Matthew J. Hilton

6.8k total citations · 1 hit paper
88 papers, 5.3k citations indexed

About

Matthew J. Hilton is a scholar working on Molecular Biology, Rheumatology and Surgery. According to data from OpenAlex, Matthew J. Hilton has authored 88 papers receiving a total of 5.3k indexed citations (citations by other indexed papers that have themselves been cited), including 45 papers in Molecular Biology, 40 papers in Rheumatology and 17 papers in Surgery. Recurrent topics in Matthew J. Hilton's work include Osteoarthritis Treatment and Mechanisms (34 papers), Developmental Biology and Gene Regulation (13 papers) and Bone Metabolism and Diseases (13 papers). Matthew J. Hilton is often cited by papers focused on Osteoarthritis Treatment and Mechanisms (34 papers), Developmental Biology and Gene Regulation (13 papers) and Bone Metabolism and Diseases (13 papers). Matthew J. Hilton collaborates with scholars based in United States, China and Japan. Matthew J. Hilton's co-authors include Fanxin Long, Xiaolin Tu, Regis J. O’Keefe, Michael J. Zuscik, Anthony J. Mirando, Hongliang Hu, Ximei Wu, David M. Ornitz, Di Chen and Kai Yu and has published in prestigious journals such as Cell, Journal of Biological Chemistry and Journal of Clinical Investigation.

In The Last Decade

Matthew J. Hilton

86 papers receiving 5.2k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Sequential roles of Hedgehog and Wnt signaling in osteoblast development

2004 536 citations Hongliang Hu, Matthew J. Hilton et al. Development profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Matthew J. Hilton United States	37	3.2k	1.4k	854	698	665	88	5.3k
T. Michael Underhill Canada	44	2.9k 0.9×	1.0k 0.7×	611 0.7×	748 1.1×	505 0.8×	108	5.1k
Shinsuke Ohba Japan	41	3.9k 1.2×	1.3k 1.0×	978 1.1×	732 1.0×	925 1.4×	135	6.5k
Mei Wan United States	45	3.8k 1.2×	1.4k 1.0×	1.4k 1.7×	727 1.0×	794 1.2×	127	6.9k
Hicham Drissi United States	49	4.2k 1.3×	1.9k 1.4×	1.4k 1.6×	1.1k 1.6×	910 1.4×	177	7.0k
Ling Qin United States	39	2.1k 0.7×	1.1k 0.8×	1.4k 1.6×	601 0.9×	479 0.7×	131	4.5k
Jennifer J. Westendorf United States	49	4.9k 1.5×	916 0.7×	1.6k 1.9×	626 0.9×	961 1.4×	141	7.5k
Kazuhisa Nakashima Japan	30	4.6k 1.4×	1.6k 1.2×	1.5k 1.7×	572 0.8×	918 1.4×	77	6.9k
Amy D. Bradshaw United States	41	1.7k 0.5×	1.7k 1.3×	791 0.9×	833 1.2×	569 0.9×	93	5.2k
Thomas A. Owen United States	38	3.6k 1.1×	1.4k 1.1×	1.1k 1.3×	578 0.8×	409 0.6×	78	6.6k
Masahiro Iwamoto United States	42	2.7k 0.8×	2.8k 2.1×	568 0.7×	719 1.0×	570 0.9×	99	5.7k

Countries citing papers authored by Matthew J. Hilton

Since Specialization

Citations

This map shows the geographic impact of Matthew J. Hilton's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Matthew J. Hilton with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Matthew J. Hilton more than expected).

Fields of papers citing papers by Matthew J. Hilton

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Matthew J. Hilton. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Matthew J. Hilton. The network helps show where Matthew J. Hilton may publish in the future.

Co-authorship network of co-authors of Matthew J. Hilton

This figure shows the co-authorship network connecting the top 25 collaborators of Matthew J. Hilton. A scholar is included among the top collaborators of Matthew J. Hilton based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Matthew J. Hilton. Matthew J. Hilton is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Levin, Jay M., et al.. (2025). Myosteatosis and skeletal muscle atrophy, but not fibrosis, are correlated with increasing tear sizes of full-thickness rotator cuff tears. Journal of Shoulder and Elbow Surgery. 35(1). e30–e45.

