Ave Eesmaa

570 total citations
8 papers, 390 citations indexed

About

Ave Eesmaa is a scholar working on Cell Biology, Cellular and Molecular Neuroscience and Molecular Biology. According to data from OpenAlex, Ave Eesmaa has authored 8 papers receiving a total of 390 indexed citations (citations by other indexed papers that have themselves been cited), including 5 papers in Cell Biology, 4 papers in Cellular and Molecular Neuroscience and 3 papers in Molecular Biology. Recurrent topics in Ave Eesmaa's work include Endoplasmic Reticulum Stress and Disease (5 papers), Nerve injury and regeneration (4 papers) and Adenosine and Purinergic Signaling (2 papers). Ave Eesmaa is often cited by papers focused on Endoplasmic Reticulum Stress and Disease (5 papers), Nerve injury and regeneration (4 papers) and Adenosine and Purinergic Signaling (2 papers). Ave Eesmaa collaborates with scholars based in Finland, Estonia and Germany. Ave Eesmaa's co-authors include Märt Saarma, Päivi Lindholm, Mari Sepp, Tõnis Timmusk, Li-Ying Yu, Maria Lindahl, Kohsuke Kanekura, Fumihiko Urano, Rie Asada and Takuya Yagi and has published in prestigious journals such as Journal of Biological Chemistry, PLoS ONE and International Journal of Molecular Sciences.

In The Last Decade

Ave Eesmaa

8 papers receiving 387 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Ave Eesmaa Finland	8	160	146	124	86	76	8	390
Amy Peaire Canada	6	217 1.4×	126 0.9×	137 1.1×	88 1.0×	57 0.8×	8	405
Laura Flinn United Kingdom	5	86 0.5×	170 1.2×	171 1.4×	61 0.7×	99 1.3×	6	440
J. Susie Zoltewicz United States	8	120 0.8×	296 2.0×	98 0.8×	57 0.7×	72 0.9×	9	424
Fengfa Huang United States	9	159 1.0×	195 1.3×	114 0.9×	68 0.8×	45 0.6×	13	432
Peter A. Barbuti Luxembourg	9	121 0.8×	207 1.4×	122 1.0×	49 0.6×	152 2.0×	15	374
José Wojnacki Argentina	11	156 1.0×	233 1.6×	209 1.7×	30 0.3×	25 0.3×	19	429
Jennifer L. Badger United Kingdom	6	124 0.8×	204 1.4×	107 0.9×	64 0.7×	183 2.4×	7	440
Ewa Bomba-Warczak United States	10	135 0.8×	175 1.2×	111 0.9×	39 0.5×	65 0.9×	13	399
Maila Giannandrea Italy	8	129 0.8×	164 1.1×	132 1.1×	33 0.4×	126 1.7×	8	384
Josta T. Kevenaar Netherlands	7	213 1.3×	229 1.6×	223 1.8×	22 0.3×	125 1.6×	7	498

Countries citing papers authored by Ave Eesmaa

Since Specialization

Citations

This map shows the geographic impact of Ave Eesmaa's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Ave Eesmaa with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Ave Eesmaa more than expected).

Fields of papers citing papers by Ave Eesmaa

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Ave Eesmaa. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Ave Eesmaa. The network helps show where Ave Eesmaa may publish in the future.

Co-authorship network of co-authors of Ave Eesmaa

This figure shows the co-authorship network connecting the top 25 collaborators of Ave Eesmaa. A scholar is included among the top collaborators of Ave Eesmaa based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Ave Eesmaa. Ave Eesmaa is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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8 of 8 papers shown

1.

Kovaleva, Vera, Li-Ying Yu, Larisa Ivanova, et al.. (2023). MANF regulates neuronal survival and UPR through its ER-located receptor IRE1α. Cell Reports. 42(2). 112066–112066. 36 indexed citations

2.

Eesmaa, Ave, Li-Ying Yu, Helka Göös, et al.. (2022). CDNF Interacts with ER Chaperones and Requires UPR Sensors to Promote Neuronal Survival. International Journal of Molecular Sciences. 23(16). 9489–9489. 19 indexed citations

3.

Albert, Katrina, Anne Panhelainen, Ave Eesmaa, et al.. (2021). Cerebral dopamine neurotrophic factor reduces α-synuclein aggregation and propagation and alleviates behavioral alterations in vivo. Molecular Therapy. 29(9). 2821–2840. 27 indexed citations

4.

Eesmaa, Ave, Li-Ying Yu, Helka Göös, et al.. (2021). The cytoprotective protein MANF promotes neuronal survival independently from its role as a GRP78 cofactor. Journal of Biological Chemistry. 296. 100295–100295. 38 indexed citations

5.

Yagi, Takuya, Rie Asada, Kohsuke Kanekura, et al.. (2020). Neuroplastin Modulates Anti-inflammatory Effects of MANF. iScience. 23(12). 101810–101810. 54 indexed citations

6.

Eesmaa, Ave, Päivi Lindholm, Christina Schlumbohm, et al.. (2016). Comparative Analysis of the Effects of Neurotrophic Factors CDNF and GDNF in a Nonhuman Primate Model of Parkinson’s Disease. PLoS ONE. 11(2). e0149776–e0149776. 71 indexed citations

7.

Mätlik, Kert, Li-Ying Yu, Ave Eesmaa, et al.. (2015). Role of two sequence motifs of mesencephalic astrocyte-derived neurotrophic factor in its survival-promoting activity. Cell Death and Disease. 6(12). e2032–e2032. 50 indexed citations

8.

Sepp, Mari, et al.. (2011). Functional Diversity of Human Basic Helix-Loop-Helix Transcription Factor TCF4 Isoforms Generated by Alternative 5′ Exon Usage and Splicing. PLoS ONE. 6(7). e22138–e22138. 95 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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