Wallace Y. Langdon

11.6k total citations · 2 hit papers
122 papers, 9.2k citations indexed

About

Wallace Y. Langdon is a scholar working on Molecular Biology, Immunology and Oncology. According to data from OpenAlex, Wallace Y. Langdon has authored 122 papers receiving a total of 9.2k indexed citations (citations by other indexed papers that have themselves been cited), including 71 papers in Molecular Biology, 42 papers in Immunology and 24 papers in Oncology. Recurrent topics in Wallace Y. Langdon's work include Ubiquitin and proteasome pathways (20 papers), T-cell and B-cell Immunology (19 papers) and Protein Kinase Regulation and GTPase Signaling (16 papers). Wallace Y. Langdon is often cited by papers focused on Ubiquitin and proteasome pathways (20 papers), T-cell and B-cell Immunology (19 papers) and Protein Kinase Regulation and GTPase Signaling (16 papers). Wallace Y. Langdon collaborates with scholars based in Australia, United States and China. Wallace Y. Langdon's co-authors include Christine B.F. Thien, Herbert C. Morse, Jerry M. Adams, Hamid Band, Ivan Đikić, David D.L. Bowtell, Christopher E. Andoniou, Suzanne Cory, Timothy J. Blake and Iwona Szymkiewicz and has published in prestigious journals such as Nature, Cell and Proceedings of the National Academy of Sciences.

In The Last Decade

Wallace Y. Langdon

120 papers receiving 9.1k citations

Hit Papers

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What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

c-Cbl/Sli-1 regulates endocytic sorting and ubiquitination of the epidermal growth factor receptor

1998 714 citations Wallace Y. Langdon et al. Genes & Development profile →
Cbl: many adaptations to regulate protein tyrosine kinases

2001 521 citations Christine B.F. Thien, Wallace Y. Langdon profile →

Author Peers

Peers are selected by citation overlap in the author's most active subfields. citations · hero ref

Author						Papers	Cites
Wallace Y. Langdon	5.9k	2.7k	2.4k	1.3k	1.0k	122	9.2k
Robert Rottapel	4.0k 0.7×	3.7k 1.3×	2.5k 1.0×	858 0.7×	766 0.8×	134	8.2k
Keith C. Robbins	5.1k 0.9×	1.6k 0.6×	1.8k 0.8×	896 0.7×	620 0.6×	94	8.1k
Jacalyn H. Pierce	6.2k 1.1×	3.4k 1.2×	4.0k 1.7×	743 0.6×	646 0.6×	128	11.5k
Larry R. Rohrschneider	4.3k 0.7×	2.0k 0.7×	1.4k 0.6×	997 0.8×	521 0.5×	100	6.9k
Thomas O. Daniel	4.6k 0.8×	1.2k 0.5×	1.4k 0.6×	966 0.7×	1.0k 1.0×	58	7.2k
Andrew F. Wilks	4.8k 0.8×	2.8k 1.0×	4.5k 1.9×	673 0.5×	472 0.5×	101	9.0k
Yuji Yamanashi	3.5k 0.6×	2.3k 0.8×	1.2k 0.5×	817 0.6×	322 0.3×	94	7.1k
Norman N. Iscove	4.5k 0.8×	3.1k 1.1×	1.7k 0.7×	638 0.5×	2.4k 2.3×	76	9.1k
David T. Weaver	6.5k 1.1×	1.4k 0.5×	2.6k 1.1×	747 0.6×	336 0.3×	150	9.0k
André Veillette	5.7k 1.0×	9.9k 3.6×	3.5k 1.4×	716 0.5×	1.1k 1.1×	182	14.7k

Countries citing papers authored by Wallace Y. Langdon

Since Specialization

Citations

This map shows the geographic impact of Wallace Y. Langdon's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Wallace Y. Langdon with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Wallace Y. Langdon more than expected).

Fields of papers citing papers by Wallace Y. Langdon

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Wallace Y. Langdon. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Wallace Y. Langdon. The network helps show where Wallace Y. Langdon may publish in the future.

Co-authorship network of co-authors of Wallace Y. Langdon

This figure shows the co-authorship network connecting the top 25 collaborators of Wallace Y. Langdon. A scholar is included among the top collaborators of Wallace Y. Langdon based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Wallace Y. Langdon. Wallace Y. Langdon is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Tang, Rong, Wallace Y. Langdon, & Jian Zhang. (2022). Negative regulation of receptor tyrosine kinases by ubiquitination: Key roles of the Cbl family of E3 ubiquitin ligases. Frontiers in Endocrinology. 13. 971162–971162. 22 indexed citations

Tang, Rong, Wallace Y. Langdon, & Jian Zhang. (2018). Regulation of immune responses by E3 ubiquitin ligase Cbl-b. Cellular Immunology. 340. 103878–103878. 58 indexed citations

Taylor, Samuel J., et al.. (2016). Dasatinib promotes the activation of quiescent hematopoietic stem cells in mice. Experimental Hematology. 44(5). 410–421.e5. 8 indexed citations

