Anthony J. Mirando

2.0k total citations
35 papers, 1.5k citations indexed

About

Anthony J. Mirando is a scholar working on Molecular Biology, Rheumatology and Immunology and Allergy. According to data from OpenAlex, Anthony J. Mirando has authored 35 papers receiving a total of 1.5k indexed citations (citations by other indexed papers that have themselves been cited), including 20 papers in Molecular Biology, 15 papers in Rheumatology and 6 papers in Immunology and Allergy. Recurrent topics in Anthony J. Mirando's work include Osteoarthritis Treatment and Mechanisms (15 papers), Developmental Biology and Gene Regulation (8 papers) and Cell Adhesion Molecules Research (6 papers). Anthony J. Mirando is often cited by papers focused on Osteoarthritis Treatment and Mechanisms (15 papers), Developmental Biology and Gene Regulation (8 papers) and Cell Adhesion Molecules Research (6 papers). Anthony J. Mirando collaborates with scholars based in United States, China and Canada. Anthony J. Mirando's co-authors include Matthew J. Hilton, Wei Hsu, Jiang Fu, Hsiao‐Man Ivy Yu, Regis J. O’Keefe, Takamitsu Maruyama, Zhaoyang Liu, Michael J. Zuscik, Ming Jiang and Yinshi Ren and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Journal of Clinical Investigation and Development.

In The Last Decade

Anthony J. Mirando

34 papers receiving 1.5k citations

Peers

Anthony J. Mirando

RHEUM

GENET

ONCOL

Jennifer H. Jonason United States

Tujun Weng China

Yangli Xie China

Takeshi Moriishi Japan

Robert J. Tower United States

Zhengjian Yan China

Jixing Ye China

Toshihiro Miyazaki Japan

Kinglun Kingston Mak Hong Kong

Hisato Komori Japan

Jennifer H. Jonason United States

Anthony J. Mirando

803 ×0.9

439 ×1.3

RHEUM

140 ×0.6

GENET

187 ×0.9

257 ×1.3

ONCOL

Citations per year, relative to Anthony J. Mirando Anthony J. Mirando (= 1×) peers Jennifer H. Jonason

Countries citing papers authored by Anthony J. Mirando

Since Specialization

Citations

This map shows the geographic impact of Anthony J. Mirando's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Anthony J. Mirando with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Anthony J. Mirando more than expected).

Fields of papers citing papers by Anthony J. Mirando

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Anthony J. Mirando. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Anthony J. Mirando. The network helps show where Anthony J. Mirando may publish in the future.

Co-authorship network of co-authors of Anthony J. Mirando

This figure shows the co-authorship network connecting the top 25 collaborators of Anthony J. Mirando. A scholar is included among the top collaborators of Anthony J. Mirando based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Anthony J. Mirando. Anthony J. Mirando is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Camarena, Adrian, et al.. (2024). Platelet-rich plasma enhances rib fracture strength and callus formation in vivo. The Journal of Trauma: Injury, Infection, and Critical Care. 97(6). 884–890. 3 indexed citations

Mirando, Anthony J., et al.. (2023). A Review of Existing Commercial Mass Timber Buildings in the United States. EPiC series in built environment. 4. 560–550.

Rampersad, Rishi R., Amanda M. Eudy, Yinshi Ren, et al.. (2023). Aged G Protein-Coupled Receptor Kinase 3 (Grk3)-Deficient Mice Exhibit Enhanced Osteoclastogenesis and Develop Bone Lesions Analogous to Human Paget’s Disease of Bone. Cells. 12(7). 981–981. 1 indexed citations

Mirando, Anthony J., Douglas Rouse, Purushothama Rao Tata, et al.. (2022). Identification of distinct non-myogenic skeletal-muscle-resident mesenchymal cell populations. Cell Reports. 39(6). 110785–110785. 35 indexed citations

Liao, Yihan, Yinshi Ren, Xin Luo, et al.. (2022). Interleukin-6 signaling mediates cartilage degradation and pain in posttraumatic osteoarthritis in a sex-specific manner. Science Signaling. 15(744). eabn7082–eabn7082. 69 indexed citations

