Nina Huber

5.3k total citations · 4 hit papers
14 papers, 3.7k citations indexed

About

Nina Huber is a scholar working on Molecular Biology, Cellular and Molecular Neuroscience and Neurology. According to data from OpenAlex, Nina Huber has authored 14 papers receiving a total of 3.7k indexed citations (citations by other indexed papers that have themselves been cited), including 9 papers in Molecular Biology, 7 papers in Cellular and Molecular Neuroscience and 3 papers in Neurology. Recurrent topics in Nina Huber's work include Hereditary Neurological Disorders (5 papers), Genetic Neurodegenerative Diseases (3 papers) and Neurogenesis and neuroplasticity mechanisms (3 papers). Nina Huber is often cited by papers focused on Hereditary Neurological Disorders (5 papers), Genetic Neurodegenerative Diseases (3 papers) and Neurogenesis and neuroplasticity mechanisms (3 papers). Nina Huber collaborates with scholars based in United States, Switzerland and Germany. Nina Huber's co-authors include Sergiu P. Pașca, Christopher D. Makinson, Nancy O’Rourke, John R. Huguenard, Ben A. Barres, Steven A. Sloan, Fikri Birey, Jinyoung Park, Chul Hoon Kim and Daniel H. Geschwind and has published in prestigious journals such as Nature, Science and Nature Communications.

In The Last Decade

Nina Huber

14 papers receiving 3.6k citations

Hit Papers

Functional cortical neurons and astrocytes from human plu... 2015 2026 2018 2022 2015 2017 2016 2017 250 500 750 1000

Peers

Nina Huber
Anca M. Pașca United States
Louise S. Bicknell New Zealand
Jimena Andersen United States
Georgia Panagiotakos United States
Su-Chun Zhang United States
Giorgia Quadrato United States
Christopher A. Fasano United States
Fikri Birey United States
Anca M. Pașca United States
Nina Huber
Citations per year, relative to Nina Huber Nina Huber (= 1×) peers Anca M. Pașca

Countries citing papers authored by Nina Huber

Since Specialization
Citations

This map shows the geographic impact of Nina Huber's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Nina Huber with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Nina Huber more than expected).

Fields of papers citing papers by Nina Huber

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Nina Huber. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Nina Huber. The network helps show where Nina Huber may publish in the future.

Co-authorship network of co-authors of Nina Huber

This figure shows the co-authorship network connecting the top 25 collaborators of Nina Huber. A scholar is included among the top collaborators of Nina Huber based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Nina Huber. Nina Huber is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

14 of 14 papers shown
1.
Huber, Nina, et al.. (2023). Spatial transcriptomics using multiplexed deterministic barcoding in tissue. Nature Communications. 14(1). 1523–1523. 21 indexed citations
2.
Huber, Nina, et al.. (2021). Virtual Reality–Based and Conventional Visual Field Examination Comparison in Healthy and Glaucoma Patients. Translational Vision Science & Technology. 10(12). 10–10. 45 indexed citations
3.
Trevino, Alexandro E., Nasa Sinnott-Armstrong, Jimena Andersen, et al.. (2020). Chromatin accessibility dynamics in a model of human forebrain development. Science. 367(6476). 149 indexed citations
4.
Zhang, Yifan, Nina Huber, Ralf Moeller, et al.. (2019). Role of DNA repair in Bacillus subtilis spore resistance to high energy and low energy electron beam treatments. Food Microbiology. 87. 103353–103353. 16 indexed citations
5.
Madelaine, Romain, Steven A. Sloan, Nina Huber, et al.. (2017). MicroRNA-9 Couples Brain Neurogenesis and Angiogenesis. Cell Reports. 20(7). 1533–1542. 97 indexed citations
6.
Birey, Fikri, Jimena Andersen, Christopher D. Makinson, et al.. (2017). Assembly of functionally integrated human forebrain spheroids. Nature. 545(7652). 54–59. 888 indexed citations breakdown →
7.
Sloan, Steven A., Spyros Darmanis, Nina Huber, et al.. (2017). Human Astrocyte Maturation Captured in 3D Cerebral Cortical Spheroids Derived from Pluripotent Stem Cells. Neuron. 95(4). 779–790.e6. 397 indexed citations breakdown →
8.
Deverman, Benjamin E., Sripriya Ravindra Kumar, Ken Y. Chan, et al.. (2016). Cre-dependent selection yields AAV variants for widespread gene transfer to the adult brain. Nature Biotechnology. 34(2). 204–209. 728 indexed citations breakdown →
9.
Huber, Nina, Christoph Bieniossek, Hans‐Peter Elsässer, et al.. (2016). Glutathione-conjugating and membrane-remodeling activity of GDAP1 relies on amphipathic C-terminal domain. Scientific Reports. 6(1). 36930–36930. 27 indexed citations
10.
Pașca, Anca M., Steven A. Sloan, Laura Clarke, et al.. (2015). Functional cortical neurons and astrocytes from human pluripotent stem cells in 3D culture. Nature Methods. 12(7). 671–678. 1079 indexed citations breakdown →
11.
Niemann, Axel, Nina Huber, Christian Somandin, et al.. (2014). The Gdap1 knockout mouse mechanistically links redox control to Charcot–Marie–Tooth disease. Brain. 137(3). 668–682. 58 indexed citations
12.
Huber, Nina, S. Guimarães, Michael Schrader, Ueli Suter, & Axel Niemann. (2013). Charcot‐Marie‐Tooth disease‐associated mutants of GDAP1 dissociate its roles in peroxisomal and mitochondrial fission. EMBO Reports. 14(6). 545–552. 70 indexed citations
13.
Kabzińska, Dagmara, Axel Niemann, H Drac, et al.. (2011). A new missense GDAP1 mutation disturbing targeting to the mitochondrial membrane causes a severe form of AR-CMT2C disease. Neurogenetics. 12(2). 145–153. 15 indexed citations
14.
Zimoń, M., Jonathan Baets, Gian Maria Fabrizi, et al.. (2011). Dominant GDAP1 mutations cause predominantly mild CMT phenotypes. Neurology. 77(6). 540–548. 61 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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