Rathinam, Chozhavendan, Christine B.F. Thien, Richard A. Flavell, & Wallace Y. Langdon. (2010). Myeloid Leukemia Development in c-Cbl RING Finger Mutant Mice Is Dependent on FLT3 Signaling. Cancer Cell. 18(4). 341–352. 74 indexed citations

Thien, Christine B.F., James H. Steer, Frank Köentgen, et al.. (2010). c-Cbl Promotes T Cell Receptor-induced Thymocyte Apoptosis by Activating the Phosphatidylinositol 3-Kinase/Akt Pathway. Journal of Biological Chemistry. 285(14). 10969–10981. 19 indexed citations

Mamchak, Alusha A., Christine B.F. Thien, Julia Lyandres, et al.. (2009). Unaltered negative selection and Treg development of self‐reactive thymocytes in TCR transgenic Fyn‐deficient mice. European Journal of Immunology. 40(2). 539–547. 2 indexed citations

Rathinam, Chozhavendan, Christine B.F. Thien, Wallace Y. Langdon, Hua Gu, & Richard A. Flavell. (2008). The E3 ubiquitin ligase c-Cbl restricts development and functions of hematopoietic stem cells. Genes & Development. 22(8). 992–997. 83 indexed citations

Gustin, Sonja E., Christine B.F. Thien, & Wallace Y. Langdon. (2006). Cbl-b Is a Negative Regulator of Inflammatory Cytokines Produced by IgE-Activated Mast Cells. The Journal of Immunology. 177(9). 5980–5989. 31 indexed citations

Gaston, Isabelle, Kara Johnson, Tsukasa Oda, et al.. (2004). Coexistence of phosphotyrosine-dependent and -independent interactions between Cbl and Bcr-Abl. Experimental Hematology. 32(1). 113–121. 7 indexed citations

10.

Oksvold, Morten P., Henrik S. Huitfeldt, & Wallace Y. Langdon. (2004). Identification of 14‐3‐3ζ as an EGF receptor interacting protein. FEBS Letters. 569(1-3). 207–210. 43 indexed citations

11.

Scaife, Robin M., Didier Job, & Wallace Y. Langdon. (2003). Rapid Microtubule-dependent Induction of Neurite-like Extensions in NIH 3T3 Fibroblasts by Inhibition of ROCK and Cbl. Molecular Biology of the Cell. 14(11). 4605–4617. 26 indexed citations

12.

Thien, Christine B.F., Francesca Walker, & Wallace Y. Langdon. (2001). RING Finger Mutations that Abolish c-Cbl-Directed Polyubiquitination and Downregulation of the EGF Receptor Are Insufficient for Cell Transformation. Molecular Cell. 7(2). 355–365. 145 indexed citations

13.

Peschard, Pascal, Tanya M. Fournier, Louie Lamorte, et al.. (2001). Mutation of the c-Cbl TKB Domain Binding Site on the Met Receptor Tyrosine Kinase Converts It into a Transforming Protein. Molecular Cell. 8(5). 995–1004. 346 indexed citations

14.

Kirsch, Kathrin H., Maria-Magdalena Georgescu, Tomoyuki Shishido, et al.. (2001). The Adapter Type Protein CMS/CD2AP Binds to the Proto-oncogenic Protein c-Cbl through a Tyrosine Phosphorylation-regulated Src Homology 3 Domain Interaction. Journal of Biological Chemistry. 276(7). 4957–4963. 74 indexed citations

15.

Fournier, Tanya M., Louie Lamorte, Christiane R. Maroun, et al.. (2000). Cbl-transforming Variants Trigger a Cascade of Molecular Alterations That Lead to Epithelial Mesenchymal Conversion. Molecular Biology of the Cell. 11(10). 3397–3410. 30 indexed citations

16.

Lupher, Mark, Kris A. Reedquist, Sachiko Miyake, Wallace Y. Langdon, & Hamid Band. (1996). A Novel Phosphotyrosine-binding Domain in the N-terminal Transforming Region of Cbl Interacts Directly and Selectively with ZAP-70 in T Cells. Journal of Biological Chemistry. 271(39). 24063–24068. 179 indexed citations

17.

Langdon, Wallace Y.. (1995). The cbl oncogene: a novel substrate of protein tyrosine kinases. Australian and New Zealand Journal of Medicine. 25(6). 859–864. 33 indexed citations

18.

Harris, A. W., Carl A. Pinkert, M Crawford, et al.. (1988). The E mu-myc transgenic mouse. A model for high-incidence spontaneous lymphoma and leukemia of early B cells.. The Journal of Experimental Medicine. 167(2). 353–371. 346 indexed citations

19.

Morse, Herbert C., George F. Tidmarsh, Kevin L. Holmes, et al.. (1987). Expression of the 6C3 antigen on murine hematopoietic neoplasms. Association with expression of abl, ras, fes, src, erbB, and Cas NS-1 oncogenes but not with myc.. The Journal of Experimental Medicine. 165(3). 920–925. 11 indexed citations

20.

Morse, Herbert C., J B Roths, Wendy F. Davidson, et al.. (1985). Abnormalities induced by the mutant gene, lpr. Patterns of disease and expression of murine leukemia viruses in SJL/J mice homozygous and heterozygous for lpr.. The Journal of Experimental Medicine. 161(3). 602–616. 38 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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