Hoque, Jiaul, Vijitha Puviindran, Hunter Newman, et al.. (2023). Radiation-induced bone loss in mice is ameliorated by inhibition of HIF-2α in skeletal progenitor cells. Science Translational Medicine. 15(724). eabo5217–eabo5217. 3 indexed citations

Rampersad, Rishi R., Amanda M. Eudy, Yinshi Ren, et al.. (2023). Aged G Protein-Coupled Receptor Kinase 3 (Grk3)-Deficient Mice Exhibit Enhanced Osteoclastogenesis and Develop Bone Lesions Analogous to Human Paget’s Disease of Bone. Cells. 12(7). 981–981. 1 indexed citations

Bokshan, Steven L., et al.. (2023). Risk factors for intracellular fatty accumulation in rotator cuff muscle: a histologic analysis. Journal of Shoulder and Elbow Surgery. 33(4). e215–e222. 2 indexed citations

Mirando, Anthony J., Douglas Rouse, Purushothama Rao Tata, et al.. (2022). Identification of distinct non-myogenic skeletal-muscle-resident mesenchymal cell populations. Cell Reports. 39(6). 110785–110785. 35 indexed citations

Liao, Yihan, Yinshi Ren, Xin Luo, et al.. (2022). Interleukin-6 signaling mediates cartilage degradation and pain in posttraumatic osteoarthritis in a sex-specific manner. Science Signaling. 15(744). eabn7082–eabn7082. 69 indexed citations

Wang, Kaiyuan, Christopher R. Donnelly, Changyu Jiang, et al.. (2021). STING suppresses bone cancer pain via immune and neuronal modulation. Nature Communications. 12(1). 4558–4558. 91 indexed citations

Sharma, Deepika, et al.. (2021). HES1 is a novel downstream modifier of the SHH-GLI3 Axis in the development of preaxial polydactyly. PLoS Genetics. 17(12). e1009982–e1009982. 5 indexed citations

Karner, Courtney M., et al.. (2021). Hypoxia depletes contaminating CD45+ hematopoietic cells from murine bone marrow stromal cell (BMSC) cultures: Methods for BMSC culture purification. Stem Cell Research. 53. 102317–102317. 3 indexed citations

10.

Lee, Chang‐Lung, Dadong Zhang, Alexander B. Sibley, et al.. (2021). Whole-Exome Sequencing of Radiation-Induced Thymic Lymphoma in Mouse Models Identifies Notch1 Activation as a Driver of p53 Wild-Type Lymphoma. Cancer Research. 81(14). 3777–3790. 12 indexed citations

11.

Wang, Kaiyuan, Yun Gu, Yihan Liao, et al.. (2020). PD-1 blockade inhibits osteoclast formation and murine bone cancer pain. Journal of Clinical Investigation. 130(7). 3603–3620. 131 indexed citations

12.

Tsushima, Hidetoshi, Yuning J. Tang, Vijitha Puviindran, et al.. (2018). Intracellular biosynthesis of lipids and cholesterol by Scap and Insig in mesenchymal cells regulates long bone growth and chondrocyte homeostasis. Development. 145(13). 21 indexed citations

13.

Kohn, Anat, et al.. (2016). HES factors regulate specific aspects of chondrogenesis and chondrocyte hypertrophy during cartilage development. Journal of Cell Science. 129(11). 2145–2155. 22 indexed citations

14.

Mirando, Anthony J., et al.. (2015). A dual role for notch signaling in joint cartilage maintenance and osteoarthritis. Osteoarthritis and Cartilage. 23. A64–A64. 3 indexed citations

15.

Jones, Kevin B., Maurizio Pacifici, & Matthew J. Hilton. (2014). Multiple hereditary exostoses (MHE): elucidating the pathogenesis of a rare skeletal disorder through interdisciplinary research. Connective Tissue Research. 55(2). 80–88. 18 indexed citations

16.

Wang, Cuicui, Jie Shen, Kiminori Yukata, et al.. (2014). Transient gamma-secretase inhibition accelerates and enhances fracture repair likely via Notch signaling modulation. Bone. 73. 77–89. 21 indexed citations

17.

Hilton, Matthew J.. (2014). Skeletal development and repair : methods and protocols. Humana Press eBooks. 14 indexed citations

18.

Dong, Yufeng, Anat Kohn, Tasuku Honjo, et al.. (2010). RBPjκ-dependent Notch signaling regulates mesenchymal progenitor cell proliferation and differentiation during skeletal development. Development. 137(9). 1461–1471. 149 indexed citations

19.

Christopher, Matthew, Fulu Liu, Matthew J. Hilton, Fanxin Long, & Daniel C. Link. (2009). Suppression of CXCL12 production by bone marrow osteoblasts is a common and critical pathway for cytokine-induced mobilization. Blood. 114(7). 1331–1339. 172 indexed citations

20.

Hu, Hongliang, Matthew J. Hilton, Xiaolin Tu, et al.. (2004). Sequential roles of Hedgehog and Wnt signaling in osteoblast development. Development. 132(1). 49–60. 536 indexed citations breakdown →

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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