Sharma, Deepika, et al.. (2021). HES1 is a novel downstream modifier of the SHH-GLI3 Axis in the development of preaxial polydactyly. PLoS Genetics. 17(12). e1009982–e1009982. 5 indexed citations

Wang, Kaiyuan, Yun Gu, Yihan Liao, et al.. (2020). PD-1 blockade inhibits osteoclast formation and murine bone cancer pain. Journal of Clinical Investigation. 130(7). 3603–3620. 131 indexed citations

Liao, Yihan, et al.. (2020). Isolation and Culture of Murine Primary Chondrocytes: Costal and Growth Plate Cartilage. Methods in molecular biology. 2230. 415–423. 5 indexed citations

Yu, Yilin, Hunter Newman, Leyao Shen, et al.. (2019). Glutamine Metabolism Regulates Proliferation and Lineage Allocation in Skeletal Stem Cells. Cell Metabolism. 29(4). 966–978.e4. 192 indexed citations

10.

Tsushima, Hidetoshi, Yuning J. Tang, Vijitha Puviindran, et al.. (2018). Intracellular biosynthesis of lipids and cholesterol by Scap and Insig in mesenchymal cells regulates long bone growth and chondrocyte homeostasis. Development. 145(13). 21 indexed citations

11.

Cao, Chike, Yinshi Ren, Adam S. Barnett, et al.. (2017). Increased Ca2+ signaling through CaV1.2 promotes bone formation and prevents estrogen deficiency–induced bone loss. JCI Insight. 2(22). 35 indexed citations

12.

Kohn, Anat, et al.. (2016). HES factors regulate specific aspects of chondrogenesis and chondrocyte hypertrophy during cartilage development. Journal of Cell Science. 129(11). 2145–2155. 22 indexed citations

13.

Mirando, Anthony J., et al.. (2015). A dual role for notch signaling in joint cartilage maintenance and osteoarthritis. Osteoarthritis and Cartilage. 23. A64–A64. 3 indexed citations

14.

Kohn, Anat, Zhaoyang Liu, Anthony J. Mirando, et al.. (2015). Notch signaling controls chondrocyte hypertrophy via indirect regulation of Sox9. Bone Research. 3(1). 15021–15021. 36 indexed citations

15.

Liu, Zhaoyang, Yinshi Ren, Anthony J. Mirando, et al.. (2015). Notch signaling in postnatal joint chondrocytes, but not subchondral osteoblasts, is required for articular cartilage and joint maintenance. Osteoarthritis and Cartilage. 24(4). 740–751. 25 indexed citations

16.

Fu, Jiang, Hongbo Yu, Shang-Yi Chiu, et al.. (2014). Disruption of SUMO-Specific Protease 2 Induces Mitochondria Mediated Neurodegeneration. PLoS Genetics. 10(10). e1004579–e1004579. 69 indexed citations

17.

Mirando, Anthony J., Yufeng Dong, Jinsil Kim, & Matthew J. Hilton. (2014). Isolation and Culture of Murine Primary Chondrocytes. Methods in molecular biology. 1130. 267–277. 23 indexed citations

18.

Fu, Jiang, Hsiao‐Man Ivy Yu, Takamitsu Maruyama, Anthony J. Mirando, & Wei Hsu. (2011). Gpr177/mouse Wntless is essential for Wnt‐mediated craniofacial and brain development. Developmental Dynamics. 240(2). 365–371. 85 indexed citations

19.

Mirando, Anthony J., Takamitsu Maruyama, Jiang Fu, Hsiao‐Man Ivy Yu, & Wei Hsu. (2010). β-catenin/cyclin D1 mediated development of suture mesenchyme in calvarial morphogenesis. BMC Developmental Biology. 10(1). 116–116. 33 indexed citations

20.

Hsu, Wei, Anthony J. Mirando, & Hsiao‐Man Ivy Yu. (2009). Manipulating gene activity in Wnt1‐expressing precursors of neural epithelial and neural crest cells. Developmental Dynamics. 239(1). 338–345. 18 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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