transplant Infectious Disease. 12(2). 151–154. 23 indexed citations
10.
Mota, Alberto, M. Q. Paiva, D. Moura, & S. Guimarães. (2000). Lack of interaction between α2-autoreceptors and prejunctional receptors mediating a facilitatory effect on noradrenaline release. Pharmacological Research. 42(4). 383–387. 9 indexed citations
11.
Gautier-Vargas, Gabriela, et al.. (2000). Risk factors for posttransplant diabetes mellitus and impact of this complication after renal transplantation. Transplantation Proceedings. 32(8). 2609–2610. 10 indexed citations
12.
Guimarães, João Tiago, M. Q. Paiva, D. Moura, & S. Guimarães. (1998). Influence of maturation and ageing on the biotransformation of noradrenaline in the rat. Journal of neural transmission. Supplementum. 52. 225–232. 3 indexed citations
13.
Guimarães, S., Isabel Vitória Figueiredo, Maria Margarida Caramona, D. Moura, & M. Q. Paiva. (1998). Prejunctional α2A-autoreceptors in the human gastric and ileocolic arteries. Naunyn-Schmiedeberg s Archives of Pharmacology. 358(2). 207–211. 11 indexed citations
14.
Lobato, Luísa, et al.. (1998). Familial amyloid polyneuropathy type I (Portuguese): Distribution and characterization of renal amyloid deposits. American Journal of Kidney Diseases. 31(6). 940–946. 52 indexed citations
15.
Paiva, M. Q., Alberto Mota, D. Moura, & S. Guimarães. (1997). Prejunctional α2A-autoreceptors in the canine saphenous vein. Naunyn-Schmiedeberg s Archives of Pharmacology. 356(3). 368–373. 3 indexed citations
16.
Vaz‐da‐Silva, Manuel, et al.. (1996). The role of the endocardium in the facilitatory effect of bradykinin on electrically‐induced release of noradrenaline in rat cardiac ventricle. British Journal of Pharmacology. 118(2). 364–368. 18 indexed citations
17.
Guimarães, S. & António Albino‐Teixeira. (1996). Hypertension due to chronic blockade of P1‐purinoceptors. Journal of Autonomic Pharmacology. 16(6). 367–370. 18 indexed citations
18.
Guimarães, S., António Albino‐Teixeira, & M. Q. Paiva. (1995). Hypertension and enhanced β‐adrenoceptor‐mediated facilitation of noradrenaline release produced by chronic blockade of adenosine receptors. British Journal of Pharmacology. 114(8). 1595–1598. 13 indexed citations
19.
Guimarães, S., M. Q. Paiva, D. Moura, Manuel Vaz‐da‐Silva, & António Albino‐Teixeira. (1994). Long-term administration of 1,3-dipropyl-8-sulphophenylxanthine (DPSPX) alters ?2-adrenoceptor-mediated effects at the pre- but not at the postjunctional level. Naunyn-Schmiedeberg s Archives of Pharmacology. 350(6). 692–5. 10 indexed citations
20.
Pereira, Oduvaldo Câmara Marques, et al.. (1991). Involvement of ?1- and ?2-adrenoceptors in the responses of proximal and distal segments of the canine saphenous vein to exogenous and endogenous noradrenaline. Naunyn-Schmiedeberg s Archives of Pharmacology. 343(6). 616–22. 4 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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